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- 2004
-
Mark
ATP-sensitive K+ channel-dependent regulation of glucagon release and electrical actiflty by glucose in wild-type and SUR1(-/-) mouse alpha-cells
(
- Chapter in Book/Report/Conference proceeding › Paper in conference proceeding
- 2003
-
Mark
Imidazoline NNC77-0074 stimulates insulin secretion and inhibits glucagon release by control of Ca2+-dependent exocytosis in pancreatic alpha- and beta-cells
(
- Contribution to journal › Article
-
Mark
Phosphatidylinositol 4-kinase serves as a metabolic sensor and regulates priming of secretory granules in pancreatic beta cells
(
- Contribution to journal › Article
- 2002
-
Mark
Nateglinide, but not repaglinide, stimulates growth hormone release in rat pituitary cells by inhibition of K+ channels and stimulation of cyclic AMP-dependent exocytosis
(
- Contribution to journal › Article
- 2000
-
Mark
Tolbutamide stimulates exocytosis of glucagon by inhibition of a mitochondrial-like ATP-sensitive K+ (KATP) conductance in rat pancreatic A-cells
(
- Contribution to journal › Article
- 1999
-
Mark
The stimulatory action of tolbutamide on Ca2+-dependent exocytosis in pancreatic beta cells is mediated by a 65-kDa mdr-like P-glycoprotein
(
- Contribution to journal › Article
- 1997
-
Mark
Adrenaline stimulates glucagon secretion in pancreatic A-cells by increasing the Ca2+ current and the number of granules close to the L-type Ca2+ channels
(
- Contribution to journal › Article
- 1995
-
Mark
Co-localization of L-type Ca2+ channels and insulin-containing secretory granules and its significance for the initiation of exocytosis in mouse pancreatic B-cells
(
- Contribution to journal › Article
-
Mark
Glucagon-like peptide I increases cytoplasmic calcium in insulin-secreting beta TC3-cells by enhancement of intracellular calcium mobilization
(
- Contribution to journal › Article
- 1994
-
Mark
Ion channels, electrical activity and insulin secretion
(
- Contribution to journal › Article
- 1993
-
Mark
Exocytosis elicited by action potentials and voltage-clamp calcium currents in individual pancreatic B-cells
(
- Contribution to journal › Article