Björn Dahlbäck
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- 1997
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Mark
Genetic and phenotypic analysis of a large (122-member) protein S-deficient kindred provides an explanation for the familial coexistence of type I and type III plasma phenotypes
(
- Contribution to journal › Article
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Mark
Activated protein C resistance caused by a common factor V mutation has a single origin
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- Contribution to journal › Article
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Mark
The factor VR506Q mutation causing APC resistance is highly prevalent amongst unselected outpatients with clinically suspected deep venous thrombosis
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- Contribution to journal › Article
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Mark
Evaluation of original and modified APC-resistance tests in unselected outpatients with clinically suspected thrombosis and in healthy controls
(
- Contribution to journal › Article
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Mark
Activated protein C resistance due to a common factor V gene mutation is a major risk factor for venous thrombosis
(
- Contribution to journal › Article
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Mark
Activated protein C resistance : Clinical implications
(
- Contribution to journal › Scientific review
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Mark
Presence of vitamin K dependent coagulation proteins in isolated lipoproteins from human plasma.
(
- Contribution to journal › Article
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Mark
The amino-terminal module of the C4b-binding protein alpha-chain is crucial for C4b binding and factor I-cofactor function
(
- Contribution to journal › Article
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Mark
Molecular cloning of rat C4b binding protein alpha- and beta-chains : structural and functional relationships among human, bovine, rabbit, mouse, and rat proteins
(
- Contribution to journal › Article
- 1996
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Mark
Elevated levels of prothrombin activation fragment 1 + 2 in plasma from patients with heterozygous Arg506 to Gln mutation in the factor V gene (APC-resistance) and/or inherited protein S deficiency
(
- Contribution to journal › Article