Zhongquan Qi
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- 2012
-
Streptococcal M1 Protein-Provoked CXC Chemokine Formation, Neutrophil Recruitment and Lung Damage Are Regulated by Rho-Kinase Signaling.
Zhang, Songen LU ; Rahman, Milladur LU
; Zhang, Su
LU
; Herwald, Heiko
LU
; Qi, Zhongquan
LU
; Jeppsson, Bengt
LU
and Thorlacius, Henrik
LU
(2012)
In Journal of Innate Immunity
4(4).
p.399-408
Mark
-
Rho-kinase regulates adhesive and mechanical mechanisms of pulmonary recruitment of neutrophils in abdominal sepsis.
Palani, Karzan LU ; Rahman, Milladur LU
; Hasan, Zirak
LU
; Zhang, Su
LU
; Qi, Zhongquan
LU
; Jeppsson, Bengt
LU
and Thorlacius, Henrik
LU
(2012)
In European Journal of Pharmacology
682(1-3).
p.181-187
Mark
- Immunosuppressive properties of a series of novel inhibitors of the monocarboxylate transporter MCT-1.
- 2011
-
Simvastatin regulates CXC chemokine formation in streptococcal M1 protein-induced neutrophil infiltration in the lung
Zhang, Songen LU ; Rahman, Milladur LU
; Zhang, Su
LU
; Qi, Zhongquan
LU
; Herwald, Heiko
LU
and Thorlacius, Henrik
LU
(2011)
In American Journal of Physiology: Lung Cellular and Molecular Physiology
300(6).
p.930-939
Mark
-
Simvastatin antagonizes CD40L secretion, CXC chemokine formation, and pulmonary infiltration of neutrophils in abdominal sepsis.
Zhang, Su LU ; Rahman, Milladur LU
; Zhang, Songen
; Qi, Zhongquan
LU
and Thorlacius, Henrik
LU
(2011)
In Journal of Leukocyte Biology
89.
p.735-742
Mark
- The major histocompatibility complex (MHC) of the secondary transplant tissue donor influences the cross-reactivity of alloreactive memory cells.
- CD44/CD70 blockade and anti-CD154/LFA-1 treatment synergistically suppress accelerated rejection and prolong cardiac allograft survival in mice.
- 2010
- Combination of antibodies inhibits accelerated rejection mediated by memory T cells in xenoantigen-primed mice.
- 2008
- Operational tolerance in nonvascularized transplant models induced by AR-C117977, a monocarboxylate transporter inhibitor.
- 2006
- Costimulation Blockade-Induced Cardiac Allograft Tolerance: Inhibition of T Cell Expansion and Accumulation of Intragraft cD4+Foxp3+ T Cells.