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- 2019
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Mark
Heparin-Binding Protein As A Prognostic Biomarker of Sepsis and Disease Severity at The Emergency Department
(
- Contribution to journal › Article
- 2018
-
Mark
Effects of Dextran-70 and Albumin on Coagulation in Experimental Hemorrhage in the Guinea Pig
(
- Contribution to journal › Article
- 2017
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Mark
Heparin-Binding Protein (HBP) : A Causative Marker and Potential Target for Heparin Treatment of Human Sepsis-Induced Acute Kidney Injury
(
- Contribution to journal › Article
- 2016
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Mark
Diverse and Tissue Specific Mitochondrial Respiratory Response in A Mouse Model of Sepsis-Induced Multiple Organ Failure.
(
- Contribution to journal › Article
- 2015
-
Mark
Metabolomic Analyses of Brain Tissue in Sepsis Induced by Cecal Ligation Reveal Specific Redox Alterations-Protective Effects of the Oxygen Radical Scavenger Edaravone.
(
- Contribution to journal › Article
- 2014
-
Mark
DYNAMIC CHANGES IN THROMBIN GENERATION IN ABDOMINAL SEPSIS IN MICE.
(
- Contribution to journal › Article
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Mark
Prehospital Coagulation Monitoring of Resuscitation With Point-of-Care Devices.
(
- Contribution to journal › Scientific review
- 2013
-
Mark
Plasma Volume Expansion by 0.9% NaCl During sepsis/SIRS, After Hemorrhage, and During a Normal State.
(
- Contribution to journal › Article
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Mark
Geranylgeranyl transferase regulates streptococcal m1 protein-induced CXC chemokine formation and neutrophil recruitment in the lung.
(
- Contribution to journal › Article
- 2012
-
Mark
Early Complementopathy After Multiple Injuries in Humans.
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- Contribution to journal › Article
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Mark
Plasma Levels of Liver-Specific miR-122 is Massively Increased in a Porcine Cardiogenic Shock Model and Attenuated by Hypothermia.
(
- Contribution to journal › Article
-
Mark
Simvastatin protects against T cell immune dysfunction in abdominal sepsis.
(
- Contribution to journal › Article
-
Mark
Rho-Kinase Signaling Regulates Pulmonary Infiltration of Neutrophils in Abdominal Sepsis via Attenuation of CXC Chemokine Formation and Mac-1 Expression on Neutrophils.
(
- Contribution to journal › Article
- 2011
-
Mark
STREPTOCOCCAL M1 PROTEIN-INDUCED LUNG INJURY IS INDEPENDENT OF PLATELETS IN MICE.
(
- Contribution to journal › Article
-
Mark
Targeting CD44 Expressed on Neutrophils Inhibits Lung Damage in Abdominal Sepsis.
(
- Contribution to journal › Article
- 2010
-
Mark
M1 protein from Streptococcus pyogenes induces nitric oxidemediated vascular hyporesponsiveness to phenylephrine: Involvement of toll-like receptor activation.
(
- Contribution to journal › Article
-
Mark
Anesthesia Aggravates Lung Damage and Precipitates Hypotension in Endotoxemic Sheep
(
- Contribution to journal › Article
-
Mark
A Model for Evaluating the Effects of Blunt Skeletal Muscle Trauma on Microvascular Permeability and Plasma Volume in the Rat
(
- Contribution to journal › Article
-
Mark
MECHANISMS OF THE BENEFICIAL EFFECT OF HYPERTONIC SALINE SOLUTION IN ACUTE PANCREATITIS.
(
- Contribution to journal › Article
- 2008
-
Mark
LFA-1 AND MAC-1 MEDIATE PULMONARY RECRUITMENT OF NEUTROPHILS AND TISSUE DAMAGE IN ABDOMINAL SEPSIS.
(
- Contribution to journal › Article
-
Mark
INCREASED LEVELS OF GLYCOSAMINOGLYCANS DURING SEPTIC SHOCK: RELATION TO MORTALITY AND THE ANTIBACTERIAL ACTIONS OF PLASMA.
(
- Contribution to journal › Article
- 2007
-
Mark
Cardiovascular effects of levosimendan in the early stages of endotoxemia
(
- Contribution to journal › Article
- 2002
-
Mark
The effect of intestinal ischemia and reperfusion injury on ICAM-1 expression, endothelial barrier function, neutrophil tissue influx, and protease inhibitor levels in rats.
(
- Contribution to journal › Article
-
Mark
Hypovolemia is a main factor behind disturbed perfusion and metabolism in the intestine during endotoxemia in cat.
(
- Contribution to journal › Article
-
Mark
NF-kappaB activation and inhibition: a review.
(
- Contribution to journal › Scientific review
- 2001
-
Mark
The involvement of multiple protease-antiprotease systems and gut origin sepsis in zymosan-associated endothelial barrier injury and multiple organ dysfunction in rats
(
- Contribution to journal › Article