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- 2002
-
Mark
Factor V and thrombotic disease: description of a janus-faced protein.
(
- Contribution to journal › Scientific review
-
Mark
Defining the factor Xa-binding site on factor Va by site-directed glycosylation.
(
- Contribution to journal › Article
-
Mark
Structural requirements for the intracellular subunit polymerization of the complement inhibitor C4b-binding protein.
(
- Contribution to journal › Article
- 2001
-
Mark
C4bp binding to porin mediates stable serum resistance of Neisseria gonorrhoeae
(
- Contribution to journal › Scientific review
-
Mark
Screening the molecular surface of human anticoagulant protein C: a search for interaction sites
(
- Contribution to journal › Article
-
Mark
A novel interaction between type IV pili of Neisseria gonorrhoeae and the human complement regulator C4B-binding protein
(
- Contribution to journal › Article
-
Mark
Binding of C4b-binding protein: A molecular mechanism of serum resistance of Neisseria gonorrhoeae
(
- Contribution to journal › Article
-
Mark
Analytical considerations for free protein S assays in protein S deficiency
(
- Contribution to journal › Article
-
Mark
Proposed lipocalin fold for apolipoprotein M based on bioinformatics and site-directed mutagenesis
(
- Contribution to journal › Article
-
Mark
Deficiency or inhibition of Gas6 causes platelet dysfunction and protects mice against thrombosis
(
- Contribution to journal › Article
-
Mark
Three-dimensional model of the SHBG-like region of anticoagulant protein S: New structure-function insights
(
- Contribution to journal › Article
-
Mark
Improved hemoglobin status and reduced menstrual blood loss among female carriers of factor V Leiden--an evolutionary advantage?
(
- Contribution to journal › Letter
-
Mark
Localization of a hydrophobic binding site for anticoagulant protein S on the beta -chain of complement regulator C4b-binding protein
(
- Contribution to journal › Article
-
Mark
Structural and energetic characteristics of the heparin-binding site in antithrombotic protein C
(
- Contribution to journal › Article
-
Mark
Bordetella pertussis binds to human C4b-binding protein (C4BP) at a site similar to that used by the natural ligand C4b
(
- Contribution to journal › Article
-
Mark
Structural requirements for the complement regulatory activities of C4BP
(
- Contribution to journal › Article
- 2000
-
Mark
Använd hela "analyspaketet" vid utredning av trombospatienten! Och glöm inte släktingarna ...
(
- Contribution to journal › Article
- 1999
-
Mark
Probing the activation of protein C by the thrombin-thrombomodulin complex using structural analysis, site-directed mutagenesis, and computer modeling
(
- Contribution to journal › Article
-
Mark
Factor V Q506 (resistance to activated protein C) and prognosis after acute coronary syndrome
(
- Contribution to journal › Article
-
Mark
Thrombophilia as a multigenic disease
(
- Contribution to journal › Article
- 1998
-
Mark
Activated protein C resistance in patients with peripheral vascular disease
(
- Contribution to journal › Article
-
Mark
A common 4G allele in the promoter of the plasminogen activator inhibitor-1 (PAI-1) gene as a risk factor for pulmonary embolism and arterial thrombosis in hereditary protein S deficiency
(
- Contribution to journal › Article
-
Mark
A new direct, fast and quantitative enzyme-linked ligandsorbent assay for measurement of free protein S antigen
(
- Contribution to journal › Article
-
Mark
Clarification of the risk for venous thrombosis associated with hereditary protein S deficiency by investigation of a large kindred with a characterized gene defect
(
- Contribution to journal › Article
-
Mark
The A20210 allele of the prothrombin gene is frequently associated with the factor V Arg 506 to Gln mutation but not with protein S deficiency in thrombophilic families
(
- Contribution to journal › Letter
-
Mark
The A20210 allele of the prothrombin gene is frequently associated with the factor V Arg 506 to Gln mutation but not with protein S deficiency in thrombophilic families [3]
(
