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- 2024
-
Mark
Hepcidin, in contrast to heparin binding protein, does not portend acute kidney injury in patients with community acquired septic shock
(
- Contribution to journal › Article
-
Mark
The role of extracellular vesicle fusion with target cells in triggering systemic inflammation
(
- Contribution to journal › Article
- 2023
-
Mark
Therapeutic S100A8/A9 blockade inhibits myocardial and systemic inflammation and mitigates sepsis-induced myocardial dysfunction
(
- Contribution to journal › Article
-
Mark
Identifying biomarkers deciphering sepsis from trauma-induced sterile inflammation and trauma-induced sepsis
(
- Contribution to journal › Article
- 2022
-
Mark
Hepcidin discriminates sepsis from other critical illness at admission to intensive care
(
- Contribution to journal › Article
- 2021
-
Mark
Once Upon a Time
(
- Contribution to journal › Debate/Note/Editorial
-
Mark
Plasma Protein Layer Concealment Protects Streptococcus pyogenes From Innate Immune Attack
(
- Contribution to journal › Article
-
Mark
Modelers Modelling Models
(
- Contribution to journal › Article
-
Mark
Mast Cells and More
(
- Contribution to journal › Debate/Note/Editorial
-
Mark
Some Like It Hot
(
- Contribution to journal › Debate/Note/Editorial
-
Mark
C-Reactive Protein : More than a Biomarker
(
- Contribution to journal › Debate/Note/Editorial
-
Mark
Heparin-binding protein is significantly increased in acute pancreatitis
(
- Contribution to journal › Article
-
Mark
Skeletons in the Cupboard of Dysfunctional Neutrophils Revealed
(
- Contribution to journal › Debate/Note/Editorial
- 2020
-
Mark
Cold Atmospheric Plasma Disarms M1 Protein, an Important Streptococcal Virulence Factor
(
- Contribution to journal › Article
-
Mark
Who is WHO?
(
- Contribution to journal › Debate/Note/Editorial
-
Mark
Interkulturell kompetens kan stärka medicinsk forskning
2020) In Läkartidningen(
- Contribution to journal › Debate/Note/Editorial
-
Mark
Plasma Levels of Hepcidin and Reticulocyte Haemoglobin during Septic Shock
(
- Contribution to journal › Article
-
Mark
Serious, Severe, Sepsis
(
- Contribution to journal › Debate/Note/Editorial
-
Mark
The Road Not Taken : Commensal or Virulent Pathogen
(
- Contribution to journal › Debate/Note/Editorial
-
Mark
A Leak in the Dike
(
- Contribution to journal › Debate/Note/Editorial
-
Mark
The interplay between host haemostatic systems and Leptospira spp. infections
(
- Contribution to journal › Scientific review
- 2019
-
Mark
Osteopontin protects against lung injury caused by extracellular histones
(
- Contribution to journal › Article
-
Mark
Neutrophils engage the kallikrein-kinin system to open up the endothelial barrier in acute inflammation
2019) In FASEB journal : official publication of the Federation of American Societies for Experimental Biology 33(2). p.2599-2609(
- Contribution to journal › Article
-
Mark
Heparin-Binding Protein Release Is Strongly Induced by Leptospira Species and Is a Candidate for an Early Diagnostic Marker of Human Leptospirosis
(
- Contribution to journal › Article
-
Mark
Osteopontin protects against pneumococcal infection in a murine model of allergic airway inflammation
(
- Contribution to journal › Article
-
Mark
A human antithrombin isoform dampens inflammatory responses and protects from organ damage during bacterial infection
(
- Contribution to journal › Article
-
Mark
Tackling the Pros and Cons of Inflammation
(
- Contribution to journal › Debate/Note/Editorial
-
Mark
Renal clearance of heparin-binding protein and elimination during renal replacement therapy : Studies in ICU patients and healthy volunteers
(
- Contribution to journal › Article
-
Mark
Plan S und der Tellerrand
(
- Contribution to journal › Debate/Note/Editorial
-
Mark
Another Brick in the Wall
(
- Contribution to journal › Debate/Note/Editorial
-
Mark
The Extracellular Matrix : Reloaded Revolutions
(
- Contribution to journal › Debate/Note/Editorial
-
Mark
Innate immunity – a clinical perspective
2019) In Journal of Internal Medicine(
- Contribution to journal › Debate/Note/Editorial
- 2018
-
Mark
Heparin-binding protein as a biomarker of post-injury sepsis in trauma patients
(
- Contribution to journal › Article
-
Mark
A fibrin biofilm covers blood clots and protects from microbial invasion
(
- Contribution to journal › Article
-
Mark
An ecoimmunological approach to study evolutionary and ancient links between coagulation, complement and Innate immunity
(
- Contribution to journal › Article
-
Mark
Early depletion of contact system in patients with sepsis : a prospective matched control observational study
(
- Contribution to journal › Article
-
Mark
Catch me if you can or actors on the run
(
- Contribution to journal › Debate/Note/Editorial
-
Mark
With Complement
(
- Contribution to journal › Debate/Note/Editorial
-
Mark
