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- 2017
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Mark
Molecular mechanisms underlying lineage bias in aging hematopoiesis
(
- Contribution to journal › Scientific review
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Mark
Critical Modulation of Hematopoietic Lineage Fate by Hepatic Leukemia Factor
(
- Contribution to journal › Article
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Mark
The slippery slope of hematopoietic stem cell aging
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- Contribution to journal › Article
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Mark
Hepatic Leukemia Factor Maintains Quiescence of Hematopoietic Stem Cells and Protects the Stem Cell Pool during Regeneration
(
- Contribution to journal › Article
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Mark
ZFP521 regulates murine hematopoietic stem cell function and facilitates MLL-AF9 leukemogenesis in mouse and human cells
(
- Contribution to journal › Article
- 2016
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Mark
Mitotic History Reveals Distinct Stem Cell Populations and Their Contributions to Hematopoiesis
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- Contribution to journal › Article
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Mark
Cellular Barcoding Links B-1a B Cell Potential to a Fetal Hematopoietic Stem Cell State at the Single-Cell Level
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- Contribution to journal › Article
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Mark
Potential Pitfalls of the Mx1-Cre System : Implications for Experimental Modeling of Normal and Malignant Hematopoiesis
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- Contribution to journal › Article
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Mark
Human and murine hematopoietic stem cell aging is associated with functional impairments and intrinsic megakaryocytic/erythroid bias
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- Contribution to journal › Article
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Mark
Hematopoiesis at single-cell resolution
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- Contribution to journal › Debate/Note/Editorial
- 2015
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Mark
SCExV: a webtool for the analysis and visualisation of single cell qRT-PCR data.
(
- Contribution to journal › Article
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Mark
Concise Review: Hematopoietic Stem Cell Aging and the Prospects for Rejuvenation.
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- Contribution to journal › Article
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Mark
Probing hematopoietic stem cell function using serial transplantation: Seeding characteristics and the impact of stem cell purification.
(
- Contribution to journal › Article
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Mark
Disruption of the 5S RNP-Mdm2 interaction significantly improves the erythroid defect in a mouse model for Diamond-Blackfan anemia.
(
- Contribution to journal › Article
- 2014
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Mark
Hematopoietic Stem Cells Are Intrinsically Protected against MLL-ENL-Mediated Transformation.
(
- Contribution to journal › Article