Lars-Åke Fransson
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- 2024
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Mark
Interplay between glypican-1, amyloid-β and tau phosphorylation in human neural stem cells
(
- Contribution to journal › Article
- 2023
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Mark
Interplay between APP and glypican-1 processing and α-synuclein aggregation in undifferentiated and differentiated human neural progenitor cells
(
- Contribution to journal › Article
- 2022
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Mark
Complex modulation of cytokine-induced α-synuclein aggregation by glypican-1-derived heparan sulfate in neural cells
(
- Contribution to journal › Article
- 2021
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Mark
Reversal of apolipoprotein E4-dependent or chemical-induced accumulation of APP degradation products by vitamin C-induced release of heparan sulfate from glypican-1
(
- Contribution to journal › Article
- 2020
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Mark
Proinflammatory cytokines induce accumulation of glypican-1-derived heparan sulfate and the C-terminal fragment of β-cleaved APP in autophagosomes of dividing neuronal cells
(
- Contribution to journal › Article
- 2019
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Mark
The cyanobacterial neurotoxin β-N-methylamino-L-alanine prevents addition of heparan sulfate to glypican-1 and increases processing of amyloid precursor protein in dividing neuronal cells
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- Contribution to journal › Article
- 2018
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Mark
Common traffic routes for imported spermine and endosomal glypican-1-derived heparan sulfate in fibroblasts
(
- Contribution to journal › Article
- 2017
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Mark
Cytochrome b561, copper, β-cleaved amyloid precursor protein and niemann-pick C1 protein are involved in ascorbate-induced release and membrane penetration of heparan sulfate from endosomal S-nitrosylated glypican-1
(
- Contribution to journal › Article
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Mark
Nucleolin is a nuclear target of heparan sulfate derived from glypican-1
(
- Contribution to journal › Article
- 2016
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Mark
Hypoxia induces NO-dependent release of heparan sulfate in fibroblasts from the Alzheimer mouse Tg2576 by activation of nitrite reduction.
(
- Contribution to journal › Article
- 2015
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Mark
Rapid nuclear transit and impaired degradation of amyloid beta and glypican-1-derived heparan sulfate in Tg2576 mouse fibroblasts.
(
- Contribution to journal › Article
-
Mark
Suppression of glypican-1 autodegradation by NO-deprivation correlates with nuclear accumulation of amyloid beta in normal fibroblasts.
(
- Contribution to journal › Article
- 2014
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Mark
APP/APLP2 expression is required to initiate endosome-nucleus-autophagosome trafficking of glypican-1-derived heparan sulfate.
(
- Contribution to journal › Article
- 2013
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Mark
Non-toxic amyloid beta formed in the presence of glypican-1 or its deaminatively generated heparan sulfate degradation products
(
- Contribution to journal › Article
- 2012
-
Mark
Non-conserved, S-nitrosylated cysteines in glypican-1 react with N-unsubstituted glucosamines in heparan sulfate and catalyze deaminative cleavage.
(
- Contribution to journal › Article
- 2011
-
Mark
Suppression of amyloid beta a11-immunoreactivity by vitamin C: possible role of heparan sulfate oligosaccharides derived from glypican-1 by ascorbate-induced, no-catalyzed degradation.
(
- Contribution to journal › Article
- 2010
-
Mark
Attenuation of tumor growth by formation of antiproliferative glycosaminoglycans correlates with low acetylation of histone H3.
(
- Contribution to journal › Article
- 2009
-
Mark
Potentiation of naphthoxyloside cytotoxicity on human tumor cells by difluoromethylornithine and spermine-NONOate.
(
- Contribution to journal › Article
- 2008
-
Mark
Involvement of glypican-1 autoprocessing in scrapie infection
(
- Contribution to journal › Article
- 2007
-
Mark
Heparan sulfate degradation products can associate with oxidized proteins and proteasomes.
(
- Contribution to journal › Article
-
Mark
Novel aspects of vitamin C: how important is glypican-1 recycling?
(
- Contribution to journal › Scientific review
- 2006
-
Mark
Constitutive and vitamin C-induced, NO-catalyzed release of heparan sulfate from recycling glypican-1 in late endosomes.
(
- Contribution to journal › Article
-
Mark
Defective NO-dependent, deaminative cleavage of glypican-1 heparan sulfate in Niemann-Pick C1 fibroblasts.
(
- Contribution to journal › Article
- 2005
-
Mark
The amyloid precursor protein(APP)of Alzheimer's disease and its paralog APLP2 modulate the Cu/Zn-NO-catalyzed degradation of glypican-1 heparan sulfate In vivo.
(
- Contribution to journal › Article
- 2004
-
Mark
Involvement of GPI-linked ceruloplasmin in the Cu/Zn-NO-dependent degradation of glypican-1 heparan sulfate in Rat C6 glioma cells.
(
- Contribution to journal › Article
-
Mark
Tumor attenuation by 2(6-hydroxynaphthyl)-{beta}-D-xylopyranoside requires priming of heparan sulfate and nuclear targeting of the products.
(
- Contribution to journal › Article
-
Mark
The heparan sulfate-specific epitope 10E4 is NO-sensitive and partly inaccessible in glypican-1.
