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- 2024
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Mark
In-depth proteomic analysis and structural modelling of protein-protein interactions identify known and novel molecular binding partners for EMP3 in erythroid cells
(
- Contribution to journal › Published meeting abstract
-
Mark
New sickle cell disease drug approved for use in England – here’s how voxelotor works
2024) In The Conversation(
- Contribution to specialist publication or newspaper › Specialist publication article
- 2023
-
Mark
Recombinant α
1-Microglobulin (rA1M) Protects against Hematopoietic and Renal Toxicity, Alone and in Combination with Amino Acids, in a
177Lu-DOTATATE Mouse Radiation Model.
(
- Contribution to journal › Article
- 2022
-
Mark
Targeting elevated heme levels to treat a mouse model for Diamond-Blackfan Anemia
(
- Contribution to journal › Article
-
Mark
Nuclear stabilization of p53 requires a functional nucleolar surveillance pathway
(
- Contribution to journal › Article
-
Mark
Successful gene therapy of Diamond-Blackfan anemia in a mouse model and human CD34+ cord blood hematopoietic stem cells using a clinically applicable lentiviral vector
(
- Contribution to journal › Article
-
Mark
Small Molecules Pushing Erythroid/Megakaryocyte Cell Specification Boundaries
(
- Contribution to journal › Article
- 2021
-
Mark
Human radical scavenger α1-microglobulin protects against hemolysis in vitro and α1-microglobulin knockout mice exhibit a macrocytic anemia phenotype
(
- Contribution to journal › Article
-
Mark
Bone marrow transplantation without myeloablative conditioning in a mouse model for Diamond-Blackfan anemia corrects the disease phenotype
(
- Contribution to journal › Article
-
Mark
Yippee like 4 (Ypel4) is essential for normal mouse red blood cell membrane integrity
(
- Contribution to journal › Article
-
Mark
The active component of ginseng, ginsenoside Rb1, improves erythropoiesis in models of Diamond–Blackfan anemia by targeting Nemo-like kinase
(
- Contribution to journal › Article
- 2020
-
Mark
Prospective isolation of radiation induced erythroid stress progenitors reveals unique transcriptomic and epigenetic signatures enabling increased erythroid output
(
- Contribution to journal › Article
-
Mark
Metformin-induced suppression of Nemo-like kinase improves erythropoiesis in preclinical models of Diamond–Blackfan anemia through induction of miR-26a
(
- Contribution to journal › Article
-
Mark
Diamond Blackfan anemia is mediated by hyperactive Nemo-like kinase
(
- Contribution to journal › Article
-
Mark
The Role of α1-Microglobulin (A1M) in Erythropoiesis and Erythrocyte Homeostasis-Therapeutic Opportunities in Hemolytic Conditions
(
- Contribution to journal › Scientific review
-
Mark
Calmodulin inhibitors improve erythropoiesis in Diamond-Blackfan anemia
(
- Contribution to journal › Article
-
Mark
Gene editing of CD34+ progenitor cells from single blood donor waste bags to create cultured early erythroid cells for study of blood group knock-outs
2020) The 36th International ISBT Congress, Virtual meeting In Vox Sanguinis 115(Suppl. s1). p.363-363(
- Contribution to journal › Published meeting abstract
- 2019
-
Mark
A Phenotypic Screening Assay Identifies Modulators of Diamond Blackfan Anemia
(
- Contribution to journal › Article
-
Mark
Cyclin-dependent kinase 8/19 inhibition suppresses osteoclastogenesis by downregulating RANK and promotes osteoblast mineralization and cancellous bone healing
(
- Contribution to journal › Article
- 2018
-
Mark
Direct Lineage Reprogramming of Adult Mouse Fibroblast to Erythroid Progenitors
2018) In Journal of visualized experiments : JoVE(
- Contribution to journal › Article
-
Mark
Small Molecule Screens Identify CDK8-Inhibitors As Candidate Diamond-Blackfan Anemia Drugs
(
- Contribution to journal › Published meeting abstract
- 2017
-
Mark
Lentiviral Vectors with Cellular Promoters Correct Anemia and Lethal Bone Marrow Failure in a Mouse Model for Diamond-Blackfan Anemia
(
- Contribution to journal › Article
-
Mark
Direct lineage reprogramming : a useful addition to the blood cell research toolbox
(
- Contribution to journal › Debate/Note/Editorial
- 2016
-
Mark
Direct Conversion of Fibroblasts to Megakaryocyte Progenitors
(
- Contribution to journal › Article
-
Mark
Defining the Minimal Factors Required for Erythropoiesis through Direct Lineage Conversion
(
- Contribution to journal › Article
-
Mark
The european hematology association roadmap for european hematology research : A consensus document
(
- Contribution to journal › Article
- 2015
-
Mark
Glucocorticoids improve erythroid progenitor maintenance and dampen Trp53 response in a mouse model of Diamond-Blackfan anaemia.
