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- 2016
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Mark
Conserved Patterns of Microbial Immune Escape: Pathogenic Microbes of Diverse Origin Target the Human Terminal Complement Inhibitor Vitronectin via a Single Common Motif.
(
- Contribution to journal › Article
- 2015
-
Mark
Pseudomonas aeruginosa Uses Dihydrolipoamide Dehydrogenase (Lpd) to Bind to the Human Terminal Pathway Regulators Vitronectin and Clusterin to Inhibit Terminal Pathway Complement Attack.
(
- Contribution to journal › Article
- 2014
-
Mark
Candida albicans Uses the Surface Protein Gpm1 to Attach to Human Endothelial Cells and to Keratinocytes via the Adhesive Protein Vitronectin.
(
- Contribution to journal › Article
- 2013
-
Mark
CspA from Borrelia burgdorferi Inhibits the Terminal Complement Pathway
(
- Contribution to journal › Article
- 2012
-
Mark
Dihydrolipoamide Dehydrogenase of Pseudomonas aeruginosa Is a Surface-Exposed Immune Evasion Protein That Binds Three Members of the Factor H Family and Plasminogen
(
- Contribution to journal › Article
- 2011
-
Mark
The pH-regulated Antigen 1 of Candida albicans Binds the Human Complement Inhibitor C4b-binding Protein and Mediates Fungal Complement Evasion
(
- Contribution to journal › Article
- 2010
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Mark
Pseudomonas aeruginosa CRASP-2 is a surface protein that uses the human terminal complement regulator vitronectin for protection against complement-mediated attack
(
- Contribution to journal › Published meeting abstract
-
Mark
CRASP2 of Candida albicans binds C4b-binding protein and mediates fungal contact with human endothelial cells
(
- Contribution to journal › Published meeting abstract
- 2009
-
Mark
Pseudomonas aeruginosa CRASP-2 is a novel multifunctional surface protein that binds Factor H, Factor H related protein 1 and plasminogen
2009) 12th European Meeting on Complement in Human Disease In Molecular Immunology 46(14). p.2835-2835(
- Contribution to journal › Published meeting abstract