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- 2011
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Mark
High levels of the adhesion molecule CD44 on leukemic cells generate acute myeloid leukemia relapse after withdrawal of the initial transforming event.
(
- Contribution to journal › Article
- 2010
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Mark
High Levels of the Adhesion Molecule CD44 on Leukemic Cells Generate Acute Myeloid Leukemia Relapse After Withdrawal of the Initial Transforming Event
(
- Contribution to journal › Published meeting abstract
- 2009
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Mark
In an inducible model of acute myeloid leukemia, secondary hits are required to generate a leukemia relapse after removal of the initial transforming event
2009) 38th Annual Scientific Meeting of the ISEH-Society-for-Hematology-and-Stem-Cells In Experimental Hematology 37(9). p.16-16(
- Contribution to journal › Published meeting abstract
- 2008
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Mark
A HOXA10-inducible mouse model demonstrates an important role of the initial transforming event in the maintenance of the leukemia
2008) 37th Annual Scientific Meeting featuring the 6th International Neonatal Hematology and Immunology Meeting In Experimental Hematology 36(7, Supplement 1). p.4-5(
- Contribution to journal › Published meeting abstract
- 2007
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Mark
Hoxa9/hoxb3/hoxb4 compound null mice display severe hematopoietic defects
(
- Contribution to journal › Article
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Mark
HOXA10 is a critical regulator for hematopoietic stem cells and erythroid/megakaryocyte development.
(
- Contribution to journal › Article
- 2006
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Mark
HOXB4-induced self-renewal of hematopoietic stem cells is significantly enhanced by p21 deficiency.
(
- Contribution to journal › Article
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Mark
Smad7 promotes self-renewal of hematopoietic stem cells in vivo.
(
- Contribution to journal › Article
- 2005
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Mark
Quiescence of hematopoietic stem cells and maintenance of the stem cell pool is not dependent on TGF-beta signaling in vivo.
(
- Contribution to journal › Article
- 2004
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Mark
Development of an adenoviral vector system with adenovirus serotype 35 tropism; efficient transient gene transfer into primary malignant hematopoietic cells.
(
- Contribution to journal › Article
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Mark
Hoxb4-deficient mice undergo normal hematopoietic development but exhibit a mild proliferation defect in hematopoietic stem cells
(
- Contribution to journal › Article
- 2003
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Mark
Reduced proliferative capacity of hematopoietic stem cells deficient in hoxb3 and hoxb4.
(
- Contribution to journal › Article
- 2002
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Mark
Transient disruption of autocrine TGF-beta signaling leads to enhanced survival and proliferation potential in single primitive human hemopoietic progenitor cells.
(
- Contribution to journal › Article