Structural insights into AQP3 channel closure upon pH and redox changes reveal an autoregulatory molecular mechanism
(2025) In Nature Communications 16(1).- Abstract
Regulation of intracellular levels of reactive oxygen species (ROS) remains poorly understood. Aquaporin 3 (AQP3) facilitates the membrane transport of hydrogen peroxide (H2O2), a key ROS signaling molecule. Here we elucidate the molecular mechanism of AQP3 and show that its regulatory properties are both pH dependent and autoregulated by H2O2. Using single particle cryo-electron microscopy, we present open and closed conformations of human AQP3. At pH 8.0, the channel adopts an open state, while acidic pH or exposure to H2O2 promotes closure via a large conformational rearrangement of extracellular loop E. These findings reveal a mechanism for autoregulation of H2O2 transport and establish AQP3 as a key modulator of redox homeostasis... (More)
Regulation of intracellular levels of reactive oxygen species (ROS) remains poorly understood. Aquaporin 3 (AQP3) facilitates the membrane transport of hydrogen peroxide (H2O2), a key ROS signaling molecule. Here we elucidate the molecular mechanism of AQP3 and show that its regulatory properties are both pH dependent and autoregulated by H2O2. Using single particle cryo-electron microscopy, we present open and closed conformations of human AQP3. At pH 8.0, the channel adopts an open state, while acidic pH or exposure to H2O2 promotes closure via a large conformational rearrangement of extracellular loop E. These findings reveal a mechanism for autoregulation of H2O2 transport and establish AQP3 as a key modulator of redox homeostasis in human pancreatic β-cells.
(Less)
- author
- Huang, Peng
LU
; Venskutonytė, Raminta
LU
; Wilson, Carter J
; Bsharat, Sara
LU
; Prasad, Rashmi B
LU
; Gourdon, Pontus
LU
; Artner, Isabella
LU
; de Groot, Bert L
and Lindkvist-Petersson, Karin
LU
- organization
- publishing date
- 2025-12-22
- type
- Contribution to journal
- publication status
- published
- subject
- keywords
- Aquaporin 3/metabolism, Humans, Hydrogen-Ion Concentration, Oxidation-Reduction, Hydrogen Peroxide/metabolism, Cryoelectron Microscopy, Homeostasis, Insulin-Secreting Cells/metabolism, Reactive Oxygen Species/metabolism, Protein Conformation
- in
- Nature Communications
- volume
- 16
- issue
- 1
- article number
- 10997
- publisher
- Nature Publishing Group
- external identifiers
-
- scopus:105025412524
- pmid:41429774
- ISSN
- 2041-1723
- DOI
- 10.1038/s41467-025-67144-2
- language
- English
- LU publication?
- yes
- additional info
- © 2025. The Author(s).
- id
- a18f3002-cbd5-44dd-9b01-411b26d6ed62
- date added to LUP
- 2025-12-30 08:19:06
- date last changed
- 2026-01-14 05:46:38
@article{a18f3002-cbd5-44dd-9b01-411b26d6ed62,
abstract = {{<p>Regulation of intracellular levels of reactive oxygen species (ROS) remains poorly understood. Aquaporin 3 (AQP3) facilitates the membrane transport of hydrogen peroxide (H2O2), a key ROS signaling molecule. Here we elucidate the molecular mechanism of AQP3 and show that its regulatory properties are both pH dependent and autoregulated by H2O2. Using single particle cryo-electron microscopy, we present open and closed conformations of human AQP3. At pH 8.0, the channel adopts an open state, while acidic pH or exposure to H2O2 promotes closure via a large conformational rearrangement of extracellular loop E. These findings reveal a mechanism for autoregulation of H2O2 transport and establish AQP3 as a key modulator of redox homeostasis in human pancreatic β-cells.</p>}},
author = {{Huang, Peng and Venskutonytė, Raminta and Wilson, Carter J and Bsharat, Sara and Prasad, Rashmi B and Gourdon, Pontus and Artner, Isabella and de Groot, Bert L and Lindkvist-Petersson, Karin}},
issn = {{2041-1723}},
keywords = {{Aquaporin 3/metabolism; Humans; Hydrogen-Ion Concentration; Oxidation-Reduction; Hydrogen Peroxide/metabolism; Cryoelectron Microscopy; Homeostasis; Insulin-Secreting Cells/metabolism; Reactive Oxygen Species/metabolism; Protein Conformation}},
language = {{eng}},
month = {{12}},
number = {{1}},
publisher = {{Nature Publishing Group}},
series = {{Nature Communications}},
title = {{Structural insights into AQP3 channel closure upon pH and redox changes reveal an autoregulatory molecular mechanism}},
url = {{http://dx.doi.org/10.1038/s41467-025-67144-2}},
doi = {{10.1038/s41467-025-67144-2}},
volume = {{16}},
year = {{2025}},
}