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- 1984
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Mark
Cerebral metabolic and circulatory effects of 1,1,1-trichloroethane, a neurotoxic industrial solvent - 1. Effects on local cerebral glucose consumption and blood flow during acute exposure
(
- Contribution to journal › Article
-
Mark
Influence of Severe Hypoglycemia on Brain Extracellular Calcium and Potassium Activities, Energy, and Phospholipid Metabolism
(
- Contribution to journal › Article
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Mark
Models for studying long‐term recovery following forebrain ischemia in the rat. 2. A 2‐vessel occlusion model
(
- Contribution to journal › Scientific review
-
Mark
The distribution of hypoglycemic brain damage
(
- Contribution to journal › Article
- 1983
-
Mark
Prostaglandins and the cerebral circulation.
(
- Contribution to journal › Article
- 1982
-
Mark
Influence of severe hypoglycemia on mitochondrial and plasma membrane function in rat brain
(
- Contribution to journal › Article
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Mark
Brain cortical fatty acids and phospholipids during and following complete and severe incomplete ischemia
(
- Contribution to journal › Article
- 1981
-
Mark
Endogenous substrates utilized by rat brain in severe insulin-induced hypoglycemia
(
- Contribution to journal › Article
-
Mark
Hypoglycemic brain injury: phospholipids, free fatty acids, and cyclic nucleotides in the cerebellum of the rat after 30 and 60 minutes of severe insulin-induced hypoglycemia
(
- Contribution to journal › Article
-
Mark
Hypoglycemic brain injury: metabolic and structural findings in rat cerebellar cortex during profound insulin-induced hypoglycemia and in the recovery period following glucose administration
(
- Contribution to journal › Article
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Mark
Cerebrovascular response during and following severe insulin-induced hypoglycemia: CO2-sensitivity, autoregulation, and influence of prostaglandin synthesis inhibition
(
- Contribution to journal › Article
-
Mark
Reply to the remarks by J. B. Brierley and A. W. Brown
(
- Contribution to journal › Letter
- 1980
-
Mark
Hypoglycemic brain injury. II. Electron-microscopic findings in rat cerebral cortical neurons during profound insulin-induced hypoglycemia and in the recovery period following glucose administration
(
- Contribution to journal › Article
-
Mark
Severe hypoglycemia leads to accumulation of arachidonic acid in brain tissue
(
- Contribution to journal › Article
-
Mark
Local cerebral blood flow in the rat during severe hypoglycemia, and in the recovery period following glucose injection
(
- Contribution to journal › Article