Molecular virology
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- 2019
- Short half-life of HPV16 E6 and E7 mRNAs sensitizes HPV16-positive tonsillar cancer cell line HN26 to DNA-damaging drugs
- 2018
- Role of the DNA damage response in human papillomavirus RNA splicing and polyadenylation
- HPV16 E5 is produced from an HPV16 early mRNA spliced from SD226 to SA3358
- Adenosine causes read-through into the late region of the HPV16 genome in a guanosine-dependent manner
- 2017
- Splicing and polyadenylation of human papillomavirus type 16 mRNAs
- hnRNP L controls HPV16 RNA polyadenylation and splicing in an Akt kinase-dependent manner
- 2015
- Acetylation of intragenic histones on HPV16 correlates with enhanced HPV16 gene expression.
- Heterogeneous nuclear ribonucleoprotein C proteins interact with the human papillomavirus type 16 (HPV16) early 3'-untranslated region and alleviate suppression of HPV16 late L1 mRNA splicing.
- RNA Binding Proteins that Control Human Papillomavirus Gene Expression.
- 2013
- Suppression of HPV-16 late L1 5'-splice site SD3632 by binding of hnRNP D proteins and hnRNP A2/B1 to upstream AUAGUA RNA motifs.