(
- Contribution to journal › Article
-
Mark
Novel aspects of glypican glycobiology.
(
- Contribution to journal › Scientific review
- 2003
-
Mark
Glypican-1 is a vehicle for polyamine uptake in mammalian cells. A pivotal role for nitrosothiol-derived nitric oxide.
(
- Contribution to journal › Article
-
Mark
Initiation of the decorin glycosaminoglycan chain in the endoplasmic reticulum-Golgi intermediate compartment
(
- Contribution to journal › Article
-
Mark
Prion or amyloid-b-derived Cu(II)- or free Zn(II)-ions support S-nitroso-dependent autocleavage of glypican-1 heparan sulfate.
(
- Contribution to journal › Article
-
Mark
Glypicans.
(
- Contribution to journal › Article
- 2002
-
Mark
Tumor attenuation by combined heparan sulfate and polyamine depletion.
(
- Contribution to journal › Article
-
Mark
Nitric oxide-dependent processing of heparan sulfate in recycling S-nitrosylated glypican-1 takes place in caveolin-1 containing endosomes.
(
- Contribution to journal › Article
-
Mark
Copper-dependent autocleavage of glypican-1heparan sulfate by nitric oxide derived fromintrinsic nitrosothiols.
(
- Contribution to journal › Article
- 2001
-
Mark
Modulations of glypican-1 heparan sulfate structure by inhibition of endogenous polyamine synthesis. Mapping of spermine-binding sites and heparanase, heparin lyase, and nitric oxide/nitrite cleavage sites
(
- Contribution to journal › Article
-
Mark
N-unsubstituted glucosamine in heparan sulfate of recycling glypican-1 from suramin-treated and nitrite-deprived endothelial cells. mapping of nitric oxide/nitrite-susceptible glucosamine residues to clustered sites near the core protein
(
- Contribution to journal › Article
- 1999
-
Mark
Proteoglycan involvement in polyamine uptake
(
- Contribution to journal › Article
- 1998
-
Mark
Heparan/chondroitin/dermatan sulfate primer 2-(6-hydroxynaphthyl)-O-beta-D-xylopyranoside preferentially inhibits growth of transformed cells
(
- Contribution to journal › Article
- 1997
-
Mark
Binding, internalization, and degradation of antiproliferative heparan sulfate by human embryonic lung fibroblasts
(
- Contribution to journal › Article
-
Mark
Biosynthesis of the proteoglycan decorin -- identification of intermediates in galactosaminoglycan assembly
(
- Contribution to journal › Article
- 1996
-
Mark
Heparan sulphate/heparin glycosaminoglycans with strong affinity for the growth-promoter spermine have high antiproliferative activity
(
- Contribution to journal › Article
- 1995
-
Mark
Recycling of a glycosylphosphatidylinositol-anchored heparan sulphate proteoglycan (glypican) in skin fibroblasts
(
- Contribution to journal › Article
- 1994
-
Mark
Patterns of uronosyl epimerization and 4-/6-O-sulphation in chondroitin/dermatan sulphate from decorin and biglycan of various bovine tissues
(
- Contribution to journal › Article
-
Mark
Analysis of heparan-sulphate chains and oligosaccharides from proliferating and quiescent fibroblasts. A proposed model for endoheparanase activity
(
- Contribution to journal › Article
- 1993
-
Mark
Hydrophobic interaction chromatography of fibroblast proteoglycans
(
- Contribution to journal › Scientific review
-
Mark
The growth promoter spermine interacts specifically with dermatan sulfate regions that are rich inl-iduronic acid and possess antiproliferative activity
(
- Contribution to journal › Article
-
Mark
L-iduronate-rich glycosaminoglycans inhibit growth of normal fibroblasts independently of serum or added growth factors
(
- Contribution to journal › Article
- 1992
-
Mark
Analysis of glycosaminoglycan chains from different proteoglycan populations in human embryonic skin fibroblasts
(
- Contribution to journal › Article
-
Mark
Transforming growth factor-beta induces selective increase of proteoglycan production and changes in the copolymeric structure of dermatan sulphate in human skin fibroblasts
(
- Contribution to journal › Article
- 1991
-
Mark
Oligosaccharide mapping of proteoglycan-bound and xyloside-initiated dermatan sulfate from fibroblasts
(
- Contribution to journal › Article
-
Mark
Proliferation of cultured fibroblasts is inhibited by L-iduronate-containing glycosaminoglycans
(
- Contribution to journal › Article
- 1990
-
Mark
Inventory of human skin fibroblast proteoglycans. Identification of multiple heparan and chondroitin/dermatan sulphate proteoglycans
(
- Contribution to journal › Article
-
Mark
A fibroblast heparan sulphate proteoglycan with a 70 kDa core protein is linked to membrane phosphatidylinositol
(
- Contribution to journal › Article
-
Mark
TGF-beta enhances the production of hyaluronan in human lung but not in skin fibroblasts
(
- Contribution to journal › Article
- 1986
-
Mark
Structure of proteoheparan sulfates from fibroblasts. Confluent and proliferating fibroblasts produce at least three types of proteoheparan sulfates with functionally different core proteins
(
- Contribution to journal › Article