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- 2023
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Mark
Measures of cortical microstructure are linked to amyloid pathology in Alzheimer's disease
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- Contribution to journal › Article
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Mark
MRI BrainAGE demonstrates increased brain aging in systemic lupus erythematosus patients
(
- Contribution to journal › Article
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Mark
The α-synuclein PET tracer [18F] ACI-12589 distinguishes multiple system atrophy from other neurodegenerative diseases
(
- Contribution to journal › Article
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Mark
Effects of Brain Pathologies on Spatiotemporal Gait Parameters in Patients with Mild Cognitive Impairment
(
- Contribution to journal › Article
- 2022
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Mark
Inter-modality assessment of medial temporal lobe atrophy in a non-demented population : application of a visual rating scale template across radiologists with varying clinical experience
(
- Contribution to journal › Article
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Mark
Amyloid-associated increases in soluble tau relate to tau aggregation rates and cognitive decline in early Alzheimer's disease
(
- Contribution to journal › Article
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Mark
Plasma and cerebrospinal fluid neurofilament light concentrations reflect neuronal damage in systemic lupus Erythematosus
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- Contribution to journal › Article
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Mark
Tau pathology mediates age effects on medial temporal lobe structure
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- Contribution to journal › Article
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Mark
Higher levels of myelin are associated with higher resistance against tau pathology in Alzheimer’s disease
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- Contribution to journal › Article
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Mark
Hippocampal subregional thinning related to tau pathology in early stages of Alzheimer’s disease
(
- Contribution to journal › Debate/Note/Editorial
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Mark
Age-related tau-PET uptake and its downstream effects extend beyond the medial temporal lobe in cognitively normal older adults
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- Contribution to journal › Debate/Note/Editorial
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Mark
A biomarker profile of elevated CSF p-tau with normal tau PET is associated with increased tau accumulation rates on PET in early Alzheimer’s disease
(
- Contribution to journal › Debate/Note/Editorial
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Mark
The mean diffusion propagator model revealed cortical microstructural changes associated with both amyloid-β and tau pathology and astroglial activation
(
- Contribution to journal › Debate/Note/Editorial
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Mark
Earlier Alzheimer's disease onset is associated with a shift of tau pathology towards brain hubs which facilitates tau spreading
(
- Contribution to journal › Debate/Note/Editorial
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Mark
Amyloid-associated increases in soluble tau is a key driver in accumulation of tau aggregates and cognitive decline in early Alzheimer
(
- Contribution to journal › Debate/Note/Editorial