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- 2022
-
Mark
Inducing synthetic lethality for selective targeting of acute myeloid leukemia cells harboring STAG2 mutations
(
- Contribution to journal › Letter
- 2018
-
Mark
Whole-exome sequencing exploration of acquired uniparental disomies in B-cell precursor acute lymphoblastic leukemia
(
- Contribution to journal › Article
- 2017
-
Mark
Distinct global binding patterns of the Wilms' tumor gene 1 (WT1) -KTS and +KTS isoforms in leukemic cells
(
- Contribution to journal › Article
- 2010
-
Mark
The Proportion of Ph+CD34(+)CD38(neg) Leukemic Stem Cells In the Bone Marrow of Newly Diagnosed Patients with Chronic Myeloid Leukemia (CML) In Chronic Phase (CP) Is Variable and Correlates with High Sokal Risk, High Leukocyte Count, Low Hemoglobin Concentration, Splenomegaly and Increased Hematological Toxicity During Initial TKI Therapy Data From a Randomized Phase II NordCML006 Study
(
- Contribution to journal › Published meeting abstract
-
Mark
Isolation and killing of candidate chronic myeloid leukemia stem cells by antibody targeting of IL-1 receptor accessory protein.
(
- Contribution to journal › Article
-
Mark
Gene expression signatures in childhood acute leukemias are largely unique and distinct from those of normal tissues and other malignancies.
(
- Contribution to journal › Article
- 2009
-
Mark
Expression of P190 and P210 BCR/ABL1 in normal human CD34(+) cells induces similar gene expression profiles and results in a STAT5-dependent expansion of the erythroid lineage
(
- Contribution to journal › Article
- 2008
-
Mark
An improved method for detecting and delineating genomic regions with altered gene expression in cancer
(
- Contribution to journal › Article
-
Mark
Gene expression analysis of BCR/ABL1-dependent transcriptional response reveals enrichment for genes involved in negative feedback regulation.
(
- Contribution to journal › Article
- 2007
-
Mark
Microarray-based classification of a consecutive series of 121 childhood acute leukemias: prediction of leukemic and genetic subtype as well as of minimal residual disease status.
(
- Contribution to journal › Article
-
Mark
Combined high-resolution array-based comparative genomic hybridization and expression profiling of ETV6/RUNX1-positive acute lymphoblastic leukemias reveal a high incidence of cryptic Xq duplications and identify several putative target genes within the commonly gained region
(
- Contribution to journal › Article
-
Mark
Deregulation of the Wilms' tumour gene 1 protein (WT1) by BCR/ABL1 mediates resistance to imatinib in human leukaemia cells
(
- Contribution to journal › Article
- 2006
-
Mark
Deregulation of cyclin D2 by juxtaposition with T-cell receptor alpha/delta locus in t(12;14)(p13;q11)-positive childhood T-cell acute lymphoblastic leukemia.
(
- Contribution to journal › Article
- 2005
-
Mark
Molecular signatures in childhood acute leukemia and their correlations to expression patterns in normal hematopoietic subpopulations.
(
- Contribution to journal › Article
- 2004
-
Mark
Identification of genes differentially regulated by the P210 BCR/ABL1 fusion oncogene using cDNA microarrays.
(
- Contribution to journal › Article
-
Mark
Establishment and phenotypic characterization of human U937 cells with inducible P210 BCR/ABL expression reveals upregulation of CEACAM1 (CD66a).
(
- Contribution to journal › Article
- 2003
-
Mark
Trisomy 8 as the sole chromosomal aberration in myelocytic malignancies. a multicolor and locus-specific fluorescence in situ hybridization study.
(
- Contribution to journal › Article
-
Mark
A Novel Gene, MSI2, Encoding a Putative RNA-binding Protein Is Recurrently Rearranged at Disease Progression of Chronic Myeloid Leukemia and Forms a Fusion Gene with HOXA9 as a Result of the Cryptic t(7;17)(p15;q23).
(
- Contribution to journal › Article
- 2002
-
Mark
Multicolor COBRA-FISH analysis of chronic myeloid leukemia reveals novel cryptic balanced translocations during disease progression.
(
- Contribution to journal › Article
- 2001
-
Mark
Fusion of the BCR and the fibroblast growth factor receptor-1 (FGFR1) genes as a result of t(8;22)(p11;q11) in a myeloproliferative disorder
(
- Contribution to journal › Published meeting abstract
-
Mark
Fusion of the BCR and the fibroblast growth factor receptor-1 (FGFR1) genes as a result of t(8;22)(p11;q11) in a myeloproliferative disorder: the first fusion gene involving BCR but not ABL
(
- Contribution to journal › Article
- 1999
-
Mark
Isochromosome 17q in blast crisis of chronic myeloid leukemia and in other hematologic malignancies is the result of clustered breakpoints in 17p11 and is not associated with coding TP53 mutations
(
- Contribution to journal › Article
-
Mark
Cytogenetic polyclonality in hematologic malignancies
(
- Contribution to journal › Article
- 1993
-
Mark
Clinical impact of breakpoint position within M-bcr in chronic myeloid leukemia
(
- Contribution to journal › Article