Javier Jimenez (Former)
1 – 10 of 11
- show: 10
- |
- sort: year (new to old)
Close
Embed this list
<iframe src=" "
width=" "
height=" "
allowtransparency="true"
frameborder="0">
</iframe>
- 2023
-
Mark
An innovative Superconducting Recoil Separator for HIE-ISOLDE
2023) In Nuclear Instruments and Methods in Physics Research, Section B: Beam Interactions with Materials and Atoms 541. p.176-179(
- Contribution to journal › Article
- 2011
-
Mark
Abnormally decreased NO and augmented CO production in islets of the leptin-deficient ob/ob mouse might contribute to explain hyperinsulinemia and islet survival in leptin-resistant type 2 obese diabetes.
(
- Contribution to journal › Article
- 2009
-
Mark
Insulin secretion is highly sensitive to desorption of plasma membrane cholesterol.
2009) In The FASEB journal : official publication of the Federation of American Societies for Experimental Biology 23(1). p.58-67(
- Contribution to journal › Article
- 2008
-
Mark
Impaired glucose-stimulated insulin secretion in the GK rat is associated with abnormalities in islet nitric oxide production.
(
- Contribution to journal › Article
-
Mark
Excessive islet NO generation in type 2 diabetic GK rats coincides with abnormal hormone secretion and is counteracted by GLP-1.
(
- Contribution to journal › Article
- 2007
-
Mark
Expression of islet iNOS and inhibition of glucose stimulated insulin release after long-term lipid infusion in the rat is counteracted by PACAP27.
(
- Contribution to journal › Article
- 2006
-
Mark
Selective induction of inducible nitric oxide synthase in pancreatic islet of rat after an intravenous glucose or intralipid challenge.
(
- Contribution to journal › Article
- 2005
-
Mark
Glucose stimulates the expression and activities of nitric oxide synthases in incubated rat islets: an effect counteracted by GLP-1 through the cyclic AMP/PKA pathway
(
- Contribution to journal › Article
-
Mark
Free fatty acid receptor 1 (FFA(1)R/GPR40) and its involvement in fatty-acid-stimulated insulin secretion.
(
- Contribution to journal › Article
-
Mark
Defective glucose-stimulated insulin release in the diabetic Goto-Kakizaki (GK) rat coincides with reduced activity of the islet carbon monoxide signaling pathway
(
- Contribution to journal › Article