Dick Heinegård (Former)
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- 2011
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Mark
Preface.
(
- Contribution to journal › Letter
- 2010
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Mark
Inflammation and joint disease: Molecules/fragments that are released upon cartilage breakdown can activate complement and provide a new diagnostics for pathological complement activation
(
- Contribution to journal › Published meeting abstract
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Mark
The NC4 domain of the cartilage-specific collagen IX is a novel complement inhibitor
(
- Contribution to journal › Published meeting abstract
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Mark
The N-terminal domain of the bone protein PRoline/arginine-rich End Leucine-rich repeat Protein (PRELP) impairs osteoclast formation by a new mechanism inhibiting NF-kappaB signaling
(
- Contribution to journal › Published meeting abstract
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Mark
Ultrastructural immunolocalization of cartilage oligomeric matrix protein (COMP) in the articular cartilage on the equine third carpal bone in trained and untrained horses
(
- Contribution to journal › Article
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Mark
A Missense Mutation in the Aggrecan C-type Lectin Domain Disrupts Extracellular Matrix Interactions and Causes Dominant Familial Osteochondritis Dissecans
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- Contribution to journal › Article
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Mark
Characterisation Of Various Human Cartilage Subtypes Using Quantitative Proteomics Reveals Unique Protein Patterns
2010) World Congress of the OsteoArthritis-Research-Society-International In Osteoarthritis and Cartilage 18. p.227-227(
- Contribution to journal › Published meeting abstract
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Mark
The Streptococcal Collagen-binding Protein CNE Specifically Interferes with alpha(V)beta(3)-mediated Cellular Interactions with Triple Helical Collagen
(
- Contribution to journal › Article
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Mark
Regulation of complement by COMP allows for a novel molecular diagnostic principle in rheumatoid arthritis.
(
- Contribution to journal › Article
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Mark
Glucosamine but not ibuprofen alters cartilage turnover in osteoarthritis patients in response to physical training
(
- Contribution to journal › Article