- Contribution to journal › Letter
-
Mark
Activation of the contact-phase system on bacterial surfaces - A clue to serious comlications in infections deseases
(
- Contribution to journal › Article
- 1997
-
Mark
The 20210 A allele of the prothrombin gene is a common risk factor among Swedish outpatients with verified deep venous thrombosis
(
- Contribution to journal › Debate/Note/Editorial
-
Mark
Genetic and phenotypic analysis of a large (122-member) protein S-deficient kindred provides an explanation for the familial coexistence of type I and type III plasma phenotypes
(
- Contribution to journal › Article
-
Mark
Activated protein C resistance caused by a common factor V mutation has a single origin
(
- Contribution to journal › Article
-
Mark
The factor VR506Q mutation causing APC resistance is highly prevalent amongst unselected outpatients with clinically suspected deep venous thrombosis
(
- Contribution to journal › Article
-
Mark
Evaluation of original and modified APC-resistance tests in unselected outpatients with clinically suspected thrombosis and in healthy controls
(
- Contribution to journal › Article
-
Mark
Activated protein C resistance due to a common factor V gene mutation is a major risk factor for venous thrombosis
(
- Contribution to journal › Article
-
Mark
The factor VR5O6Q mutation causing APC resistance is highly prevalent amongst unselected outpatients with clinically suspected deep venous thrombosis
(
- Contribution to journal › Article
-
Mark
Activated protein C resistance : Clinical implications
(
- Contribution to journal › Scientific review
-
Mark
The amino-terminal module of the C4b-binding protein alpha-chain is crucial for C4b binding and factor I-cofactor function
(
- Contribution to journal › Article
-
Mark
Molecular cloning of rat C4b binding protein alpha- and beta-chains : structural and functional relationships among human, bovine, rabbit, mouse, and rat proteins
(
- Contribution to journal › Article
- 1996
-
Mark
Elevated levels of prothrombin activation fragment 1 + 2 in plasma from patients with heterozygous Arg506 to Gln mutation in the factor V gene (APC-resistance) and/or inherited protein S deficiency
(
- Contribution to journal › Article
-
Mark
Resistance to activated protein C due to a factor V gene mutation : The most common inherited risk factor of thrombosis
(
- Contribution to journal › Article
-
Mark
Prevalence of factor V gene mutation amongst myocardial infarction patients and healthy controls is higher in Sweden than in other countries
(
- Contribution to journal › Article
-
Mark
Resistance to activated protein C, the FV:Q506 allele, and venous thrombosis
(
- Contribution to journal › Scientific review
-
Mark
High prevalence of the FVR506Q mutation causing APC resistance in a region of southern Sweden with a high incidence of venous thrombosis.
(
- Contribution to journal › Letter
-
Mark
Inherited resistance to activated protein C caused by presence of the FV:Q506 allele as a basis of venous thrombosis
(
- Contribution to journal › Article
-
Mark
Factor V:Q506 mutation and anticardiolipin antibodies in systemic lupus erythematosus
(
- Contribution to journal › Article
-
Mark
Activated protein C resistance as a basis for venous thrombosis.
(
- Contribution to journal › Article
- 1995
-
Mark
Ig-binding surface proteins of Streptococcus pyogenes also bind human C4b-binding protein (C4BP), a regulatory component of the complement system
(
- Contribution to journal › Article
-
Mark
Activated protein C resistance : from phenotype to genotype and clinical practice
(
- Contribution to journal › Article
-
Mark
Resistens mot aktiverat protein C. Vanlig genetisk riskfaktor för venös trombos
(
- Contribution to journal › Article
-
Mark
Resistance to activated protein C caused by a factor V gene mutation
(
- Contribution to journal › Article
-
Mark
Evaluation of the relationship between protein S and C4b-binding protein isoforms in hereditary protein S deficiency demonstrating type I and type III deficiencies to be phenotypic variants of the same genetic disease
(
- Contribution to journal › Article