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- 2021
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Mark
A-769662 inhibits adipocyte glucose uptake in an AMPK-independent manner
(
- Contribution to journal › Article
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Mark
Investigation of the specificity and mechanism of action of the ULK1/AMPK inhibitor SBI-0206965
(
- Contribution to journal › Article
- 2019
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Mark
Chemical genetic screen identifies Gapex-5/GAPVD1 and STBD1 as novel AMPK substrates
(
- Contribution to journal › Article
- 2018
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Mark
The Salt-Inducible Kinases : Emerging Metabolic Regulators
(
- Contribution to journal › Scientific review
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Mark
AMPK activation by A-769662 and 991 does not affect catecholamine-induced lipolysis in human adipocytes
(
- Contribution to journal › Article
- 2017
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Mark
GFAT1 phosphorylation by AMPK promotes VEGF-induced angiogenesis
(
- Contribution to journal › Article
- 2016
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Mark
SIKs control osteocyte responses to parathyroid hormone
(
- Contribution to journal › Article
- 2015
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Mark
SIK2 regulates CRTCs, HDAC4 and glucose uptake in adipocytes
(
- Contribution to journal › Article
- 2014
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Mark
The LKB1-salt-inducible kinase pathway functions as a key gluconeogenic suppressor in the liver.
(
- Contribution to journal › Article
- 2013
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Mark
AMPK alpha 1 Regulates Macrophage Skewing at the Time of Resolution of Inflammation during Skeletal Muscle Regeneration
(
- Contribution to journal › Article
- 2012
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Mark
The AMPK-related kinase SIK2 is regulated by cAMP via phosphorylation at Ser(358) in adipocytes
(
- Contribution to journal › Article
- 2011
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Mark
Regulation of AMP-activated protein kinase by LKB1 and CaMKK in adipocytes.
(
- Contribution to journal › Article
- 2008
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Mark
Use of Akt inhibitor and a drug-resistant mutant validates a critical role for protein kinase B/Akt in the insulin-dependent regulation of glucose and system A amino acid uptake
(
- Contribution to journal › Article
- 2007
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Mark
Mechanism of action of A-769662, a valuable tool for activation of AMP-activated protein kinase
(
- Contribution to journal › Article
- 2004
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Mark
Activity of LKB1 and AMPK-related kinases in skeletal muscle: effects of contraction, phenformin, and AICAR
(
- Contribution to journal › Article