Diabetes - Islet Patophysiology
201 – 210 of 240
- show: 10
- |
- sort: year (new to old)
Close
Embed this list
<iframe src=" "
width=" "
height=" "
allowtransparency="true"
frameborder="0">
</iframe>
- 2005
-
Mark
Redox control of exocytosis - Regulatory role of NADPH, thioredoxin, and glutaredoxin
(
- Contribution to journal › Article
-
Mark
CaV2.3 calcium channels control second-phase insulin release.
(
- Contribution to journal › Article
-
Mark
The ablation of the Ca(v)2.3/E-type voltage-gated Ca2+ channel causes a mild phenotype despite an altered glucose induced glucagon response in isolated islets of Langerhans
(
- Contribution to journal › Article
-
Mark
Characterization of the PCR inhibitory effect of bile to optimize real-time PCR detection of Helicobacter species.
(
- Contribution to journal › Article
-
Mark
The R6/2 transgenic mouse model of Huntington's disease develops diabetes due to deficient {beta}-cell mass and exocytosis.
(
- Contribution to journal › Article
-
Mark
Center of volume and total heart volume variation in healthy subjects and patients before and after coronary bypass surgery.
(
- Contribution to journal › Article
-
Mark
Myosin 5a controls insulin granule recruitment during late-phase secretion.
(
- Contribution to journal › Article
- 2004
-
Mark
Large dense-core vesicle exocytosis in pancreatic beta-cells monitored by capacitance measurements
(
- Contribution to journal › Article
-
Mark
Hormone-sensitive Lipase deficiency in mouse islets abolishes neutral cholesterol ester hydrolase activity but leaves lipolysis, acylglycerides, fat oxidation, and insulin secretion intact.
(
- Contribution to journal › Article
-
Mark
Enhanced cAMP/Protein Kinase A signaling determines improved insulin secretion in a clonal insulin-producing {beta}-cell line (INS-1 832/13).
(
- Contribution to journal › Article