Emma Berntman (Former)

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2008
Mark Molecular profiling reveals distinct functional attributes of CD1d-restricted natural killer (NK) T cell subsets
Rolf, Julia ^LU ; Berntman, Emma ^LU ; Stenström, Martin ^LU ; Smith, Emma ^LU ; Månsson, Robert ^LU ; Stenstad, Hanna ^LU ; Yamagata, Tetsuya ; Agace, William ^LU ; Sigvardsson, Mikael ^LU and Cardell, Susanna ^LU (2008) In Molecular Immunology 45(9). p.2607-2620
- Contribution to journal › Article
2007
Mark Severe defect in thymic development in an insertional mutant mouse model
Assarsson, Erika ; Chambers, Benedict J. ; Hogstrand, Kari ; Berntman, Emma ^LU ; Lundmark, Carin ; Fedorova, Ludmila ; Imreh, Stefan ; Grandien, Alf ; Cardell, Susanna ^LU and Rozell, Bjorn , et al. (2007) In Journal of Immunology 178(8). p.5018-5027
- Contribution to journal › Article
2006
Mark Immuno-modulatory functions of CD1d-restricted natural killer T cells
Berntman, Emma ^LU (2006)
- Thesis › Doctoral thesis (compilation)
Mark Congenic nonobese diabetic mouse strains fail to confirm linkage of a marginal zone B lymphocyte phenotype to the Idd11 locus on chromosome 4 - Response
Rolf, Julia ^LU ; Motta, V ; Duarte, N ; Lundholm, M ; Berntman, Emma ^LU ; Bergman, ML ; Sorokin, Lydia ^LU ; Cardell, Susanna ^LU and Holmberg, D (2006) In Journal of Immunology 176(2). p.702-702
- Contribution to journal › Letter
2005
Mark The role of CD1d-restricted NK T lymphocytes in the immune response to oral infection with Salmonella typhimurium.
Berntman, Emma ^LU ; Rolf, Julia ^LU ; Johansson, Cecilia ^LU ; Anderson, Per ^LU and Cardell, Susanna ^LU (2005) In European Journal of Immunology 35(7). p.2100-2109
- Contribution to journal › Article
Mark The enlarged population of marginal zone/CD1d(high) B lymphocytes in nonobese diabetic mice maps to diabetes susceptibility region Idd11.
Rolf, Julia ^LU ; Motta, Vinicius ; Duarte, Nadia ; Lundholm, Marie ; Berntman, Emma ^LU ; Bergman, Marie-Louise ; Sorokin, Lydia ^LU ; Cardell, Susanna ^LU and Holmberg, Dan (2005) In Journal of Immunology 174(8). p.4821-4827
- Contribution to journal › Article