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- 2024
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Mark
G protein-coupled estrogen receptor (GPER)/GPR30 forms a complex with the β1-adrenergic receptor, a membrane-associated guanylate kinase (MAGUK) scaffold protein, and protein kinase A anchoring protein (AKAP) 5 in MCF7 breast cancer cells
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- Contribution to journal › Article
- 2021
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Mark
Ligand-independent G protein-coupled estrogen receptor/G protein-coupled receptor 30 activity : Lack of receptor-dependent effects of G-1 and 17b-estradiol
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- Contribution to journal › Article
- 2019
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Mark
Human G protein-coupled Receptor 30 (GPR30) is N -glycosylated and N-terminal Domain Asparagine 44 is Required for Receptor Structure and Activity
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- Contribution to journal › Article
- 2018
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Mark
Roles of PDZ-dependent Interactions and N-glycosylation in G Protein-coupled Estrogen Receptor 1 (GPER1)/GPR30-mediated Stimulation of ERK1/2 Activity
(
- Contribution to journal › Published meeting abstract
- 2017
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Mark
G protein-coupled Estrogen Receptor 1 (GPER1)/GPR30 Increases ERK1/2 Activity Through PDZ-dependent and -independent Mechanisms
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- Contribution to journal › Article
- 2016
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Mark
G protein-coupled Receptor 30 (GPR30) PDZ-dependently and Constitutively Increases ERK1/2 Signaling Through Calcineurin and Kinase Suppressor of Ras 2 (KSR2)
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- Contribution to journal › Published meeting abstract
- 2015
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Mark
G Protein-Coupled Estrogen Receptor 1 Mediates Acute Estrogen-Induced Cardioprotection via MEK/ERK/GSK-3β Pathway after Ischemia/Reperfusion.
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- Contribution to journal › Article
- 2014
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Mark
G Protein-coupled Receptor 30 (GPR30) Forms a Plasma Membrane Complex With Membrane-associated Guanylate Kinases (MAGUKs) and AKAP5 That Constitutively Inhibits cAMP Production.
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- Contribution to journal › Article
- 2013
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Mark
G protein-coupled estrogen receptor is apoptotic and correlates with increased distant disease-free survival of estrogen receptor-positive breast cancer patients.
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- Contribution to journal › Article
- 2012
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Mark
The G protein-coupled oestrogen receptor 1 agonist G-1 disrupts endothelial cell microtubule structure in a receptor-independent manner.
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- Contribution to journal › Article
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Mark
17 beta-Estradiol induces nongenomic effects in renal intercalated cells through G protein-coupled estrogen receptor 1
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- Contribution to journal › Article
- 2011
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Mark
G protein-coupled oestrogen receptor 1 (GPER1)/GPR30: a new player in cardiovascular and metabolic oestrogenic signalling
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- Contribution to journal › Scientific review
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Mark
G Protein-Coupled Estrogen Receptor 1 (GPER1)/GPR30 Localizes in the Plasma Membrane and Trafficks Intracellularly on Cytokeratin Intermediate Filaments.
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- Contribution to journal › Article
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Mark
The GPER1 Agonist G-1 Attenuates Endothelial Cell Proliferation by Inhibiting DNA Synthesis and Accumulating Cells in the S and G2 Phases of the Cell Cycle.
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- Contribution to journal › Article
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Mark
P4-09-02: G Protein-Coupled Estrogen Receptor 1 Positively Correlates with Estrogen Receptor a Expression and Increased Distant Disease-Free Survival of Breast Cancer Patients.
(
- Contribution to journal › Published meeting abstract
- 2010
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Mark
ADP mediates inhibition of insulin secretion by activation of P2Y13 receptors in mice.
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- Contribution to journal › Article
- 2009
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Mark
Activation Of The Putative Estrogen Receptor Gpr30 Attenuates Endothelial Cell Dna Synthesis
2009) 14th Annual Meeting of the European-Council-for-Cardiovascular-Research In Hypertension 54(5). p.1186-1186(
- Contribution to journal › Published meeting abstract
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Mark
Palmitate-stimulated hormone secretion in relation to GPR40 expression in pancreatic islets of spontaneous obesity and type 2 diabetes in rats
2009) 45th Annual Meeting of the European-Association-for-the-Study-of-Diabetes In Diabetologia 52(Suppl. 1). p.405-405(
- Contribution to journal › Published meeting abstract
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Mark
The role of the G protein-coupled receptor GPR30 in the effects of estrogen in ovariectomized mice
(
- Contribution to journal › Article
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Mark
Deletion of the G protein-coupled Receptor GPR30 Impairs Glucose Tolerance, Reduces Bone Growth, Increases Blood Pressure, and Eliminates Estradiol-stimulated Insulin Release in Female Mice.
(
- Contribution to journal › Article