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- 2002
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Mark
Maintenance of susceptibility to neurodegeneration following intrastriatal injections of quinolinic acid in a new transgenic mouse model of Huntington's disease.
(
- Contribution to journal › Article
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Mark
Increased Sensitivity to N-Methyl-D-Aspartate Receptor-Mediated Excitotoxicity in a Mouse Model of Huntington's Disease.
(
- Contribution to journal › Article
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Mark
Deep brain stimulation of subthalamic neurons increases striatal dopamine metabolism and induces contralateral circling in freely moving 6-hydroxydopamine-lesioned rats
(
- Contribution to journal › Article
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Mark
Papel del poro de permeabilidad transitoria mitocondrial en los procesos neurodegenerativos
(
- Contribution to journal › Article
- 2001
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Mark
Delayed infusion of GDNF promotes recovery of motor function in the partial lesion model of Parkinson's disease
(
- Contribution to journal › Article
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Mark
Resistance to NMDA toxicity correlates with appearance of nuclear inclusions, behavioural deficits and changes in calcium homeostasis in mice transgenic for exon 1 of the huntington gene
(
- Contribution to journal › Article
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Mark
Mice transgenic for exon 1 of the Huntington's disease gene display reduced striatal sensitivity to neurotoxicity induced by dopamine and 6-hydroxydopamine
(
- Contribution to journal › Article
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Mark
Partial resistance to malonate-induced striatal cell death in transgenic mouse models of Huntington's disease is dependent on age and CAG repeat length
(
- Contribution to journal › Article
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Mark
Adult rabbit retinal transplants
(
- Contribution to journal › Article
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Mark
Restorative and neuroprotective treatment strategies for Parkinson’s disease: tudies on fetal dopamine cell transplants and GDNF delivery by intracerebral injections and recombinant AAV vectors.
2001)(
- Thesis › Doctoral thesis (compilation)