TFPI-2 protects against gram-negative bacterial infection
(
- Contribution to journal › Article
-
Mark
Selected biomarkers correlate with the origin and severity of sepsis
(
- Contribution to journal › Article
-
Mark
Leptospira interrogans outer membrane protein lipl21 is a potent inhibitor of neutrophil myeloperoxidase
(
- Contribution to journal › Article
-
Mark
Globular C1q receptor (p33) binds and stabilizes pro-inflammatory MCP-1 : a novel mechanism for regulation of MCP-1 production and function
(
- Contribution to journal › Article
-
Mark
The Balancing Act of Inflammation
(
- Contribution to journal › Debate/Note/Editorial
-
Mark
Full Complement
(
- Contribution to journal › Debate/Note/Editorial
-
Mark
Going Fishing
(
- Contribution to journal › Debate/Note/Editorial
-
Mark
Journal of Innate Immunity Ten Years Later
(
- Contribution to journal › Debate/Note/Editorial
-
Mark
Protein SIC secreted from Streptococcus pyogenes forms complexes with extracellular histones that boost cytokine production
(
- Contribution to journal › Article
-
Mark
Neutrophil extracellular trap-microparticle complexes enhance thrombin generation via the intrinsic pathway of coagulation in mice
(
- Contribution to journal › Article
-
Mark
Comprehensive Mass Spectrometric Survey of Streptococcus pyogenes Subcellular Proteomes
(
- Contribution to journal › Article
-
Mark
Immunoregulation of Neutrophil Extracellular Trap Formation by Endothelial-Derived p33 (gC1q Receptor)
(
- Contribution to journal › Article
-
Mark
Leucocyte recruitment and molecular fortification of keratinocytes triggered by streptococcal M1 protein
(
- Contribution to journal › Article
-
Mark
Heparin-binding protein in ventilator-induced lung injury
(
- Contribution to journal › Article
- 2017
-
Mark
A rapid method for selecting suitable animal species for studying pathogen interactions with plasma protein ligands in vivo
(
- Contribution to journal › Article
-
Mark
Cells of Innate and Adaptive Immunity : A Matter of Class?
(
- Contribution to journal › Debate/Note/Editorial
-
Mark
Intracellular Clearance by Nobel Laureates
(
- Contribution to journal › Debate/Note/Editorial
-
Mark
DNA-fragmentation is a source of bactericidal activity against Pseudomonas aeruginosa
(
- Contribution to journal › Article
-
Mark
Visions and the Progress of Science
(
- Contribution to journal › Debate/Note/Editorial
-
Mark
Heparin-binding protein as a biomarker of acute kidney injury in critical illness
(
- Contribution to journal › Article
-
Mark
Bystander Cells Taking Action
(
- Contribution to journal › Debate/Note/Editorial
-
Mark
The nonantibiotic macrolide em703 improves survival in a model of quinolone-treated pseudomonas aeruginosa airway infection
(
- Contribution to journal › Article
-
Mark
Paracelsus, poison, and colistin
(
- Contribution to journal › Debate/Note/Editorial
-
Mark
Extracellular nucleic acids in immunity and cardiovascular responses : Between alert and disease
(
- Contribution to journal › Scientific review
- 2016
-
Mark
Osteopontin That Is Elevated in the Airways during COPD Impairs the Antibacterial Activity of Common Innate Antibiotics.
(
- Contribution to journal › Article
-
Mark
Differential neutrophil responses to bacterial stimuli: Streptococcal strains are potent inducers of heparin-binding protein and resistin-release.
(
- Contribution to journal › Article
-
Mark
Large-scale inference of protein tissue origin in gram-positive sepsis plasma using quantitative targeted proteomics.
(
- Contribution to journal › Article
-
Mark
LL-37-induced host cell cytotoxicity depends on cellular expression of the globular C1q receptor (p33).
(
- Contribution to journal › Article
-
Mark
The Origin of a Paradigm
(
- Contribution to journal › Debate/Note/Editorial
-
Mark
Gone with the Wind--Innate Immunity and Airway Inflammation
(
- Contribution to journal › Debate/Note/Editorial
-
Mark
Nobel Parasites in the Kingdom of Invertebrates
(
- Contribution to journal › Debate/Note/Editorial
-
Mark
On PAMPs and DAMPs
(
- Contribution to journal › Debate/Note/Editorial
-
Mark
Modulation of Hemostatic and Inflammatory Responses by Leptospira Spp
(
- Contribution to journal › Article
-
Mark
Increased plasma levels of heparin-binding protein on admission to intensive care are associated with respiratory and circulatory failure
(
- Contribution to journal › Article
- 2015
-
Mark
STAT3-dependent CXC chemokine formation and neutrophil migration in Streptococcal M1 protein-induced acute lung inflammation.
2015) In American Journal of Physiology: Lung Cellular and Molecular Physiology 308(11). p.1159-1167(
- Contribution to journal › Article
-
Mark
Coagulation factor XIII: a multifunctional transglutaminase with clinical potential in a range of conditions
(
- Contribution to journal › Scientific review
-
Mark
Active but inoperable thrombin is accumulated in a plasma protein layer surrounding Streptococcus pyogenes.
