Dysregulation of MMP2-dependent TGF-ß2 activation impairs fibrous cap formation in type 2 diabetes-associated atherosclerosis
(2024) In Nature Communications 15. p.1-18- Abstract
Type 2 diabetes is associated with cardiovascular disease, possibly due to impaired vascular fibrous repair. Yet, the mechanisms are elusive. Here, we investigate alterations in the fibrous repair processes in type 2 diabetes atherosclerotic plaque extracellular matrix by combining multi-omics from the human Carotid Plaque Imaging Project cohort and functional studies. Plaques from type 2 diabetes patients have less collagen. Interestingly, lower levels of transforming growth factor-ß distinguish type 2 diabetes plaques and, in these patients, lower levels of fibrous repair markers are associated with cardiovascular events. Transforming growth factor-ß2 originates mostly from contractile vascular smooth muscle cells that interact with... (More)
Type 2 diabetes is associated with cardiovascular disease, possibly due to impaired vascular fibrous repair. Yet, the mechanisms are elusive. Here, we investigate alterations in the fibrous repair processes in type 2 diabetes atherosclerotic plaque extracellular matrix by combining multi-omics from the human Carotid Plaque Imaging Project cohort and functional studies. Plaques from type 2 diabetes patients have less collagen. Interestingly, lower levels of transforming growth factor-ß distinguish type 2 diabetes plaques and, in these patients, lower levels of fibrous repair markers are associated with cardiovascular events. Transforming growth factor-ß2 originates mostly from contractile vascular smooth muscle cells that interact with synthetic vascular smooth muscle cells in the cap, leading to collagen formation and vascular smooth muscle cell differentiation. This is regulated by free transforming growth factor-ß2 which is affected by hyperglycemia. Our findings underscore the importance of transforming growth factor-ß2-driven fibrous repair in type 2 diabetes as an area for future therapeutic strategies.
(Less)
- author
- organization
-
- Cardiovascular Research - Translational Studies (research group)
- EXODIAB: Excellence of Diabetes Research in Sweden
- Vascular Biology (research group)
- Diabetes - Islet Patophysiology (research group)
- Cardiovascular Research - Immunity and Atherosclerosis (research group)
- Cardiovascular Research - Matrix and Inflammation in Atherosclerosis (research group)
- Cardiovascular research - Immune regulation (research group)
- Cardiovascular Research - Cellular Metabolism and Inflammation (research group)
- Diabetes - Cardiovascular Disease (research group)
- EpiHealth: Epidemiology for Health
- WCMM-Wallenberg Centre for Molecular Medicine
- publishing date
- 2024-12
- type
- Contribution to journal
- publication status
- published
- subject
- in
- Nature Communications
- volume
- 15
- article number
- 10464
- pages
- 1 - 18
- publisher
- Nature Publishing Group
- external identifiers
-
- pmid:39653743
- scopus:85211816682
- ISSN
- 2041-1723
- DOI
- 10.1038/s41467-024-50753-8
- language
- English
- LU publication?
- yes
- additional info
- Publisher Copyright: © The Author(s) 2024.
- id
- 4fe4265c-c95c-4a3c-addf-a662f761bdc8
- date added to LUP
- 2025-01-10 14:40:13
- date last changed
- 2025-07-12 05:58:21
@article{4fe4265c-c95c-4a3c-addf-a662f761bdc8, abstract = {{<p>Type 2 diabetes is associated with cardiovascular disease, possibly due to impaired vascular fibrous repair. Yet, the mechanisms are elusive. Here, we investigate alterations in the fibrous repair processes in type 2 diabetes atherosclerotic plaque extracellular matrix by combining multi-omics from the human Carotid Plaque Imaging Project cohort and functional studies. Plaques from type 2 diabetes patients have less collagen. Interestingly, lower levels of transforming growth factor-ß distinguish type 2 diabetes plaques and, in these patients, lower levels of fibrous repair markers are associated with cardiovascular events. Transforming growth factor-ß2 originates mostly from contractile vascular smooth muscle cells that interact with synthetic vascular smooth muscle cells in the cap, leading to collagen formation and vascular smooth muscle cell differentiation. This is regulated by free transforming growth factor-ß2 which is affected by hyperglycemia. Our findings underscore the importance of transforming growth factor-ß2-driven fibrous repair in type 2 diabetes as an area for future therapeutic strategies.</p>}}, author = {{Singh, Pratibha and Sun, Jiangming and Cavalera, Michele and Al-Sharify, Dania and Matthes, Frank and Barghouth, Mohammad and Tengryd, Christoffer and Dunér, Pontus and Persson, Ana and Sundius, Lena and Nitulescu, Mihaela and Bengtsson, Eva and Rattik, Sara and Engelbertsen, Daniel and Orho-Melander, Marju and Nilsson, Jan and Monaco, Claudia and Goncalves, Isabel and Edsfeldt, Andreas}}, issn = {{2041-1723}}, language = {{eng}}, pages = {{1--18}}, publisher = {{Nature Publishing Group}}, series = {{Nature Communications}}, title = {{Dysregulation of MMP2-dependent TGF-ß2 activation impairs fibrous cap formation in type 2 diabetes-associated atherosclerosis}}, url = {{http://dx.doi.org/10.1038/s41467-024-50753-8}}, doi = {{10.1038/s41467-024-50753-8}}, volume = {{15}}, year = {{2024}}, }