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- 2011
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Mark
Insulinotropic and Antidiabetic Effects of 17{beta}-Estradiol and the GPR30 Agonist G-1 on Human Pancreatic Islets.
(
- Contribution to journal › Article
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Mark
Abnormally decreased NO and augmented CO production in islets of the leptin-deficient ob/ob mouse might contribute to explain hyperinsulinemia and islet survival in leptin-resistant type 2 obese diabetes.
(
- Contribution to journal › Article
- 2010
-
Mark
ADP mediates inhibition of insulin secretion by activation of P2Y13 receptors in mice.
(
- Contribution to journal › Article
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Mark
Proghrelin peptides: Desacyl ghrelin is a powerful inhibitor of acylated ghrelin, likely to impair physiological effects of acyl ghrelin but not of obestatin A study of pancreatic polypeptide secretion from mouse islets.
(
- Contribution to journal › Article
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Mark
Defective secretion of islet hormones in chromogranin-B deficient mice.
(
- Contribution to journal › Article
- 2009
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Mark
Palmitate-stimulated hormone secretion in relation to GPR40 expression in pancreatic islets of spontaneous obesity and type 2 diabetes in rats
2009) 45th Annual Meeting of the European-Association-for-the-Study-of-Diabetes In Diabetologia 52(Suppl. 1). p.405-405(
- Contribution to journal › Published meeting abstract
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Mark
Deletion of the G protein-coupled Receptor GPR30 Impairs Glucose Tolerance, Reduces Bone Growth, Increases Blood Pressure, and Eliminates Estradiol-stimulated Insulin Release in Female Mice.
(
- Contribution to journal › Article
-
Mark
Islet-cell secretion of pancreatic polypeptide in response to ghrelin and obestatin acting on distinct receptors
2009) 45th Annual Meeting of the European-Association-for-the-Study-of-Diabetes In Diabetologia 52(Suppl 1). p.605-605(
- Contribution to journal › Published meeting abstract
- 2008
-
Mark
Proghrelin-derived peptides influence the secretion of insulin, glucagon, pancreatic polypeptide and somatostatin: A study on isolated islets from mouse and rat pancreas.
(
- Contribution to journal › Article
-
Mark
Palmitate-induced beta-cell dysfunction is associated with excessive NO pro-duction and is reversed by thiazolidinedione-mediated inhibition of GPR40 transduction mechanisms
2008) In PLoS ONE(
- Contribution to specialist publication or newspaper › Specialist publication article