1 – 41 of 41
- show: 100
- |
- sort: year (new to old)
Close
Embed this list
<iframe src=" "
width=" "
height=" "
allowtransparency="true"
frameborder="0">
</iframe>
- 2024
-
Mark
LOX-1 and MMP-9 Inhibition Attenuates the Detrimental Effects of Delayed rt-PA Therapy and Improves Outcomes after Acute Ischemic Stroke
(
- Contribution to journal › Article
- 2022
-
Mark
Repair-related molecular changes during recovery phase of ischemic stroke in female rats
(
- Contribution to journal › Article
-
Mark
Changes in P2Y6 receptor-mediated vasoreactivity following focal and global ischemia
(
- Contribution to journal › Article
-
Mark
Short-term exposure to urban PM2.5 particles induces histopathological and inflammatory changes in the rat small intestine
(
- Contribution to journal › Article
-
Mark
Airway exposure to urban aerosolized PM2.5 particles induces neuroinflammation and endothelin-mediated contraction of coronary arteries in adult rats
(
- Contribution to journal › Article
-
Mark
Purkinje cell vulnerability induced by diffuse traumatic brain injury is linked to disruption of long-range neuronal circuits
(
- Contribution to journal › Article
- 2021
-
Mark
An Improved Method for Physical Separation of Cerebral Vasculature and Parenchyma Enables Detection of Blood-Brain-Barrier Dysfunction
(
- Contribution to journal › Article
-
Mark
Combination treatment with U0126 and rt-PA prevents adverse effects of the delayed rt-PA treatment after acute ischemic stroke
(
- Contribution to journal › Article
- 2020
-
Mark
Validation of a stroke model in rat compatible with rt-PA-induced thrombolysis : new hope for successful translation to the clinic
(
- Contribution to journal › Article
-
Mark
Transcriptome profiling revealed early vascular smooth muscle cell gene activation following focal ischemic stroke in female rats – comparisons with males
(
- Contribution to journal › Article
- 2019
-
Mark
Stimulating brain recovery after stroke using theranostic albumin nanocarriers loaded with nerve growth factor in combination therapy
(
- Contribution to journal › Article
- 2018
-
Mark
Contractile Responses in Spontaneously Hypertensive Rats after Transient Middle Cerebral Artery Occlusion
(
- Contribution to journal › Article
- 2017
-
Mark
Cerebrovascular gene expression in spontaneously hypertensive rats
(
- Contribution to journal › Article
-
Mark
Cerebrovascular Gene Expression in Spontaneously Hypertensive Rats After Transient Middle Cerebral Artery Occlusion
(
- Contribution to journal › Article
- 2016
-
Mark
Investigating potentially salvageable penumbra tissue in an in vivo model of transient ischemic stroke using sodium, diffusion, and perfusion magnetic resonance imaging
(
- Contribution to journal › Article
- 2015
-
Mark
Inhibition of mitogen-activated protein kinase 1/2 in the acute phase of stroke improves long-term neurological outcome and promotes recovery processes in rats.
2015) In Acta Physiologica(
- Contribution to journal › Article
-
Mark
U0126 attenuates cerebral vasoconstriction and improves long-term neurologic outcome after stroke in female rats.
(
- Contribution to journal › Article
- 2013
-
Mark
MAPK signaling pathway regulates cerebrovascular receptor expression in human cerebral arteries
(
- Contribution to journal › Article
- 2012
-
Mark
Sodium-23 magnetic resonance imaging during and after transient cerebral ischemia: multinuclear stroke protocols for double-tuned Na-23/H-1 resonator systems
(
- Contribution to journal › Article
- 2011
-
Mark
Inhibition of cerebrovascular raf activation attenuates cerebral blood flow and prevents upregulation of contractile receptors after subarachnoid hemorrhage
(
- Contribution to journal › Article
-
Mark
Blockade of the MEK/ERK pathway with a raf inhibitor prevents activation of pro-inflammatory mediators in cerebral arteries and reduction in cerebral blood flow after subarachnoid hemorrhage in a rat model.
