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- 2008
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Mark
Palmitate-induced beta-cell dysfunction is associated with excessive NO production and is reversed by thiazolidinedione-mediated inhibition of GPR40 transduction mechanisms.
- Contribution to journal › Article
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Mark
Excessive islet NO generation in type 2 diabetic GK rats coincides with abnormal hormone secretion and is counteracted by GLP-1.
- Contribution to journal › Article
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Mark
ADP mediates inhibition of insulin secretion by activation of P2Y(13) receptors
(2008) 44th EASD Annual Meeting of the European Association for the Study of Diabetes In Diabetologia 51(S1). p.197-197
- Contribution to journal › Published meeting abstract
-
Mark
Long-term exposure of mouse pancreatic islets to oleate or palmitate results in reduced glucose-induced somatostatin and oversecretion of glucagon
- Contribution to journal › Article
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Mark
K-ATP-channels and glucagon secretion glucose-regulated
- Contribution to journal › Scientific review
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Mark
GLP-1 inhibits glucagon secretion by affecting several cellular mechanisms
(2008) 44th EASD Annual Meeting of the European Association for the Study of Diabetes In Diabetologia 51(S1). p.256-256
- Contribution to journal › Published meeting abstract
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Mark
Proghrelin-derived peptides influence the secretion of insulin, glucagon, pancreatic polypeptide and somatostatin: A study on isolated islets from mouse and rat pancreas.
- Contribution to journal › Article
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Mark
Challenges Associated with Scaling up Artemisinin Combination Therapy in Sub-Saharan Africa A Review Article
- Contribution to journal › Scientific review
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Mark
Rosiglitazone counteracts palmitate-induced beta-cell dysfunction by suppression of MAP kinase, inducible nitric oxide synthase and caspase 3 activities.
- Contribution to journal › Article
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Mark
Nature of Large Vesicle Exocytosis in Pancreatic β-cells: Release of ATP and GABA
- Thesis › Doctoral thesis (compilation)
