Is type 1 diabetes a food-induced disease?
(2013) In Medical Hypotheses 81(2). p.338-342- Abstract
- The incidence of type 1 diabetes among children has almost doubled during the last decades in Sweden. Type 1 diabetes is considered as an autoimmune disease with unknown aetiology. Here we propose that the immune reaction may be initiated by food-derived mechanisms. The incidence of diabetes parallels an increased consumption of pasta, white bread, meat, cheese, low-fat milk, exotic fruits, soda, and snacks. Simultaneously, the consumption of potatoes, butter, high-fat milk, and domestic fruit has decreased. Three categories of food related reaction mechanisms are discussed against the following items (1) proteins from wheat, meat, and milk, (2) fat from processed food, and (3) exotic fruits. The current food consumption is suggested to... (More)
- The incidence of type 1 diabetes among children has almost doubled during the last decades in Sweden. Type 1 diabetes is considered as an autoimmune disease with unknown aetiology. Here we propose that the immune reaction may be initiated by food-derived mechanisms. The incidence of diabetes parallels an increased consumption of pasta, white bread, meat, cheese, low-fat milk, exotic fruits, soda, and snacks. Simultaneously, the consumption of potatoes, butter, high-fat milk, and domestic fruit has decreased. Three categories of food related reaction mechanisms are discussed against the following items (1) proteins from wheat, meat, and milk, (2) fat from processed food, and (3) exotic fruits. The current food consumption is suggested to initiate a pro-inflammatory reaction in the intestine and thereby reduce the intestinal barrier function. This may influence tolerance development and thus pave the way for an autoimmune attack on pancreatic beta cells. (Less)
Please use this url to cite or link to this publication:
https://lup.lub.lu.se/record/3804343
- author
- Landin-Olsson, Mona LU ; Hillman, Magnus LU and Erlanson-Albertsson, Charlotte LU
- organization
- publishing date
- 2013
- type
- Contribution to journal
- publication status
- published
- subject
- in
- Medical Hypotheses
- volume
- 81
- issue
- 2
- pages
- 338 - 342
- publisher
- Churchill Livingstone
- external identifiers
-
- wos:000322348900049
- pmid:23688738
- scopus:84880040372
- pmid:23688738
- ISSN
- 1532-2777
- DOI
- 10.1016/j.mehy.2013.03.046
- language
- English
- LU publication?
- yes
- id
- 36a02125-53e7-471d-a12c-daec3f3766c4 (old id 3804343)
- alternative location
- http://www.ncbi.nlm.nih.gov/pubmed/23688738?dopt=Abstract
- date added to LUP
- 2016-04-01 10:20:35
- date last changed
- 2024-02-21 14:26:17
@article{36a02125-53e7-471d-a12c-daec3f3766c4,
abstract = {{The incidence of type 1 diabetes among children has almost doubled during the last decades in Sweden. Type 1 diabetes is considered as an autoimmune disease with unknown aetiology. Here we propose that the immune reaction may be initiated by food-derived mechanisms. The incidence of diabetes parallels an increased consumption of pasta, white bread, meat, cheese, low-fat milk, exotic fruits, soda, and snacks. Simultaneously, the consumption of potatoes, butter, high-fat milk, and domestic fruit has decreased. Three categories of food related reaction mechanisms are discussed against the following items (1) proteins from wheat, meat, and milk, (2) fat from processed food, and (3) exotic fruits. The current food consumption is suggested to initiate a pro-inflammatory reaction in the intestine and thereby reduce the intestinal barrier function. This may influence tolerance development and thus pave the way for an autoimmune attack on pancreatic beta cells.}},
author = {{Landin-Olsson, Mona and Hillman, Magnus and Erlanson-Albertsson, Charlotte}},
issn = {{1532-2777}},
language = {{eng}},
number = {{2}},
pages = {{338--342}},
publisher = {{Churchill Livingstone}},
series = {{Medical Hypotheses}},
title = {{Is type 1 diabetes a food-induced disease?}},
url = {{http://dx.doi.org/10.1016/j.mehy.2013.03.046}},
doi = {{10.1016/j.mehy.2013.03.046}},
volume = {{81}},
year = {{2013}},
}