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- 2010
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Mark
Overexpression of Alpha2A-Adrenergic Receptors Contributes to Type 2 Diabetes.
- Contribution to journal › Article
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Mark
Molecular Function of TCF7L2: Consequences of TCF7L2 Splicing for Molecular Function and Risk for Type 2 Diabetes.
- Contribution to journal › Article
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Mark
GLP-1 inhibits and adrenaline stimulates glucagon release by differential modulation of N- and L-type Ca2+ channel-dependent exocytosis.
- Contribution to journal › Article
- 2009
-
Mark
Complement factors are expressed in pancreatic islets and affect glucose induced insulin secretion
(2009) 45th Annual Meeting of the European-Association-for-the-Study-of-Diabetes In Diabetologia 52(Suppl 1). p.455-455
- Contribution to journal › Published meeting abstract
-
Mark
The impact of differential splicing of TCF7L2 on target gene expression in human islet of Langerhans
(2009) 45th Annual Meeting of the European-Association-for-the-Study-of-Diabetes In Diabetologia 52(Suppl 1). p.78-78
- Contribution to journal › Published meeting abstract
-
Mark
Evaluation of chloride channel-3 as a potential target for TCF7L2-dependent impairment of insulin secretion
(2009) 45th Annual Meeting of the European-Association-for-the-Study-of-Diabetes In Diabetologia 52(Suppl 1). p.332-332
- Contribution to journal › Published meeting abstract
-
Mark
Rapid changes in surface expression of L-type Cav1.2 channels in stimulated INS-1 cells
(2009) 45th Annual Meeting of the European-Association-for-the-Study-of-Diabetes In Diabetologia 52(Suppl. 1). p.436-436
- Contribution to journal › Published meeting abstract
-
Mark
Hyperactivity in adrenergic signalling via alpha2A receptors contributes to human type 2 diabetes
(2009) 45th Annual Meeting of the European-Association-for-the-Study-of-Diabetes In Diabetologia 52(Suppl 1). p.108-108
- Contribution to journal › Published meeting abstract
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Mark
Bone marrow transplantation stimulates pancreatic β-cell replication after tissue damage.
- Contribution to journal › Article
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Mark
Suppression of sulfonylurea- and glucose-induced insulin secretion in vitro and in vivo in mice lacking the chloride transport protein ClC-3.
- Contribution to journal › Article
