IgG Binds Escherichia coli Serine Protease EspP and Protects Mice From E. coli O157:H7 Infection
(2022) In Frontiers in Immunology 13.- Abstract
Shiga toxin-producing Escherichia coli O157:H7 is a virulent strain causing severe gastrointestinal infection, hemolytic uremic syndrome and death. To date there are no specific therapies to reduce progression of disease. Here we investigated the effect of pooled immunoglobulins (IgG) on the course of disease in a mouse model of intragastric E. coli O157:H7 inoculation. Intraperitoneal administration of murine IgG on day 3, or both on day 3 and 6, post-inoculation improved survival and decreased intestinal and renal pathology. When given on both day 3 and 6 post-inoculation IgG treatment also improved kidney function in infected mice. Murine and human commercially available IgG preparations bound to proteins in culture filtrates from E.... (More)
Shiga toxin-producing Escherichia coli O157:H7 is a virulent strain causing severe gastrointestinal infection, hemolytic uremic syndrome and death. To date there are no specific therapies to reduce progression of disease. Here we investigated the effect of pooled immunoglobulins (IgG) on the course of disease in a mouse model of intragastric E. coli O157:H7 inoculation. Intraperitoneal administration of murine IgG on day 3, or both on day 3 and 6, post-inoculation improved survival and decreased intestinal and renal pathology. When given on both day 3 and 6 post-inoculation IgG treatment also improved kidney function in infected mice. Murine and human commercially available IgG preparations bound to proteins in culture filtrates from E. coli O157:H7. Bound proteins were extracted from membranes and peptide sequences were identified by mass spectrometry. The findings showed that murine and human IgG bound to E. coli extracellular serine protease P (EspP) in the culture filtrate, via the IgG Fc domain. These results were confirmed using purified recombinant EspP and comparing culture filtrates from the wild-type E. coli O157:H7 strain to a deletion mutant lacking espP. Culture filtrates from wild-type E. coli O157:H7 exhibited enzymatic activity, specifically associated with the presence of EspP and demonstrated as pepsin cleavage, which was reduced in the presence of murine and human IgG. EspP is a virulence factor previously shown to promote colonic cell injury and the uptake of Shiga toxin by intestinal cells. The results presented here suggest that IgG binds to EspP, blocks its enzymatic activity, and protects the host from E. coli O157:H7 infection, even when given post-inoculation.
(Less)
- author
- Tontanahal, Ashmita LU ; Sperandio, Vanessa ; Kovbasnjuk, Olga ; Loos, Sebastian LU ; Kristoffersson, Ann Charlotte LU ; Karpman, Diana LU and Arvidsson, Ida LU
- organization
- publishing date
- 2022-02
- type
- Contribution to journal
- publication status
- published
- subject
- keywords
- Escherichia coli O157:H7, EspP, hemolytic uremic syndrome, immunoglobulin G, mouse, Shiga toxin
- in
- Frontiers in Immunology
- volume
- 13
- article number
- 807959
- publisher
- Frontiers Media S. A.
- external identifiers
-
- pmid:35250980
- scopus:85125838014
- ISSN
- 1664-3224
- DOI
- 10.3389/fimmu.2022.807959
- language
- English
- LU publication?
- yes
- id
- 5d37453a-ad09-459d-9ce6-aaa8eeb0854d
- date added to LUP
- 2022-05-02 11:00:28
- date last changed
- 2024-11-01 03:03:06
@article{5d37453a-ad09-459d-9ce6-aaa8eeb0854d, abstract = {{<p>Shiga toxin-producing Escherichia coli O157:H7 is a virulent strain causing severe gastrointestinal infection, hemolytic uremic syndrome and death. To date there are no specific therapies to reduce progression of disease. Here we investigated the effect of pooled immunoglobulins (IgG) on the course of disease in a mouse model of intragastric E. coli O157:H7 inoculation. Intraperitoneal administration of murine IgG on day 3, or both on day 3 and 6, post-inoculation improved survival and decreased intestinal and renal pathology. When given on both day 3 and 6 post-inoculation IgG treatment also improved kidney function in infected mice. Murine and human commercially available IgG preparations bound to proteins in culture filtrates from E. coli O157:H7. Bound proteins were extracted from membranes and peptide sequences were identified by mass spectrometry. The findings showed that murine and human IgG bound to E. coli extracellular serine protease P (EspP) in the culture filtrate, via the IgG Fc domain. These results were confirmed using purified recombinant EspP and comparing culture filtrates from the wild-type E. coli O157:H7 strain to a deletion mutant lacking espP. Culture filtrates from wild-type E. coli O157:H7 exhibited enzymatic activity, specifically associated with the presence of EspP and demonstrated as pepsin cleavage, which was reduced in the presence of murine and human IgG. EspP is a virulence factor previously shown to promote colonic cell injury and the uptake of Shiga toxin by intestinal cells. The results presented here suggest that IgG binds to EspP, blocks its enzymatic activity, and protects the host from E. coli O157:H7 infection, even when given post-inoculation.</p>}}, author = {{Tontanahal, Ashmita and Sperandio, Vanessa and Kovbasnjuk, Olga and Loos, Sebastian and Kristoffersson, Ann Charlotte and Karpman, Diana and Arvidsson, Ida}}, issn = {{1664-3224}}, keywords = {{Escherichia coli O157:H7; EspP; hemolytic uremic syndrome; immunoglobulin G; mouse; Shiga toxin}}, language = {{eng}}, publisher = {{Frontiers Media S. A.}}, series = {{Frontiers in Immunology}}, title = {{IgG Binds Escherichia coli Serine Protease EspP and Protects Mice From E. coli O157:H7 Infection}}, url = {{http://dx.doi.org/10.3389/fimmu.2022.807959}}, doi = {{10.3389/fimmu.2022.807959}}, volume = {{13}}, year = {{2022}}, }