Advanced

Endogenous IFN-β signaling exerts anti-inflammatory actions in experimentally induced focal cerebral ischemia.

Inacio, Ana LU ; Liu, Yawei ; Clausen, Bettina H ; Svensson, Martina LU ; Kucharz, Krzysztof LU ; Yang, Yiyi LU ; Stankovich, Totte ; Khorooshi, Reza ; Lambertsen, Kate L and Issazadeh-Navikas, Shohreh , et al. (2015) In Journal of Neuroinflammation 12(1).
Abstract
Interferon (IFN)-β exerts anti-inflammatory effects, coupled to remarkable neurological improvements in multiple sclerosis, a neuroinflammatory condition of the central nervous system. Analogously, it has been hypothesized that IFN-β, by limiting inflammation, decreases neuronal death and promotes functional recovery after stroke. However, the core actions of endogenous IFN-β signaling in stroke are unclear.
Please use this url to cite or link to this publication:
author
, et al. (More)
(Less)
organization
publishing date
type
Contribution to journal
publication status
published
subject
in
Journal of Neuroinflammation
volume
12
issue
1
article number
211
publisher
BioMed Central
external identifiers
  • pmid:26581581
  • wos:000364902200002
  • scopus:84960370618
  • pmid:26581581
ISSN
1742-2094
DOI
10.1186/s12974-015-0427-0
language
English
LU publication?
yes
additional info
The information about affiliations in this record was updated in December 2015. The record was previously connected to the following departments: Neuroinflammation (013210006), Faculty of Medicine (000022000), Laboratory for Experimental Brain Research (013041000)
id
ce6c86eb-b4e9-4880-b016-9701b7927404 (old id 8235218)
alternative location
http://www.ncbi.nlm.nih.gov/pubmed/26581581?dopt=Abstract
date added to LUP
2016-04-01 13:42:15
date last changed
2020-03-24 03:24:24
@article{ce6c86eb-b4e9-4880-b016-9701b7927404,
  abstract     = {Interferon (IFN)-β exerts anti-inflammatory effects, coupled to remarkable neurological improvements in multiple sclerosis, a neuroinflammatory condition of the central nervous system. Analogously, it has been hypothesized that IFN-β, by limiting inflammation, decreases neuronal death and promotes functional recovery after stroke. However, the core actions of endogenous IFN-β signaling in stroke are unclear.},
  author       = {Inacio, Ana and Liu, Yawei and Clausen, Bettina H and Svensson, Martina and Kucharz, Krzysztof and Yang, Yiyi and Stankovich, Totte and Khorooshi, Reza and Lambertsen, Kate L and Issazadeh-Navikas, Shohreh and Deierborg, Tomas},
  issn         = {1742-2094},
  language     = {eng},
  number       = {1},
  publisher    = {BioMed Central},
  series       = {Journal of Neuroinflammation},
  title        = {Endogenous IFN-β signaling exerts anti-inflammatory actions in experimentally induced focal cerebral ischemia.},
  url          = {http://dx.doi.org/10.1186/s12974-015-0427-0},
  doi          = {10.1186/s12974-015-0427-0},
  volume       = {12},
  year         = {2015},
}