51 – 100 of 233
- show: 50
- |
- sort: year (new to old)
Close
Embed this list
<iframe src=" "
width=" "
height=" "
allowtransparency="true"
frameborder="0">
</iframe>
- 2009
-
Mark
Tumor necrosis factor receptor-1 is essential for LPS-induced sensitization and tolerance to oxygen-glucose deprivation in murine neonatal organotypic hippocampal slices.
2009) In Journal of cerebral blood flow and metabolism : official journal of the International Society of Cerebral Blood Flow and Metabolism 29. p.73-86(
- Contribution to journal › Article
-
Mark
Enriched environment reduces apolipoprotein E (ApoE) in reactive astrocytes and attenuates inflammation of the peri-infarct tissue after experimental stroke.
2009) In Journal of cerebral blood flow and metabolism : official journal of the International Society of Cerebral Blood Flow and Metabolism 29. p.1796-1805(
- Contribution to journal › Article
- 2008
-
Mark
Intranasal selective brain cooling in pigs
(
- Contribution to journal › Article
-
Mark
Improving outcome after stroke: Overcoming the translational roadblock
(
- Contribution to journal › Scientific review
-
Mark
Apolipoprotein D is elevated in oligodendrocytes in the peri-infarct region after experimental stroke: influence of enriched environment.
(
- Contribution to journal › Article
-
Mark
Overexpression of UCP2 protects thalamic neurons following global ischemia in the mouse.
(
- Contribution to journal › Article
- 2007
-
Mark
Rapid and long-term induction of effector immediate early genes (BDNF, Neuritin and Arc) in peri-infarct cortex and dentate gyrus after ischemic injury in rat brain.
(
- Contribution to journal › Article
-
Mark
Hypothermia Affects Translocation of Numerous Cytoplasmic Proteins Following Global Cerebral Ischemia.
(
- Contribution to journal › Article
-
Mark
Mitochondrial Permeability Transition in the CNS - Composition, Regulation, and Pathophysiological Relevance
2007) p.667-702(
- Chapter in Book/Report/Conference proceeding › Book chapter
- 2006
-
Mark
On the move to stimulate cell plasticity in the substantia nigra in Parkinson's disease.
(
- Contribution to journal › Letter
-
Mark
Npas4, a novel helix-loop-helix PAS domain protein, is regulated in response to cerebral ischemia
(
- Contribution to journal › Article
-
Mark
A New Method of Selective, Rapid Cooling of the Brain: An Experimental Study.
(
- Contribution to journal › Article
-
Mark
Mechanisms of neural plasticity following brain injury.
(
- Contribution to journal › Article
-
Mark
Enriched Environment Attenuates Cell Genesis in Subventricular Zone After Focal Ischemia in Mice and Decreases Migration of Newborn Cells to the Striatum.
(
- Contribution to journal › Article
-
Mark
Decreased expression of brain-derived neurotrophic factor in BDNF(+/-) mice is associated with enhanced recovery of motor performance and increased neuroblast number following experimental stroke.
(
- Contribution to journal › Article
-
Mark
Comprehensive regional and temporal gene expression profiling of the rat brain during the first 24 h after experimental stroke identifies dynamic ischemia-induced gene expression patterns, and reveals a biphasic activation of genes in surviving tissue
(
- Contribution to journal › Article
- 2005
-
Mark
Chelation of intracellular calcium reduces cell death after hyperglycemic in vitro ischemia in murine hippocampal slice cultures.
(
- Contribution to journal › Article
-
Mark
Selective sparing of hippocampal CA3 cells following in vitro ischemia is due to selective inhibition by acidosis.
(
- Contribution to journal › Article
-
Mark
Combining neuroprotective treatment of embryonic nigral donor tissue with mild hypothermia of the graft recipient.
(
- Contribution to journal › Article
-
Mark
Actin redistribution underlies the sparing effect of mild hypothermia on dendritic spine morphology after in vitro ischemia.
(
- Contribution to journal › Article
-
Mark
The temperature dependence and involvement of mitochondria permeability transition and caspase activation in damage to organotypic hippocampal slices following in vitro ischemia.
(
- Contribution to journal › Article
-
Mark
Enriched environment enhances recovery of motor function after focal ischemia in mice, and downregulates the transcription factor NGFI-A.
(
- Contribution to journal › Article
-
Mark
Death-associated protein kinase is activated by dephosphorylation in response to cerebral ischemia
(
- Contribution to journal › Article
- 2004
-
Mark
Deletion of the adenosine A receptor gene does not alter neuronal damage following ischaemia in vivo or in vitro.
