Madeleine Durbeej-Hjalt
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- 2022
-
Mark
Skeletal muscle regeneration is altered in the R6/2 mouse model of Huntington’s disease
2022) p.1-23(
- Working paper/Preprint › Preprint in preprint archive
- 2021
-
Mark
A guide to the composition and functions of the extracellular matrix
(
- Contribution to journal › Article
-
Mark
Merosin deficient congenital muscular dystrophy type 1A : An international workshop on the road to therapy 15-17 November 2019, Maastricht, the Netherlands
(
- Contribution to journal › Article
- 2020
-
Mark
A Family of Laminin α2 Chain-Deficient Mouse Mutants : Advancing the Research on LAMA2-CMD
(
- Contribution to journal › Scientific review
-
Mark
Antioxidants reduce muscular dystrophy in the dy2J/dy2J mouse model of laminin α2 chain-deficient muscular dystrophy
(
- Contribution to journal › Article
- 2019
-
Mark
A mutation-independent approach for muscular dystrophy via upregulation of a modifier gene
(
- Contribution to journal › Letter
-
Mark
Effects of N-acetyl-L-cysteine and vitamin E on congenital muscular dystrophy type 1A disease progression in mice
(
- Chapter in Book/Report/Conference proceeding › Paper in conference proceeding
- 2018
-
Mark
Laminin α1 reduces muscular dystrophy in dy2Jmice
(
- Contribution to journal › Article
-
Mark
Effects of metformin on congenital muscular dystrophy type 1A disease progression in mice: a gender impact study
2018) In Scientific Reports(
- Contribution to journal › Article
-
Mark
Exploratory Profiling of Urine MicroRNAs in the dy2J/dy2J Mouse Model of LAMA2-CMD : Relation to Disease Progression
(
- Contribution to journal › Article
- 2017
-
Mark
Potent pro-inflammatory and profibrotic molecules, osteopontin and galectin-3, are not major disease modulators of laminin α2 chaindeficient muscular dystrophy
(
- Contribution to journal › Article
-
Mark
Bioenergetic Impairment in Congenital Muscular Dystrophy Type 1A and Leigh Syndrome Muscle Cells
(
- Contribution to journal › Article
-
Mark
Absence of microRNA-21 does not reduce muscular dystrophy in mouse models of LAMA2-CMD
(
- Contribution to journal › Article
- 2016
-
Mark
Bortezomib Does Not Reduce Muscular Dystrophy in the dy2J/dy2J Mouse Model of Laminin α2 Chain-Deficient Muscular Dystrophy.
(
- Contribution to journal › Article
-
Mark
212th ENMC International Workshop:: Animal models of congenital muscular dystrophies, Naarden, The Netherlands, 29-31 May 2015.
(
- Contribution to journal › Article
- 2015
-
Mark
Deletion of integrin α7 subunit does not aggravate the phenotype of laminin α2 chain-deficient mice.
(
- Contribution to journal › Article
-
Mark
Dystrophin deficiency reduces atherosclerotic plaque development in ApoE-null mice.
(
- Contribution to journal › Article
-
Mark
Laminin-α2 Chain-Deficient Congenital Muscular Dystrophy: Pathophysiology and Development of Treatment.
(
- Contribution to journal › Article
- 2014
-
Mark
Bortezomib Partially Improves Laminin α2 Chain-Deficient Muscular Dystrophy.
(
- Contribution to journal › Article
-
Mark
Laminin α2 Chain-Deficiency is Associated with microRNA Deregulation in Skeletal Muscle and Plasma.
(
- Contribution to journal › Article
-
Mark
Quantitative proteomic analysis reveals metabolic alterations, calcium dysregulation and increased expression of extracellular matrix proteins in laminin α2 chain-deficient muscle.
(
- Contribution to journal › Article
-
Mark
Loss of Dystrophin and β-Sarcoglycan, Respectively, Significantly Exacerbates the Phenotype of Laminin α2 Chain-Deficient Animals.
(
- Contribution to journal › Article
- 2013
-
Mark
Isobaric Tagging-Based Quantification for Proteomic Analysis: A Comparative Study of Spared and Affected Muscles from mdx Mice at the Early Phase of Dystrophy
(
- Contribution to journal › Article
-
Mark
Evaluation of macroporous blood and plasma scaffolds for skeletal muscle tissue engineering
(
- Contribution to journal › Article
- 2012
-
Mark
Increased Neointimal Thickening in Dystrophin-Deficient mdx Mice.
(
- Contribution to journal › Article
-
Mark
Guidelines for the use and interpretation of assays for monitoring autophagy
(
- Contribution to journal › Scientific review
-
Mark
Cell-matrix interactions in muscle disease.
(
- Contribution to journal › Article
-
Mark
Laminin-211 in skeletal muscle function.
(
- Contribution to journal › Article
-
Mark
Porous protein-based scaffolds prepared through freezing as potential scaffolds for tissue engineering.
(
- Contribution to journal › Article
- 2011
-
Mark
Transgenic Expression of Laminin α1 Chain Does Not Prevent Muscle Disease in the mdx Mouse Model for Duchenne Muscular Dystrophy.
(
- Contribution to journal › Article
-
Mark
Proteasome Inhibition Improves the Muscle of Laminin {alpha}2 Chain Deficient Mice.
(
- Contribution to journal › Article
-
Mark
Autophagy is increased in laminin {alpha}2 chain-deficient muscle and its inhibition improves muscle morphology in a mouse model of MDC1A.
(
- Contribution to journal › Article
-
Mark
Laminin isoforms in atherosclerotic arteries from mice and man.
