On the transient interactions of α-synuclein in different dimensions
(2025)- Abstract
- α-Synuclein (αSyn) is a neuronal protein predominantly found in the brain, whose native function seems to be associated with vesicle trafficking. While intrinsically disordered in solution, the first ca. 100 residues adopt an amphipathic α-helical structure when the protein adsorbs onto membranes. Additionally, the aggregation of αSyn into highly ordered β-sheet rich amyloid fibrils is associated with Parkinson′s disease. The different regions of αSyn and the interactions between them have been reported to play a key role in the behaviour of the protein in solution, its membrane binding, and its aggregation into fibrils. This study employs photo-induced cross-linking of unmodified proteins (PICUP) to capture and identify the transient... (More)
- α-Synuclein (αSyn) is a neuronal protein predominantly found in the brain, whose native function seems to be associated with vesicle trafficking. While intrinsically disordered in solution, the first ca. 100 residues adopt an amphipathic α-helical structure when the protein adsorbs onto membranes. Additionally, the aggregation of αSyn into highly ordered β-sheet rich amyloid fibrils is associated with Parkinson′s disease. The different regions of αSyn and the interactions between them have been reported to play a key role in the behaviour of the protein in solution, its membrane binding, and its aggregation into fibrils. This study employs photo-induced cross-linking of unmodified proteins (PICUP) to capture and identify the transient contacts of αSyn in different conformational states: free in solution, adsorbed to membranes, and aggregated into fibrils. By using tyrosine-to-phenylalanine mutations to block the reactivity of specific amino acid residues, we establish key cross-links in each state. In solution, we identify internal contacts between the N and C termini of monomers, as well as inter-monomer contacts between C termini in oligomers. When αSyn is adsorbed to membranes, the internal cross-linking is blocked, while cross-linking between C-terminal regions persists. In fibrils, cross-linking is significantly reduced, primarily occurring between C-terminal residues of adjacent monomers. This work highlights the utility of PICUP for reporting on the transient contacts that occur on the pathways of self- and co-assembly of αSyn. (Less)
Please use this url to cite or link to this publication:
https://lup.lub.lu.se/record/f108e6f4-93ad-46ce-b171-61986bd5702d
- author
- Ortigosa-Pascual, L.
LU
; Ferrante-Carrante, N. LU ; Bernfur, K. LU ; Makasewicz, K. LU ; Sparr, E. LU and Linse, S. LU
- organization
- publishing date
- 2025
- type
- Working paper/Preprint
- publication status
- published
- subject
- pages
- 30 pages
- publisher
- bioRxiv
- DOI
- 10.1101/2025.01.29.635265
- language
- English
- LU publication?
- yes
- id
- f108e6f4-93ad-46ce-b171-61986bd5702d
- date added to LUP
- 2025-02-26 10:16:35
- date last changed
- 2025-04-04 15:10:24
@misc{f108e6f4-93ad-46ce-b171-61986bd5702d, abstract = {{α-Synuclein (αSyn) is a neuronal protein predominantly found in the brain, whose native function seems to be associated with vesicle trafficking. While intrinsically disordered in solution, the first ca. 100 residues adopt an amphipathic α-helical structure when the protein adsorbs onto membranes. Additionally, the aggregation of αSyn into highly ordered β-sheet rich amyloid fibrils is associated with Parkinson′s disease. The different regions of αSyn and the interactions between them have been reported to play a key role in the behaviour of the protein in solution, its membrane binding, and its aggregation into fibrils. This study employs photo-induced cross-linking of unmodified proteins (PICUP) to capture and identify the transient contacts of αSyn in different conformational states: free in solution, adsorbed to membranes, and aggregated into fibrils. By using tyrosine-to-phenylalanine mutations to block the reactivity of specific amino acid residues, we establish key cross-links in each state. In solution, we identify internal contacts between the N and C termini of monomers, as well as inter-monomer contacts between C termini in oligomers. When αSyn is adsorbed to membranes, the internal cross-linking is blocked, while cross-linking between C-terminal regions persists. In fibrils, cross-linking is significantly reduced, primarily occurring between C-terminal residues of adjacent monomers. This work highlights the utility of PICUP for reporting on the transient contacts that occur on the pathways of self- and co-assembly of αSyn.}}, author = {{Ortigosa-Pascual, L. and Ferrante-Carrante, N. and Bernfur, K. and Makasewicz, K. and Sparr, E. and Linse, S.}}, language = {{eng}}, note = {{Preprint}}, publisher = {{bioRxiv}}, title = {{On the transient interactions of α-synuclein in different dimensions}}, url = {{http://dx.doi.org/10.1101/2025.01.29.635265}}, doi = {{10.1101/2025.01.29.635265}}, year = {{2025}}, }