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- 2006
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Mark
A new arthritis therapy with oxidative burst inducers
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- Contribution to journal › Article
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Mark
Side-chain and backbone amide bond requirements for glycopeptide stimulation of T-cells obtained in a mouse model for rheumatoid arthritis
(
- Contribution to journal › Article
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Mark
Pristane, a non-antigenic adjuvant, induces MHC class II-restricted, arthritogenic T cells in the rat.
(
- Contribution to journal › Article
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Mark
Therapeutic Vaccination of Active Arthritis with a Glycosylated Collagen Type II Peptide in Complex with MHC Class II Molecules.
(
- Contribution to journal › Article
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Mark
Identification of collagen-induced arthritis loci in aged multiparous female mice.
(
- Contribution to journal › Article
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Mark
Backcross and partial advanced intercross analysis of nonobese diabetic gene-mediated effects on collagen-induced arthritis reveals an interactive effect by two major loci.
(
- Contribution to journal › Article
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Mark
Integrated bioprocess for the production and isolation of urokinase from animal cell culture using supermacroporous cryogel matrices
(
- Contribution to journal › Article
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Mark
The impact of glycosylation on HLA-DR1-restricted T cell recognition of type II collagen in a mouse model
(
- Contribution to journal › Article
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Mark
Identification of Genetic Regions of Importance for Reproductive Performance in Female Mice.
(
- Contribution to journal › Article
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Mark
Association between protein tyrosine phosphatase 22 variant R620W in conjunction with the HLA-DRB1 shared epitope and humoral autoimmunity to an immunodominant epitope of cartilage-specific type II collagen in early rheumatoid arthritis
(
- Contribution to journal › Article
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Mark
Modulation of granulocyte-endothelium interactions by antileukoproteinase: inhibition of anti-type II collagen antibody-induced leukocyte attachment to the synovial endothelium
(
- Contribution to journal › Article
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Mark
Advanced intercross line mapping of Eae5 reveals Ncf-1 and CLDN4 as candidate genes for experimental autoimmune encephalomyelitis
(
- Contribution to journal › Article
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Mark
Inhibition of macropinocytosis blocks antigen presentation of type II collagen in vitro and in vivo in HLA-DR1 transgenic mice
(
- Contribution to journal › Article
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Mark
Identification of susceptibility loci for skin disease in a murine psoriasis model
(
- Contribution to journal › Article
- 2005
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Mark
A genetic basis for shared autoimmunity in mouse models.
(
- Contribution to journal › Scientific review
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Mark
Ncf1 (p47phox) polymorphism determines oxidative burst and the severity of arthritis in rats and mice.
(
- Contribution to journal › Article
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Mark
A genetic contamination in MHC-congenic mouse strains reveals a locus on chromosome 10 that determines autoimmunity and arthritis susceptibility.
(
- Contribution to journal › Article
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Mark
Efficient promotion of collagen antibody induced arthritis (CAIA) using four monoclonal antibodies specific for the major epitopes recognized in both collagen induced arthritis and rheumatoid arthritis.
(
- Contribution to journal › Article
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Mark
Induction of a B-cell-dependent chronic arthritis with glucose-6-phosphate isomerase.
(
- Contribution to journal › Article
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Mark
Identification of the minimal glycopeptide core recognized by T cells in a model for rheumatoid arthritis
(
- Contribution to journal › Article
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Mark
Intrinsic Tolerance in Autologous Collagen-Induced Arthritis Is Generated by CD152-Dependent CD4+ Suppressor Cells.
(
- Contribution to journal › Article
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Mark
Chronic development of collagen-induced arthritis is associated with arthritogenic antibodies against specific epitopes on type II collagen.
(
- Contribution to journal › Article
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Mark
Contrasting roles of plasminogen deficiency in different rheumatoid arthritis models
(
- Contribution to journal › Article
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Mark
Stromal cells and osteoclasts are responsible for exacerbated collagen-induced arthritis in interferon-beta-deficient mice.
