Xingjun Jing (Former)
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        - 2021
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                        Mark
        Evaluation of hypoglycemic effect, safety and immunomodulation of Prevotella copri in mice
    
    - Contribution to journal › Article
 
- 2012
- 
                        Mark
        A systems genetics approach identifies genes and pathways for type 2 diabetes in human islets.
    
    - Contribution to journal › Article
 
- 2011
- 
                        Mark
        Survival of rat pancreatic islets is partly controlled by a TCF7L2-p53-p53INP1 dependent pathway
    
    - Contribution to journal › Article
 
- 2009
- 
                        Mark
        Evaluation of chloride channel-3 as a potential target for TCF7L2-dependent impairment of insulin secretion
    (2009) 45th Annual Meeting of the European-Association-for-the-Study-of-Diabetes In Diabetologia 52(Suppl 1). p.332-332- Contribution to journal › Published meeting abstract
 
- 
                        Mark
        Suppression of sulfonylurea- and glucose-induced insulin secretion in vitro and in vivo in mice lacking the chloride transport protein ClC-3.
    
    - Contribution to journal › Article
 
- 2007
- 
                        Mark
        Ion channel control of phasic insulin secretion.
    
    - Thesis › Doctoral thesis (compilation)
 
- 2005
- 
                        Mark
        Myosin 5a controls insulin granule recruitment during late-phase secretion.
    
    - Contribution to journal › Article
 
- 
                        Mark
        CaV2.3 calcium channels control second-phase insulin release.
    
    - Contribution to journal › Article
 
- 2003
- 
                        Mark
        Impaired insulin secretion and glucose tolerance in beta cell-selective Ca(V)1.2 Ca(2+) channel null mice.
    
    - Contribution to journal › Article
 
- 2002
- 
                        Mark
        Important role of phosphodiesterase 3B for the stimulatory action of cAMP on pancreatic beta -cell exocytosis and release of insulin.
    
    - Contribution to journal › Article
 
