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- 2019
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Mark
Improved survival of men 50 to 75 years old with acute myeloid leukemia over a 20-year period
(
- Contribution to journal › Letter
- 2017
-
Mark
Increased proportion of mature NK cells is associated with successful imatinib discontinuation in chronic myeloid leukemia
(
- Contribution to journal › Article
- 2008
-
Mark
Segmentation-based detection of allelic imbalance and loss-of-heterozygosity in cancer cells using whole genome SNP arrays
(
- Contribution to journal › Article
- 2005
-
Mark
Molecular signatures in childhood acute leukemia and their correlations to expression patterns in normal hematopoietic subpopulations.
(
- Contribution to journal › Article
- 2004
-
Mark
High frequencies of chromosomal aberrations in multiple myeloma and monoclonal gammopathy of undetermined significance in direct chromosome preparation.
(
- Contribution to journal › Article
- 2003
-
Mark
A pooled analysis of karyotypic patterns, breakpoints and imbalances in 783 cytogenetically abnormal multiple myelomas reveals frequently involved chromosome segments as well as significant age- and sex-related differences.
(
- Contribution to journal › Article
-
Mark
Clinical and genetic studies of ETV6/ABL1-positive chronic myeloid leukaemia in blast crisis treated with imatinib mesylate.
(
- Contribution to journal › Article
- 2002
-
Mark
Cytogenetic features of multiple myeloma: impact of gender, age, disease phase, culture time, and cytokine stimulation
(
- Contribution to journal › Article
-
Mark
Acute myeloid leukemia with inv(16)(p13q22): Involvement of cervical lymph nodes and tonsils is common and may be a negative prognostic sign.
(
- Contribution to journal › Article
- 2000
-
Mark
Cytogenetic and FISH studies of a single center consecutive series of 152 childhood acute lymphoblastic leukemias
(
- Contribution to journal › Article
- 1999
-
Mark
Isochromosome 17q in blast crisis of chronic myeloid leukemia and in other hematologic malignancies is the result of clustered breakpoints in 17p11 and is not associated with coding TP53 mutations
(
- Contribution to journal › Article