(
- Contribution to journal › Article
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Mark
Humoral immune response to citrullinated collagen type II determinants in early rheumatoid arthritis
(
- Contribution to journal › Article
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Mark
The plasminogen activator/plasmin system is essential for development of the joint inflammatory phase of collagen type II-induced arthritis
(
- Contribution to journal › Article
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Mark
Methotrexate ameliorates T cell dependent autoimmune arthritis and encephalomyelitis but not antibody induced or fibroblast induced arthritis
(
- Contribution to journal › Article
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Mark
Genetic Interactions in Eae2 Control Collagen-Induced Arthritis and the CD4+/CD8+ T Cell Ratio.
(
- Contribution to journal › Article
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Mark
Influence on spontaneous tissue inflammation by the major histocompatibility complex region in the nonobese diabetic mouse.
(
- Contribution to journal › Article
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Mark
The major T cell epitope on type II collagen is glycosylated in normal cartilage but modified by arthritis in both rats and humans
(
- Contribution to journal › Article
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Mark
Arthritogenic anti-type II collagen antibodies are pathogenic for cartilage-derived chondrocytes independent of inflammatory cells
(
- Contribution to journal › Article
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Mark
Affinity binding of cells to cryogel adsorbents with immobilized specific ligands: effect of ligand coupling and matrix architecture
(
- Contribution to journal › Article
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Mark
Perforin deficiency attenuates collagen-induced arthritis
(
- Contribution to journal › Article
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Mark
Gene expression profiling of arthritis using a QTL chip reveals a complex gene regulation of the Cia5 region in mice.
(
- Contribution to journal › Article
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Mark
Identification of epistasis through a partial advanced intercross reveals three arthritis loci within the Cia5 QTL in mice.
(
- Contribution to journal › Article
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Mark
Destructive effects of murine arthritogenic antibodies to type II collagen on cartilage explants in vitro
(
- Contribution to journal › Article
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Mark
Bb2Bb3 regulation of murine Lyme arthritis is distinct from Ncf1 and independent of the phagocyte nicotinamide adenine dinucleotide phosphate oxidase
(
- Contribution to journal › Article
- 2004
-
Mark
Cysteine proteases in Langerhans cells limits presentation of cartilage derived type II collagen for autoreactive T cells.
(
- Contribution to journal › Article
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Mark
Critical role of the major histocompatibility complex and IL-10 in matrilin-1-induced relapsing polychondritis in mice
(
- Contribution to journal › Article
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Mark
Oligoclonal IgG bands synthesized in the central nervous system are present in rats with experimental autoimmune encephalomyelitis.
(
- Contribution to journal › Article
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Mark
Collagen type II (CII)-specific antibodies induce arthritis in the absence of T or B cells but the arthritis progression is enhanced by CII-reactive T cells.
(
- Contribution to journal › Article
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Mark
A Transient Post-Translationally Modified Form of Cartilage Type II Collagen Is Ignored by Self-Reactive T Cells.
(
- Contribution to journal › Article
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Mark
Complement activation by both classical and alternative pathways is critical for the effector phase of arthritis
(
- Contribution to journal › Article
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Mark
Enhanced autoimmunity, arthritis, and encephalomyelitis in mice with a reduced oxidative burst due to a mutation in the Ncf1 gene.
(
- Contribution to journal › Article
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Mark
Relapsing polychondritis, induced in mice with matrilin 1, is an antibody- and complement-dependent disease
(
- Contribution to journal › Article
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Mark
Analysis of autoreactive T cells associated with murine collagen-induced arthritis using peptide-MHC multimers.
(
- Contribution to journal › Article
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Mark
Tracking of proinflammatory collagen-specific T cells in early and late collagen-induced arthritis in humanized mice
(
- Contribution to journal › Article
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Mark
Inconsistent susceptibility to autoimmunity in inbred LEW rats is due to genetic crossbreeding involving segregation of the arthritis-regulating gene Ncf1.
(
- Contribution to journal › Article
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Mark
Antileukoproteinase - Modulation of neutrophil function and therapeutic effects on anti-type II collagen antibody-induced arthritis
(
- Contribution to journal › Article
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Mark
CD1-dependent regulation of chronic central nervous system inflammation in experimental autoimmune encephalomyelitis.
(
- Contribution to journal › Article