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- 2012
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Mark
Viral Stimuli Trigger Exaggerated Thymic Stromal Lymphopoietin Expression by Chronic Obstructive Pulmonary Disease Epithelium: Role of Endosomal TLR3 and Cytosolic RIG-I-Like Helicases.
(
- Contribution to journal › Article
- 2011
-
Mark
Exit of leucocytes across the alveolar epithelium worsens lung injury Authors' response
(
- Contribution to journal › Letter
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Mark
Effects of the cyclin-dependent kinase inhibitor R-roscovitine on eosinophil survival and clearance.
(
- Contribution to journal › Article
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Mark
Letter to Respiratory Medicine: "Drugs reducing transepithelial leukocyte traffic may worsen lung disease"
(
- Contribution to journal › Letter
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Mark
dsRNA-induced expression of thymic stromal lymphopoietin (TSLP) in asthmatic epithelial cells is inhibited by a small airway relaxant.
(
- Contribution to journal › Article
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Mark
Increased fecal leukocytes may reflect resolution of IBD.
(
- Contribution to journal › Article
- 2010
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Mark
Resolution of cell-mediated airways diseases
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- Contribution to journal › Scientific review
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Mark
Early phase resolution of mucosal eosinophilic inflammation in allergic rhinitis.
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- Contribution to journal › Article
- 2009
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Mark
Roles of plasma exudation in asthma and COPD
(
- Contribution to journal › Debate/Note/Editorial
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Mark
Allergic Airway Inflammation Initiates Long-Term Vascular Remodeling of the Pulmonary Circulation.
(
- Contribution to journal › Article
- 2008
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Mark
Remodeling of extra-bronchial lung vasculature following allergic airway inflammation.
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- Contribution to journal › Article
- 2007
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Mark
Antagonism of the prostaglandin D2 receptor CRTH2 attenuates asthma pathology in mouse eosinophilic airway inflammation.
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- Contribution to journal › Article
- 2006
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Mark
Effects of steroid treatment on lung CC chemokines, apoptosis and transepithelial cell clearance during development and resolution of allergic airway inflammation.
(
- Contribution to journal › Article
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Mark
Resolution of airway disease: removal of inflammatory cells through apoptosis, egression or both?
(
- Contribution to journal › Scientific review
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Mark
Novel selective orally active CRTH2 antagonists for allergic inflammation developed from in silico derived hits
(
- Contribution to journal › Article