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- 2018
-
Mark
Vitamin K–Dependent Protein S : Beyond the Protein C Pathway
(
- Contribution to journal › Article
- 2017
-
Mark
Novel insights into the regulation of coagulation by factor V isoforms, tissue factor pathway inhibitorα, and protein S
(
- Contribution to journal › Article
- 2016
-
Mark
Pro- and anticoagulant properties of factor V in pathogenesis of thrombosis and bleeding disorders
(
- Contribution to journal › Article
- 2014
-
Mark
Vitamin K-dependent protein S - studies of synthesis, structure and function
(
- Thesis › Doctoral thesis (compilation)
- 2012
-
Mark
Factor VIIa binding to endothelial cell protein C receptor: Differences between mouse and human systems
(
- Contribution to journal › Article
- 2011
-
Mark
C4b-Binding Protein: A Forgotten Factor in Thrombosis and Hemostasis
(
- Contribution to journal › Article
- 2010
-
Mark
Further insight into the roles of the glycans attached to human blood protein C inhibitor
(
- Contribution to journal › Article
-
Mark
Novel APC-cleavage sites in FVa providing insights into mechanisms of action of APC and its cofactor protein S.
(
- Contribution to journal › Article
- 2006
-
Mark
Coagulation disturbances in the critically ill patient with special reference to prediction of outcome
2006)(
- Thesis › Doctoral thesis (compilation)
- 2005
-
Mark
Regulation of Blood Coagulation by the Protein C Anticoagulant Pathway. Novel Insights Into Structure-Function Relationships and Molecular Recognition.
(
- Contribution to journal › Scientific review
-
Mark
Selective modulation of protein C affinity for EPCR and phospholipids by Gla domain mutation
(
- Contribution to journal › Article
- 2004
-
Mark
Altered inactivation pathway of factor Va by activated protein C in the presence of heparin
(
- Contribution to journal › Article
-
Mark
Antithrombotic Effects of Activated Protein C and Heparins in Deep Arterial Injury
2004)(
- Thesis › Doctoral thesis (compilation)
- 2001
-
Mark
Anti- and procoagulant activities in factor VII-deficient subjects
(
- Contribution to journal › Article
-
Mark
Screening the molecular surface of human anticoagulant protein C: a search for interaction sites
(
- Contribution to journal › Article
- 2000
-
Mark
Interaction of Triglyceride-rich Lipoproteins with Platelets and Vitamin K-dependent Coagulation Factors
2000)(
- Thesis › Doctoral thesis (compilation)
- 1999
-
Mark
Probing the activation of protein C by the thrombin-thrombomodulin complex using structural analysis, site-directed mutagenesis, and computer modeling
(
- Contribution to journal › Article
-
Mark
Anticoagulant protein C--structural and functional studies
1999)(
- Thesis › Doctoral thesis (compilation)
-
Mark
Pro- and anticoagulant mechanisms in coronary artery disease. Clinical studies on factor VII and resistance to activated protein C
1999)(
- Thesis › Doctoral thesis (compilation)
- 1998
-
Mark
Resistance to activated protein C, highly prevalent amongst users of oral contraceptives with venous thromboembolism
(
- Contribution to journal › Article
- 1997
-
Mark
Resistance to activated protein C a novel risk factor for venous thrombosis
1997)(
- Thesis › Doctoral thesis (compilation)
-
Mark
A novel thrombomodulin gene mutation in a patient suffering from sagittal sinus thrombosis
(
- Contribution to journal › Article
-
Mark
The 20210 A allele of the prothrombin gene is a common risk factor among Swedish outpatients with verified deep venous thrombosis
(
- Contribution to journal › Debate/Note/Editorial
-
Mark
Activated protein C resistance caused by a common factor V mutation has a single origin
(
- Contribution to journal › Article
-
Mark
The factor VR506Q mutation causing APC resistance is highly prevalent amongst unselected outpatients with clinically suspected deep venous thrombosis
(
- Contribution to journal › Article
-
Mark
A common thrombomodulin amino acid dimorphism is associated with myocardial infarction
(
- Contribution to journal › Article
-
Mark
Activated protein C resistance due to a common factor V gene mutation is a major risk factor for venous thrombosis
(
- Contribution to journal › Article
-
Mark
The factor VR5O6Q mutation causing APC resistance is highly prevalent amongst unselected outpatients with clinically suspected deep venous thrombosis
(
- Contribution to journal › Article
-
Mark
Activated protein C resistance : Clinical implications
(
- Contribution to journal › Scientific review
- 1996
-
Mark
Prevalence of factor V gene mutation amongst myocardial infarction patients and healthy controls is higher in Sweden than in other countries
(
- Contribution to journal › Article
-
Mark
Resistance to activated protein C, the FV:Q506 allele, and venous thrombosis
(
- Contribution to journal › Scientific review
-
Mark
High prevalence of the FVR506Q mutation causing APC resistance in a region of southern Sweden with a high incidence of venous thrombosis.
(
- Contribution to journal › Letter
-
Mark
Activated protein C resistance as a basis for venous thrombosis.
(
- Contribution to journal › Article
-
Mark
Structural investigation of the alpha-1-antichymotrypsin : Prostate-specific antigen complex by comparative model building
(
- Contribution to journal › Article
- 1995
-
Mark
Effects of hemodialysis and longterm erythropoietin treatment on protein C, and on free and total protein S
(
- Contribution to journal › Article
-
Mark
Resistance to activated protein C caused by a factor V gene mutation
(
- Contribution to journal › Article
-
Mark
Resistens mot aktiverat protein C. Vanlig genetisk riskfaktor för venös trombos
(
- Contribution to journal › Article
-
Mark
Resistance to activated protein C as an additional genetic risk factor in hereditary deficiency of protein S
(
- Contribution to journal › Article
-
Mark
The gene encoding vitamin K-dependent anticoagulant protein C is expressed in human male reproductive tissues
(
- Contribution to journal › Article
- 1994
-
Mark
Identification of the same factor V gene mutation in 47 out of 50 thrombosis-prone families with inherited resistance to activated protein C
(
- Contribution to journal › Article
- 1983
-
Mark
The effects of plasmin and protein Ca on factor VIII:C and VIII:CAg
(
- Contribution to journal › Article