2011) In Journal of cerebral blood flow and metabolism : official journal of the International Society of Cerebral Blood Flow and Metabolism May 4. p.144-154(
- Contribution to journal › Article
- 2010
-
Mark
Comparison of MEK/ERK pathway inhibitors on the upregulation of vascular G-protein coupled receptors in rat cerebral arteries
(
- Contribution to journal › Article
-
Mark
Subarachnoid Hemorrhage Induces Enhanced Expression of Thromboxane A(2) Receptors in Rat Cerebral Arteries.
(
- Contribution to journal › Article
- 2009
-
Mark
Equal contribution of increased intracranial pressure and subarachnoid blood to cerebral blood flow reduction and receptor upregulation after subarachnoid hemorrhage.
(
- Contribution to journal › Article
-
Mark
RAF inhibition prevents activation of pro-inflammatory mediators in cerebral arteries after subarachnoid hemorrhage
2009) 24th International Symposium on Cerebral Blood Flow and Metabolism/9th International Conference on Quantification of Brain Function with PET In Journal of Cerebral Blood Flow and Metabolism 29. p.305-305(
- Contribution to journal › Published meeting abstract
-
Mark
Cerebral ischemia enhances vascular thromboxane receptor expressions
2009) 24th International Symposium on Cerebral Blood Flow and Metabolism/9th International Conference on Quantification of Brain Function with PET In Journal of Cerebral Blood Flow and Metabolism 29. p.307-307(
- Contribution to journal › Published meeting abstract
-
Mark
MAPK signalling pathway regulate cerebrovascular receptor expression in human cerebral arteries
2009) 24th International Symposium on Cerebral Blood Flow and Metabolism/9th International Conference on Quantification of Brain Function with PET In Journal of Cerebral Blood Flow and Metabolism 29. p.495-496(
- Contribution to journal › Published meeting abstract
- 2008
-
Mark
Elevated intracranial pressure or subarachnoid blood responsible for reduction in cerebral blood flow after SAH
(
- Chapter in Book/Report/Conference proceeding › Paper in conference proceeding
-
Mark
Subtype activation and interaction of protein kinase C and mitogen-activated protein kinase controlling receptor expression in cerebral arteries and microvessels after subarachnoid hemorrhage
(
- Contribution to journal › Article
-
Mark
Subarachnoid hemorrhage induces upregulation of vascular receptors and reduction in rCBF via an ERKI/2 mechanism
(
- Chapter in Book/Report/Conference proceeding › Paper in conference proceeding
-
Mark
Cerebral vasoconstriction after subarachnoid hemorrhage - Role of changes in vascular receptor phenotype
(
- Contribution to journal › Article
- 2007
-
Mark
Protein Kinase Inhibition in Late Cerebral Ischemia after Subarachnoid Hemorrhage
2007)(
- Thesis › Doctoral thesis (compilation)
-
Mark
Vasospasm - Response
(
- Contribution to journal › Debate/Note/Editorial
-
Mark
Transcriptional regulation of inflammatory and extracellular matrix-regulating genes in experimental subarachnoid cerebral arteries following hemorrhage in rats
(
- Contribution to journal › Article
-
Mark
Neurokinin-1 receptor antagonism in a rat model of subarachnoid hemorrhage: prevention of upregulation of contractile ETB and 5-HT1B receptors and cerebral blood flow reduction
(
- Contribution to journal › Article
-
Mark
Cerebrovascular ETB, 5-HT1B and AT1 Receptor Upregulation Correlates with Reduction in Regional CBF after Subarachnoid Hemorrhage.
(
- Contribution to journal › Article
-
Mark
Cerebral ischemia induces transcription of inflammatory and extracellular-matrix-related genes in rat cerebral arteries.
(
- Contribution to journal › Article
-
Mark
Protein kinase C inhibition prevents upregulation of vascular ET(B) and 5-HT(1B) receptors and reverses cerebral blood flow reduction after subarachnoid haemorrhage in rats.
(
- Contribution to journal › Article
- 2006
-
Mark
Inhibition of PKC activity blocks the increase of ETB receptor expression in cerebral arteries.
(
- Contribution to journal › Article
-
Mark
Gene expression and molecular changes in cerebral arteries following subarachnoid hemorrhage in the rat.
(
- Contribution to journal › Article
-
Mark
ERK1/2 inhibition attenuates cerebral blood flow reduction and abolishes ET(B) and 5-HT(1B) receptor upregulation after subarachnoid hemorrhage in rat.
(
- Contribution to journal › Article