1 – 36 of 36
- show: 100
- |
- sort: year (new to old)
Close
Embed this list
<iframe src=" "
width=" "
height=" "
allowtransparency="true"
frameborder="0">
</iframe>
- 2023
-
Mark
Pathophysiology of type 2 diabetes and the impact of altered metabolic interorgan crosstalk
(
- Contribution to journal › Scientific review
-
Mark
Ablation of GPR56 Causes β-Cell Dysfunction by ATP Loss through Mistargeting of Mitochondrial VDAC1 to the Plasma Membrane
(
- Contribution to journal › Article
- 2022
-
Mark
Metformin enhances LDL-cholesterol uptake by suppressing the expression of the pro-protein convertase subtilisin/kexin type 9 (PCSK9) in liver cells
(
- Contribution to journal › Article
- 2021
-
Mark
Reduced Expression of Chl1 gene Impairs Insulin Secretion by Down-Regulating the Expression of Key Molecules of β-cell Function
(
- Contribution to journal › Article
- 2020
-
Mark
Inhibitory effect of UDP-glucose on cAMP generation and insulin secretion
(
- Contribution to journal › Article
-
Mark
An integrative phenotype–genotype approach using phenotypic characteristics from the UAE national diabetes study identifies HSD17B12 as a candidate gene for obesity and type 2 diabetes
(
- Contribution to journal › Article
-
Mark
Orphan G-protein coupled receptor 183 (GPR183) potentiates insulin secretion and prevents glucotoxicity-induced β-cell dysfunction
(
- Contribution to journal › Article
-
Mark
Somatostatin secretion by Na+-dependent Ca2+-induced Ca2+ release in pancreatic delta cells
(
- Contribution to journal › Letter
- 2019
-
Mark
Expression levels of enzymes generating NO and CO in islets of murine and human diabetes
(
- Contribution to journal › Article
-
Mark
The functional impact of G protein-coupled receptor 142 (Gpr142) on pancreatic β-cell in rodent
(
- Contribution to journal › Article
-
Mark
RORB and RORC associate with human islet dysfunction and inhibit insulin secretion in INS-1 cells
(
- Contribution to journal › Article
-
Mark
GNAS gene is an important regulator of insulin secretory capacity in pancreatic β-cells
(
- Contribution to journal › Article
-
Mark
Reduced Expression of PLCXD3 Associates With Disruption of Glucose Sensing and Insulin Signaling in Pancreatic β-Cells
(
- Contribution to journal › Article
-
Mark
Insulin inhibits glucagon release by SGLT2-induced stimulation of somatostatin secretion
(
- Contribution to journal › Article
- 2018
-
Mark
Activation of imidazoline receptor I2, and improved pancreatic β-cell function in human islets
(
- Contribution to journal › Article
-
Mark
Role of osteopontin and its regulation in pancreatic islet
(
- Contribution to journal › Article
-
Mark
α-cell glucokinase suppresses glucose-regulated glucagon secretion
(
- Contribution to journal › Article
-
Mark
Defining G protein-coupled receptor peptide ligand expressomes and signalomes in human and mouse islets
(
- Contribution to journal › Article
-
Mark
Adrenaline Stimulates Glucagon Secretion by Tpc2-Dependent Ca2+ Mobilization From Acidic Stores in Pancreatic α-Cells
(
- Contribution to journal › Article
- 2009
-
Mark
Impaired Insulin Exocytosis in Neural Cell Adhesion Molecule(-/-) Mice Due to Defective Reorganization of the Submembrane F-Actin Network
(
- Contribution to journal › Article
- 2007
-
Mark
Expression of islet iNOS and inhibition of glucose stimulated insulin release after long-term lipid infusion in the rat is counteracted by PACAP27.
(
- Contribution to journal › Article
-
Mark
A dominant mutation in Snap25 causes impaired vesicle trafficking, sensorimotor gating, and ataxia in the blind-drunk mouse
(
- Contribution to journal › Article
- 2006
-
Mark
Nitric oxide inhibits, and carbon monoxide activates, islet acid {alpha}-glucoside hydrolase activities in parallel with glucose-stimulated insulin secretion.
(
- Contribution to journal › Article
-
Mark
Does Epidermal Growth Factor Participate in the Regulation of Glucose, Insulin and Glucagon Levels?
(
- Contribution to journal › Article
- 2005
-
Mark
Ghrelin activates neuronal constitutive nitric oxide synthase in pancreatic islet cells while inhibiting insulin release and stimulating glucagon release.
(
- Contribution to journal › Article
-
Mark
Defective glucose-stimulated insulin release in the diabetic Goto-Kakizaki (GK) rat coincides with reduced activity of the islet carbon monoxide signaling pathway
(
- Contribution to journal › Article
-
Mark
Glucagon stimulates exocytosis in mouse and rat pancreatic {alpha} cells by binding to glucagon receptors.
(
- Contribution to journal › Article
-
Mark
CaV2.3 calcium channels control second-phase insulin release.
(
- Contribution to journal › Article
- 2004
-
Mark
Palmitate increases L-type Ca2+ currents and the size of the readily releasable granule pool in mouse pancreatic beta-cells.
(
- Contribution to journal › Article
-
Mark
Palmitate Stimulation of Glucagon Secretion in Mouse Pancreatic {alpha}-Cells Results From Activation of L-Type Calcium Channels and Elevation of Cytoplasmic Calcium.
(
- Contribution to journal › Article
-
Mark
GABAB-receptor activation inhibits exocytosis in rat pancreatic {beta}-cells by G-protein-dependent activation of calcineurin.
(
- Contribution to journal › Article
-
Mark
Capacitance measurements of exocytosis in mouse pancreatic {alpha}-, {beta}- and {delta}-cells studied in intact islets of Langerhans.
(
- Contribution to journal › Article
- 2003
-
Mark
Acute pancreatitis, expression of inducible nitric oxide synthase and defective insulin secretion.
(
- Contribution to journal › Article
-
Mark
SUR1 Regulates PKA-independent cAMP-induced Granule Priming in Mouse Pancreatic B-cells.
(
- Contribution to journal › Article
- 2002
-
Mark
Role of nitric oxide synthase isoforms in glucose-stimulated insulin release.
(
- Contribution to journal › Article
-
Mark
Fast insulin secretion reflects exocytosis of docked granules in mouse pancreatic B-cells.
(
- Contribution to journal › Article