Diabetes - Islet Patophysiology
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- 2019
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Mark
Complement Component C3 Is Highly Expressed in Human Pancreatic Islets and Prevents β Cell Death via ATG16L1 Interaction and Autophagy Regulation
(
- Contribution to journal › Article
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Mark
Intracellular cytosolic complement component C3 regulates cytoprotective autophagy in pancreatic beta cells by interaction with ATG16L1
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- Contribution to journal › Debate/Note/Editorial
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Mark
Prevention of Pancreatic β-Cell Failure in Type 2 Diabetes. By Targeting a Mitochondrial Voltage Gated Channel 1 and a Novel class of GPCRs.
(
- Thesis › Doctoral thesis (compilation)
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Mark
The Best Leading Role for Maintaining Normal β-cell Function Goes to MafA and Voltage-gated Calcium Channel Subunit γ4
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- Thesis › Doctoral thesis (compilation)
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Mark
The N680S variant in the follicle-stimulating hormone receptor gene identifies hyperresponders to controlled ovarian stimulation
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- Contribution to journal › Article
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Mark
Expression levels of enzymes generating NO and CO in islets of murine and human diabetes
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- Contribution to journal › Article
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Mark
Simvastatin downregulates adipogenesis in 3T3-l1 preadipocytes and orbital fibroblasts from graves’ ophthalmopathy patients
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- Contribution to journal › Article
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Mark
Loss of ZnT8 function protects against diabetes by enhanced insulin secretion
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- Contribution to journal › Article
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Mark
Pancreatic β-cell mRNA modification as a marker for type 2 diabetes
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- Contribution to journal › Scientific review
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Mark
GNAS gene is an important regulator of insulin secretory capacity in pancreatic β-cells
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- Contribution to journal › Article