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        - 2015
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                        Mark
        Non-HLA type 1 diabetes genes modulate disease risk together with HLA-DQ and islet autoantibodies.
    
    
- Contribution to journal › Article
 
 - 2014
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                        Mark
        Prevalence of obesity was related to HLA-DQ in 2-4-year-old children at genetic risk for type 1 diabetes.
    
    
- Contribution to journal › Article
 
 - 2013
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                        Mark
        Next-generation sequencing for viruses in children with rapid-onset type 1 diabetes
    
    
- Contribution to journal › Article
 
 - 2011
 - 
                        Mark
        Country-specific birth weight and length in type 1 diabetes high-risk HLA genotypes in combination with prenatal characteristics.
    
    
- Contribution to journal › Article
 
 - 2010
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                        Mark
        The association between the PTPN22 1858C > T variant and type 1 diabetes depends on HLA risk and GAD65 autoantibodies
    
    
- Contribution to journal › Article
 
 - 1998
 - 
                        Mark
        Association between autoantibody markers and subtypes of DR4 and DR4-DQ in Swedish children with insulin-dependent diabetes reveals closer association of tyrosine pyrophosphatase autoimmunity with DR4 than DQ8
    
    
- Contribution to journal › Article
 
 - 1996
 - 
                        Mark
        HLA and glutamic acid decarboxylase in human insulin-dependent diabetes mellitus
    
    
- Contribution to journal › Article
 
 - 
                        Mark
        Genetic and immunological findings in patients with newly diagnosed insulin-dependent diabetes mellitus
    
    
- Contribution to journal › Article
 
 - 1995
 - 
                        Mark
        Glutamate decarboxylase antibody levels predict rate of β-cell decline in adult-onset diabetes
    
    
- Contribution to journal › Article
 
 - 
                        Mark
        Differential detection of rat islet and brain glutamic acid decarboxylase (GAD) isoforms with sequence-specific peptide antibodies
    
    
- Contribution to journal › Article
 
 
