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- 2019
-
Mark
C3 glomerulopathy — understanding a rare complement-driven renal disease
2019) In Nature Reviews Nephrology(
- Contribution to journal › Scientific review
- 2017
-
Mark
Vitronectin Binds to a Specific Stretch within the Head Region of Yersinia Adhesin A and Thereby Modulates Yersinia enterocolitica Host Interaction
(
- Contribution to journal › Article
- 2016
-
Mark
Conserved Patterns of Microbial Immune Escape: Pathogenic Microbes of Diverse Origin Target the Human Terminal Complement Inhibitor Vitronectin via a Single Common Motif.
(
- Contribution to journal › Article
-
Mark
The complement inhibitor factor H generates an anti-inflammatory and tolerogenic state in monocyte-derived dendritic cells
(
- Contribution to journal › Article
- 2015
-
Mark
Pseudomonas aeruginosa Uses Dihydrolipoamide Dehydrogenase (Lpd) to Bind to the Human Terminal Pathway Regulators Vitronectin and Clusterin to Inhibit Terminal Pathway Complement Attack.
(
- Contribution to journal › Article
-
Mark
Moraxella catarrhalis binds plasminogen to evade host innate immunity.
(
- Contribution to journal › Article
- 2014
-
Mark
Candida albicans Uses the Surface Protein Gpm1 to Attach to Human Endothelial Cells and to Keratinocytes via the Adhesive Protein Vitronectin.
(
- Contribution to journal › Article
-
Mark
Identification of a Haemophilus influenzae Factor H-Binding Lipoprotein Involved in Serum Resistance.
(
- Contribution to journal › Article
- 2013
-
Mark
Human complement control and complement evasion by pathogenic microbes - Tipping the balance
(
- Contribution to journal › Scientific review
-
Mark
CspA from Borrelia burgdorferi Inhibits the Terminal Complement Pathway
(
- Contribution to journal › Article
-
Mark
The Choline-binding Protein PspC of Streptococcus pneumoniae Interacts with the C-terminal Heparin-binding Domain of Vitronectin
(
- Contribution to journal › Article
- 2012
-
Mark
Haemophilus influenzae Uses the Surface Protein E To Acquire Human Plasminogen and To Evade Innate Immunity
(
- Contribution to journal › Debate/Note/Editorial
-
Mark
Dihydrolipoamide Dehydrogenase of Pseudomonas aeruginosa Is a Surface-Exposed Immune Evasion Protein That Binds Three Members of the Factor H Family and Plasminogen
(
- Contribution to journal › Article
-
Mark
Factor H autoantibodies and deletion of Complement Factor H-Related protein-1 in rheumatic diseases in comparison to atypical hemolytic uremic syndrome
(
- Contribution to journal › Article
- 2011
-
Mark
The pH-regulated Antigen 1 of Candida albicans Binds the Human Complement Inhibitor C4b-binding Protein and Mediates Fungal Complement Evasion
(
- Contribution to journal › Article
-
Mark
Monomeric C-reactive protein modulates classic complement activation on necrotic cells
(
- Contribution to journal › Article
-
Mark
Immune Evasion of Moraxella catarrhalis Involves Ubiquitous Surface Protein A-Dependent C3d Binding.
(
- Contribution to journal › Article
- 2010
-
Mark
Pseudomonas aeruginosa CRASP-2 is a surface protein that uses the human terminal complement regulator vitronectin for protection against complement-mediated attack
(
- Contribution to journal › Published meeting abstract
-
Mark
CRASP2 of Candida albicans binds C4b-binding protein and mediates fungal contact with human endothelial cells
(
- Contribution to journal › Published meeting abstract
- 2009
-
Mark
Nontypeable Haemophilus influenzae Protein E Binds Vitronectin and Is Important for Serum Resistance
(
- Contribution to journal › Article
-
Mark
Pseudomonas aeruginosa CRASP-2 is a novel multifunctional surface protein that binds Factor H, Factor H related protein 1 and plasminogen
2009) 12th European Meeting on Complement in Human Disease In Molecular Immunology 46(14). p.2835-2835(
- Contribution to journal › Published meeting abstract
- 2008
-
Mark
Haemophilus influenzae interacts with the human complement inhibitor factor H
(
- Contribution to journal › Article
-
Mark
Factor H dysfunction in patients with atypical hemolytic uremic syndrome contributes to complement deposition on platelets and their activation.
(
- Contribution to journal › Article
- 2002
-
Mark
Streptococcus pneumoniae evades complement attack and opsonophagocytosis by expressing the pspC locus-encoded Hic protein that binds to short consensus repeats 8-11 of factor H
(
- Contribution to journal › Article