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- 2004
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Mark
Glucose but Not Lactate in Combination With Acidosis Aggravates Ischemic Neuronal Death In Vitro.
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- Contribution to journal › Article
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Mark
Protein Kinase C-γ and Calcium/Calmodulin-Dependent Protein Kinase II-α Are Persistently Translocated to Cell Membranes of the Rat Brain during and after Middle Cerebral Artery Occlusion
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- Contribution to journal › Article
- 2003
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Mark
Mineralocorticoid receptor expression and increased survival following neuronal injury.
(
- Contribution to journal › Article
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Mark
Powerful cyclosporin inhibition of calcium-induced permeability transition in brain mitochondria.
(
- Contribution to journal › Article
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Mark
Brain damage in a mouse model of global cerebral ischemia. Effect of NMDA receptor blockade.
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- Contribution to journal › Article
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Mark
Flow cytometric analysis of mitochondria from CA1 and CA3 regions of rat hippocampus reveals differences in permeability transition pore activation.
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- Contribution to journal › Article
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Mark
Structural and functional damage sustained by mitochondria after traumatic brain injury in the rat: Evidence for differentially sensitive populations in the cortex and hippocampus
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- Contribution to journal › Article
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Mark
Mouse hippocampal organotypic tissue cultures exposed to in vitro "ischemia" show selective and delayed CA1 damage that is aggravated by glucose
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- Contribution to journal › Article
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Mark
Cyclosporin A prevents calpain activation despite increased intracellular calcium concentrations, as well as translocation of apoptosis-inducing factor, cytochrome c and caspase-3 activation in neurons exposed to transient hypoglycemia.
(
- Contribution to journal › Article
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Mark
Uncoupling protein-2 prevents neuronal death and diminishes brain dysfunction after stroke and brain trauma.
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- Contribution to journal › Article