(
- Contribution to journal › Article
-
Mark
Disruption of the 5S RNP-Mdm2 interaction significantly improves the erythroid defect in a mouse model for Diamond-Blackfan anemia.
(
- Contribution to journal › Article
- 2014
-
Mark
Gene therapy cures the anemia and lethal bone marrow failure in mouse model for RPS19-deficient Diamond-Blackfan anemia.
(
- Contribution to journal › Article
- 2013
-
Mark
Fetal hepatic progenitors support long-term expansion of hematopoietic stem cells
(
- Contribution to journal › Article
-
Mark
A Robust Screening Assay For Diamond Blackfan Anemia Candidate Drugs
(
- Contribution to journal › Published meeting abstract
- 2012
-
Mark
Dietary L-leucine improves the anemia in a mouse model for Diamond-Blackfan anemia.
(
- Contribution to journal › Article
-
Mark
Progress towards Mechanism-Based Treatment for Diamond-Blackfan Anemia.
(
- Contribution to journal › Article
- 2011
-
Mark
From stem cell to red cell: regulation of erythropoiesis at multiple levels by multiple proteins, RNAs, and chromatin modifications
(
- Contribution to journal › Scientific review
-
Mark
Mice with ribosomal protein S19 deficiency develop bone marrow failure and symptoms like patients with Diamond-Blackfan anemia.
(
- Contribution to journal › Article
- 2010
-
Mark
Chronic RPS19 Deficiency Leads to Bone Marrow Failure In a Mouse Model for Diamond Blackfan Anemia
(
- Contribution to journal › Published meeting abstract
- 2009
-
Mark
A Novel Mouse Model for RPS19-Deficient Diamond-Blackfan Anemia Locates the Erythroid Defect at CFU-E/Proerythroblast Transition
(
- Contribution to journal › Published meeting abstract
- 2008
-
Mark
RPS19 Deficiency Leads to Reduced Proliferation and Increased Apoptosis but Does Not Affect Terminal Erythroid Differentiation in a Cell Line Model of Diamond-Blackfan Anemia
(
- Contribution to journal › Article
-
Mark
Gene therapy of Diamond Blackfan anemia CD34(+) cells leads to improved erythroid development and engraftment following transplantation.
(
- Contribution to journal › Article
- 2007
-
Mark
Diamond-Blackfan anemia: erythropoiesis lost in translation.
(
- Contribution to journal › Scientific review
-
Mark
Human RPS19, the gene mutated in Diamond Blackfan anemia, encodes a ribosomal protein required for the maturation of 40S ribosomal subunits.
(
- Contribution to journal › Article
-
Mark
Endoglin is not critical for hematopoietic stem cell engraftment and reconstitution but regulates adult erythroid development
(
- Contribution to journal › Article
-
Mark
Diamond-Blackfan Anemia: Erythropoiesis Lost in Translation
2007)(
- Thesis › Doctoral thesis (compilation)
-
Mark
IAP antagonists induce autoubiquitination of c-IAPs, NF-kappaB activation, and TNFalpha-dependent apoptosis
(
- Contribution to journal › Article
- 2006
-
Mark
Erythropoiesis in the Rps19 disrupted mouse: Analysis of erythropoietin response and biochemical markers for Diamond-Blackfan anemia
(
- Contribution to journal › Article
- 2005
-
Mark
Development of cellular models for ribosomal protein S19 (RPS19)-deficient diamond-blackfan anemia using inducible expression of siRNA against RPS19.
(
- Contribution to journal › Article
-
Mark
Deficiency of ribosomal protein S19 in CD34+ cells generated by siRNA blocks erythroid development and mimics defects seen in Diamond-Blackfan anemia.
(
- Contribution to journal › Article
- 2003
-
Mark
Proliferation deficiency of multipotent hematopoietic progenitors in ribosomal protein S19 (RPS19)-deficient Diamond-Blackfan anemia improves following RPS19 gene transfer
(
- Contribution to journal › Article
-
Mark
Lentiviral vector transduction of NOD/SCID repopulating cells results in multiple vector integrations per transduced cell: risk of insertional mutagenesis.
(
- Contribution to journal › Article