INGA NEWIE (Former)

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2019
Mark HER2 and p95HER2 differentially regulate miRNA expression in MCF-7 breast cancer cells and downregulate MYB proteins through miR-221/222 and miR-503
Gorbatenko, Andrej ; Søkilde, Rolf ^LU ; Sorensen, Ester E. ; Newie, Inga ^LU ; Persson, Helena ^LU ; Morancho, Beatriz ; Arribas, Joaquin ; Litman, Thomas ; Rovira, Carlos ^LU and Pedersen, Stine Falsig (2019) In Scientific Reports 9(1).
- Contribution to journal › Article
2016
Mark HER2-encoded mir-4728 forms a receptor-independent circuit with miR-21-5p through the non-canonical poly(A) polymerase PAPD5
Newie, Inga ^LU ; Søkilde, Rolf ^LU ; Persson, Helena ^LU ; Jacomasso, Thiago ^LU ; Gorbatenko, Andrej ; Borg, Åke ^LU ; De Hoon, Michiel ; Pedersen, Stine F. and Rovira, Carlos ^LU (2016) In Scientific Reports 6.
- Contribution to journal › Article
2015
Mark MicroRNAs in HER2-Amplified Breast Cancer
Newie, Inga ^LU (2015) In Lund University Faculty of Medicine Doctoral Dissertation Series 2015:87.
- Thesis › Doctoral thesis (compilation)
Mark Passenger strand loading in overexpression experiments using microRNA mimics.
Sökilde, Rolf ^LU ; Newie, Inga ^LU ; Persson, Helena ^LU ; Borg, Åke ^LU and Rovira, Carlos ^LU (2015) In RNA Biology 12(8). p.787-791
- Contribution to journal › Article
2014
Mark The HER2-Encoded miR-4728-3p Regulates ESR1 through a Non-Canonical Internal Seed Interaction.
Newie, Inga ^LU ; Sökilde, Rolf ^LU ; Persson, Helena ^LU ; Grabau, Dorthe ^LU ; Rego, Natalia ; Kvist, Anders ^LU ; von Stedingk, Kristoffer ^LU ; Axelson, Håkan ^LU ; Borg, Åke ^LU and Vallon-Christersson, Johan ^LU , et al. (2014) In PLoS ONE 9(5).
- Contribution to journal › Article
Mark PAPD5-mediated 3' adenylation and subsequent degradation of miR-21 is disrupted in proliferative disease.
Boele, Joost ; Persson, Helena ^LU ; Shin, Jay W ; Ishizu, Yuri ; Newie, Inga ^LU ; Sökilde, Rolf ^LU ; Hawkins, Shannon M ; Coarfa, Cristian ; Ikeda, Kazuhiro and Takayama, Ken-Ichi , et al. (2014) In Proceedings of the National Academy of Sciences 111(31). p.11467-11472
- Contribution to journal › Article