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- 2021
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Mark
Cell Type Dependent Suppression of Inflammatory Mediators by Myocardin Related Transcription Factors
(
- Contribution to journal › Article
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Mark
NG2/CSPG4, CD146/MCAM and VAP1/AOC3 are regulated by myocardin-related transcription factors in smooth muscle cells
(
- Contribution to journal › Article
- 2020
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Mark
Human host defense peptide LL-37 facilitates double-stranded RNA pro-inflammatory signaling through up-regulation of TLR3 expression in vascular smooth muscle cells
(
- Contribution to journal › Article
- 2018
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Mark
Nexilin/NEXN controls actin polymerization in smooth muscle and is regulated by myocardin family coactivators and YAP
(
- Contribution to journal › Article
- 2015
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Mark
Inhibition of polyamine uptake potentiates the anti-proliferative effect of polyamine synthesis inhibition and preserves the contractile phenotype of vascular smooth muscle cells.
2015) In Journal of Cellular Physiology(
- Contribution to journal › Article
- 2014
-
Mark
Vascular smooth muscle cell proliferation depends on caveolin-1-regulated polyamine uptake
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- Contribution to journal › Article
- 2012
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Mark
Deletion of dicer in smooth muscle affects voiding pattern and reduces detrusor contractility and neuroeffector transmission.
(
- Contribution to journal › Article
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Mark
Impaired contractility and detrusor hypertrophy in cavin-1-deficient mice.
(
- Contribution to journal › Article
- 2009
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Mark
Unaltered Size-selectivity of the Glomerular Filtration Barrier in Caveolin-1 Knock-out (KO) mice.
(
- Contribution to journal › Article
- 2007
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Mark
Disproportionally low clearance of macromolecules from the plasma to the peritoneal cavity in a mouse model of peritoneal dialysis (PD).
(
- Contribution to journal › Article
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Mark
Arterial remodeling and plasma volume expansion in caveolin-1 deficient mice.
2007) In American Journal of Physiology: Regulatory, Integrative and Comparative Physiology 293. p.1222-1231(
- Contribution to journal › Article
- 2006
-
Mark
Transvascular protein transport in mice lacking endothelial caveolae.
(
- Contribution to journal › Article