(
- Contribution to journal › Article
-
Mark
Skeletal muscle laminin and MDC1A: pathogenesis and treatment strategies.
(
- Contribution to journal › Article
- 2010
-
Mark
Transgenic overexpression of laminin 1 chain in laminin 2 chain-deficient mice rescues the disease throughout the lifespan
(
- Contribution to journal › Article
-
Mark
Laminins.
(
- Contribution to journal › Article
-
Mark
Distinct roles for laminin globular domains in laminin alpha1 chain mediated rescue of murine laminin alpha2 chain deficiency.
(
- Contribution to journal › Article
- 2009
-
Mark
E3 domain of laminin alpha 1 chain that binds to dystroglycan is not essential for survival and muscle regeneration in laminin alpha 1 chain mediated correction of laminin alpha 2 chain deficiency
2009) 14th International Congress of the World-Muscle-Society In Neuromuscular Disorders 19(8-9). p.598-599(
- Contribution to journal › Published meeting abstract
-
Mark
Intracellular signaling pathway alterations in laminin alpha 2 chain deficient skeletal muscle and brain
2009) 14th International Congress of the World-Muscle-Society In Neuromuscular Disorders 19(8-9). p.574-574(
- Contribution to journal › Published meeting abstract
-
Mark
Substantial improvement of laminin alpha 2 chain deficiency by laminin alpha 1 chain overexpression persists throughout life
2009) 14th International Congress of the World-Muscle-Society In Neuromuscular Disorders 19(8-9). p.618-618(
- Contribution to journal › Published meeting abstract
-
Mark
Laminin alpha1 domains LG4-5 are essential for the complete differentiation of visceral endoderm.
(
- Contribution to journal › Article
-
Mark
Intrinsic laryngeal muscles are spared from degeneration in the dy(3k)/dy(3k) mouse model of congenital muscular dystrophy type 1A.
(
- Contribution to journal › Article
- 2008
-
Mark
Cib2 binds integrin a7Bb1D and is reduced in laminin a2 chain deficient muscular dystrophy
(
- Contribution to journal › Article
-
Mark
Expression of the novel gene Ened during mouse and Xenopus embryonic development.
(
- Contribution to journal › Article
- 2007
-
Mark
Dystroglycan: a possible mediator for reducing congenital muscular dystrophy?
(
- Contribution to journal › Scientific review
-
Mark
Laminin isoforms in development and disease.
(
- Contribution to journal › Scientific review
-
Mark
Role of the extracellular matrix and its receptors in smooth muscle cell function: implications in vascular development and disease
(
- Contribution to journal › Scientific review
-
Mark
Gene expression profiling of differentiating embryonic stem cells expressing dominant negative fibroblast growth factor receptor 2.
(
- Contribution to journal › Article
-
Mark
Compositional differences between infant and adult human corneal basement membranes
(
- Contribution to journal › Article
- 2006
-
Mark
Laminin {alpha}1 chain improves laminin {alpha}2 chain deficient peripheral neuropathy.
(
- Contribution to journal › Article
-
Mark
Dystroglycan is selectively cleaved at the parenchymal basement membrane at sites of leukocyte extravasation in experimental autoimmune encephalomyelitis.
(
- Contribution to journal › Article
-
Mark
Extraocular muscle is spared upon complete laminin alpha2 chain deficiency: Comparative expression of laminin and integrin isoforms.
(
- Contribution to journal › Article
-
Mark
Monoclonal anti-mouse laminin antibodies: AL-1 reacts with laminin alpha1 chain, AL-2 with laminin beta1 chain, and AL-4 with the coiled-coil domain of laminin beta1 chain.
(
- Contribution to journal › Article
-
Mark
Laminin alpha1 chain mediated reduction of laminin alpha2 chain deficient muscular dystrophy involves integrin alpha7beta1 and dystroglycan.
(
- Contribution to journal › Article
- 2005
-
Mark
Laminin {alpha}1 Chain Corrects Male Infertility Caused by Absence of Laminin {alpha}2 Chain.
(
- Contribution to journal › Article
-
Mark
Distinctive functions of membrane type 1 matrix-metalloprotease (MT1-MMP or MMP-14) in lung and submandibular gland development are independent of its role in pro-MMP-2 activation
(
- Contribution to journal › Article
- 2004
-
Mark
Laminin {alpha}1 chain reduces muscular dystrophy in laminin {alpha}2 chain deficient mice.
(
- Contribution to journal › Article
- 2003
-
Mark
Gene transfer establishes primacy of striated vs. smooth muscle sarcoglycan complex in limb-girdle muscular dystrophy
(
- Contribution to journal › Article
- 2002
-
Mark
Nulp1, a novel basic helix-loop-helix protein expressed broadly during early embryonic organogenesis and prominently in developing dorsal root ganglia.
(
- Contribution to journal › Article
-
Mark
Muscular dystrophies involving the dystrophin-glycoprotein complex: an overview of current mouse models
(
- Contribution to journal › Article
- 1999
-
Mark
Characterization of bone marrow laminins and identification of alpha5-containing laminins as adhesive proteins for multipotent hematopoietic FDCP-Mix cells
(
- Contribution to journal › Article
- 1997
-
Mark
Transient expression of Dp140, a product of the Duchenne muscular dystrophy locus, during kidney tubulogenesis
(
- Contribution to journal › Article
- 1996
-
Mark
Expression of laminin alpha 1, alpha 5 and beta 2 chains during embryogenesis of the kidney and vasculature
(
- Contribution to journal › Article