(
- Contribution to journal › Article
-
Mark
Lack of neuroprotection by heat shock protein 70 overexpression in a mouse model of global cerebral ischemia.
(
- Contribution to journal › Article
-
Mark
Mitochondrial damage and dysfunction in traumatic brain injury
(
- Contribution to journal › Article
-
Mark
Hyperglycemia and hypercapnia differently affect post-ischemic changes in protein kinases and protein phosphorylation in the rat cingulate cortex
(
- Contribution to journal › Article
-
Mark
Gene deletion of cystatin C aggravates brain damage following focal ischemia but mitigates the neuronal injury after global ischemia in the mouse.
(
- Contribution to journal › Article
-
Mark
Protein Kinase C-gamma and Calcium/Calmodulin-Dependent Protein Kinase II-alpha Are Persistently Translocated to Cell Membranes of the Rat Brain During and After Middle Cerebral Artery Occlusion.
(
- Contribution to journal › Scientific review
-
Mark
Terapeutisk hypotermi efter hjärtstopp - ny länk i kedjan som kan rädda liv
(
- Contribution to journal › Article
-
Mark
Glucose but Not Lactate in Combination With Acidosis Aggravates Ischemic Neuronal Death In Vitro.
(
- Contribution to journal › Article
-
Mark
Protein Kinase C-γ and Calcium/Calmodulin-Dependent Protein Kinase II-α Are Persistently Translocated to Cell Membranes of the Rat Brain during and after Middle Cerebral Artery Occlusion
(
- Contribution to journal › Article
- 2003
-
Mark
Mineralocorticoid receptor expression and increased survival following neuronal injury.
(
- Contribution to journal › Article
-
Mark
Brain damage in a mouse model of global cerebral ischemia. Effect of NMDA receptor blockade.
(
- Contribution to journal › Article
-
Mark
Powerful cyclosporin inhibition of calcium-induced permeability transition in brain mitochondria.
(
- Contribution to journal › Article
-
Mark
Mouse hippocampal organotypic tissue cultures exposed to in vitro "ischemia" show selective and delayed CA1 damage that is aggravated by glucose
(
- Contribution to journal › Article
-
Mark
Flow cytometric analysis of mitochondria from CA1 and CA3 regions of rat hippocampus reveals differences in permeability transition pore activation.
(
- Contribution to journal › Article
-
Mark
Structural and functional damage sustained by mitochondria after traumatic brain injury in the rat: Evidence for differentially sensitive populations in the cortex and hippocampus
(
- Contribution to journal › Article
-
Mark
Cyclosporin A prevents calpain activation despite increased intracellular calcium concentrations, as well as translocation of apoptosis-inducing factor, cytochrome c and caspase-3 activation in neurons exposed to transient hypoglycemia.
(
- Contribution to journal › Article
-
Mark
Uncoupling protein-2 prevents neuronal death and diminishes brain dysfunction after stroke and brain trauma.
(
- Contribution to journal › Article
- 2002
-
Mark
Mitochondrial oxidative stress after global brain ischemia in rats.
(
- Contribution to journal › Article
-
Mark
Mitochondrial permeability transition in acute neurodegeneration.
(
- Contribution to journal › Article
-
Mark
Restricted clinical efficacy of cyclosporin A on rat transient middle cerebral artery occlusion
(
- Contribution to journal › Article
-
Mark
Persistent phosphorylation of synaptic proteins following middle cerebral artery occlusion
(
- Contribution to journal › Article
-
Mark
Cerebral ischemia upregulates vascular endothelin ETB receptors in rat
(
- Contribution to journal › Article
- 2001
-
Mark
Neuroprotective and behavioral efficacy of nerve growth factor-transfected hippocampal progenitor cell transplants after experimental traumatic brain injury
(
- Contribution to journal › Article
-
Mark
Plasma fibronectin supports neuronal survival and reduces brain injury following transient focal cerebral ischemia but is not essential for skin-wound healing and hemostasis
(
- Contribution to journal › Article
-
Mark
Mitochondrial involvement in acute neurodegeneration
(
- Contribution to journal › Article
-
Mark
Hög tid att pröva hypotermi i behandlingen av slaganfall
(
- Contribution to journal › Article
-
Mark
Workshop on hypoglycemia and the brain
(
- Contribution to journal › Article