Lund Cardiac MR Group
381 – 390 of 551
- show: 10
- |
- sort: year (new to old)
Close
Embed this list
<iframe src=""
width=""
height=""
allowtransparency="true"
frameborder="0">
</iframe>
- 2013
-
Mark
Long-term impact of postconditioning on infarct size and left ventricular ejection fraction in patients with ST-elevation myocardial infarction
- Contribution to journal › Article
-
Mark
Myocardial infarction with normal coronary arteries is common and associated with normal findings on cardiovascular magnetic resonance imaging: results from the Stockholm Myocardial Infarction with Normal Coronaries study
- Contribution to journal › Article
-
Mark
Reduced administered activity, reduced acquisition time, and preserved image quality for the new CZT camera.
- Contribution to journal › Article
-
Mark
Quantification of left and right atrial kinetic energy using four-dimensional intracardiac magnetic resonance imaging flow measurements.
- Contribution to journal › Article
-
Mark
Pulmonary blood volume indexed to lung volume is reduced in newly diagnosed systemic sclerosis compared to normals - a prospective clinical cardiovascular magnetic resonance study addressing pulmonary vascular changes
- Contribution to journal › Article
-
Mark
A longitudinal study on cardiac effects of deconditioning and physical reconditioning using the anterior cruciate ligament injury as a model.
- Contribution to journal › Article
-
Mark
Accuracy of four-dimensional phase-contrast velocity mapping for blood flow visualizations: a phantom study.
- Contribution to journal › Article
-
Mark
Head-to-head comparison of a 2-day myocardial perfusion gated SPECT protocol and cardiac magnetic resonance late gadolinium enhancement for the detection of myocardial infarction.
- Contribution to journal › Article
-
Mark
Restrictive right ventricular physiology after Tetralogy of Fallot repair is associated with fibrosis of the right ventricular outflow tract visualized on cardiac magnetic resonance imaging.
- Contribution to journal › Article
-
Mark
Atrial aspiration from pulmonary and caval veins is caused by ventricular contraction and secures 70% of the total stroke volume independent of resting heart rate and heart size.
- Contribution to journal › Article
