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- 2011
-
Mark
Differential Glucose-Regulation of MicroRNAs in Pancreatic Islets of Non-Obese Type 2 Diabetes Model Goto-Kakizaki Rat.
- Contribution to journal › Article
-
Mark
Beta-cell specific deletion of dicer1 leads to defective insulin secretion and diabetes mellitus.
- Contribution to journal › Article
-
Mark
Dissecting the role of micro-RNAs in diabetes
- Thesis › Doctoral thesis (compilation)
- 2010
-
Mark
Apoptosis resistance of non-obese diabetic mouse thymocytes is mediated by a defective p53 expression and altered microRNA network controlling cell death and proliferation
(2010) 46th Annual Meeting of the European-Association-for-the- Study-of-Diabetes (EASD) In Diabetologia 53(Suppl. 1). p.419-419
- Contribution to journal › Published meeting abstract
-
Mark
MicroRNA differences in human islets and glucose-sensitive tissues
(2010) 46th Annual Meeting of the European-Association-for-the- Study-of-Diabetes (EASD) In Diabetologia 53(Suppl. 1). p.527-527
- Contribution to journal › Published meeting abstract
- 2009
-
Mark
Predicted targets of co-expressed microRNAs in pancreatic islets of GK rats are enriched for genes involved in regulation of beta cell mass and glucose-stimulated insulin secretion
(2009) 45th Annual Meeting of the European-Association-for-the-Study-of-Diabetes In Diabetologia 52(Suppl 1). p.391-391
- Contribution to journal › Published meeting abstract
- 2008
-
Mark
Regulatory T cell-associated activity in photopheresis-induced immune tolerance in recent onset type 1 diabetes children
- Contribution to journal › Article
- 2007
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Mark
CT60 genotype does not affect CTLA-4 isoform expression despite association to TID and AITD in northern Sweden
- Contribution to journal › Article
- 1995
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Mark
Molecular characterization of human monoclonal antibodies specific for the human cytomegalovirus : Relationship of variable region sequence to antigen specificity and rheumatoid factor-associated idiotype expression
- Contribution to journal › Article
-
Mark
Generation of diversity in a human anti-viral response
- Contribution to journal › Article
