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- 2005
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Mark
The extracellular matrix and inflammation - Fibromodulin activates the classical pathway of complement by directly binding C1q
- Contribution to journal › Article
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Mark
A novel mechanism of action of the fumagillin analog, TNP-470, in the B16F10 murine melanoma cell line
- Contribution to journal › Article
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Mark
Human CO-binding protein selectively interacts with Neisseria gonorrhoeae and results in species-specific infection
- Contribution to journal › Article
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Mark
C4b-binding protein binds to necrotic cells and DNA, limiting DNA release and inhibiting complement activation.
- Contribution to journal › Article
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Mark
Plasma bikunin: half-life and tissue uptake
- Contribution to journal › Article
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Mark
Inhibition of coxsackie B virus infection by soluble forms of its receptors: Binding affinities, altered particle formation, and competition with cellular receptors
- Contribution to journal › Article
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Mark
Ionic binding of C3 to the human pathogen Moraxella catarrhalis is a unique mechanism for combating innate immunity
- Contribution to journal › Article
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Mark
Activation of CD40 in cervical carcinoma cells facilitates CTL responses and augments chemotherapy-induced apoptosis
- Contribution to journal › Article
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Mark
Interactions of the extracellular matrix proteoglycans decorin and biglycan with C1q and collectins
- Contribution to journal › Article
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Mark
Binding of the complement inhibitor C4bp to serogroup B Neisseria meningitidis
- Contribution to journal › Article