(
- Contribution to journal › Article
-
Mark
Alteration of Leukocyte Count Correlates With Increased Pulmonary Vascular Permeability and Decreased PaO2: FiO2-Ratio Early After Major Burns.
(
- Contribution to journal › Article
-
Mark
Extracellular Histones Induce Chemokine Production in Whole Blood Ex Vivo and Leukocyte Recruitment In Vivo.
(
- Contribution to journal › Article
-
Mark
Vigilant Keratinocytes Trigger PAMP Signaling in Response to Streptococcal M1 Protein.
(
- Contribution to journal › Article
-
Mark
Streptococcal M1 protein triggers chemokine formation, neutrophil infiltration, and lung injury in an NFAT-dependent manner.
(
- Contribution to journal › Article
-
Mark
Human endogenous peptide p33 inhibits detrimental effects of LL-37 on osteoblast viability.
(
- Contribution to journal › Article
-
Mark
The Neutrophil : A Beautiful Beast or a Beastly Beauty?
(
- Contribution to journal › Debate/Note/Editorial
-
Mark
Back to the Present
(
- Contribution to journal › Debate/Note/Editorial
-
Mark
Foodies of Innate Immunity
(
- Contribution to journal › Debate/Note/Editorial
-
Mark
Interferon-λ : Inters Ferocity or Inter-Ferocities?
(
- Contribution to journal › Debate/Note/Editorial
-
Mark
All that glisters is not gold - Staphylococcus aureus and innate immunity
(
- Contribution to journal › Debate/Note/Editorial
-
Mark
Chasing flies because time flies
(
- Contribution to journal › Debate/Note/Editorial
-
Mark
Sepsis - vår tids okända folksjukdom
(
- Contribution to journal › Debate/Note/Editorial
- 2014
-
Mark
Inflammatory role and prognostic value of platelet chemokines in acute coronary syndrome
(
- Contribution to journal › Article
-
Mark
Treatment with p33 Curtails Morbidity and Mortality in a Histone-Induced Murine Shock Model.
(
- Contribution to journal › Article
-
Mark
Targeted mass spectrometry analysis of neutrophil-derived proteins released during sepsis progression.
(
- Contribution to journal › Article
-
Mark
Ras regulates alveolar macrophage formation of CXC chemokines and neutrophil activation in streptococcal M1 protein-induced lung injury.
(
- Contribution to journal › Article
-
Mark
Heparin-binding protein (HBP/CAP37) - a link to endothelin-1 in endotoxemia-induced pulmonary oedema?
(
- Contribution to journal › Article
-
Mark
A Farewell to Arms: Streptococcal Strategies to Cope with Innate Immunity
(
- Contribution to journal › Debate/Note/Editorial
-
Mark
Staphylococcus aureus induced clotting of plasma is an immune evasion mechanism to persist within the fibrin network.
2014) In Microbiology(
- Contribution to journal › Article
-
Mark
The Janus face of macrophages in immunity
(
- Contribution to journal › Debate/Note/Editorial
-
Mark
Unexpected and novel functions of complement proteins
(
- Contribution to journal › Debate/Note/Editorial
-
Mark
Surveillance and countermeasures in innate immunity
(
- Contribution to journal › Debate/Note/Editorial
-
Mark
The coagulation system and its function in early immune defense.
(
- Contribution to journal › Article
- 2013
-
Mark
Antimicrobial activity of fibrinogen and fibrinogen-derived peptides - a novel link between coagulation and innate immunity.
(
- Contribution to journal › Article
-
Mark
Anti-inflammatory, anti-coagulant, anti-biotic?
(
- Contribution to journal › Debate/Note/Editorial
-
Mark
Simvastatin decreases the level of heparin-binding protein in patients with acute lung injury
(
- Contribution to journal › Article
-
Mark
Heparin binding protein in patients with acute respiratory failure treated with granulocyte colony-stimulating factor (filgrastim) - a prospective, placebo-controlled, double-blind study
(
- Contribution to journal › Article
-
Mark
Targeting rac1 signaling inhibits streptococcal m1 protein-induced CXC chemokine formation, neutrophil infiltration and lung injury.
(
- Contribution to journal › Article
-
Mark
Heparin-binding protein (HBP) in critically ill patients with influenza A (H1N1) infection
(
- Contribution to journal › Article
-
Mark
Heparin-binding protein (HBP): an early marker of respiratory failure after trauma?
(
- Contribution to journal › Article
-
Mark
Geranylgeranyl transferase regulates streptococcal m1 protein-induced CXC chemokine formation and neutrophil recruitment in the lung.
(
- Contribution to journal › Article
-
Mark
A Novel Role for Pro-Coagulant Microvesicles in the Early Host Defense against Streptococcus pyogenes
(
- Contribution to journal › Article
-
Mark
Targeting CD162 protects against streptococcal M1 protein-evoked neutrophil recruitment and lung injury
(
- Contribution to journal › Article
-
Mark
p33 (gC1q Receptor) Prevents Cell Damage by Blocking the Cytolytic Activity of Antimicrobial Peptides.
(
- Contribution to journal › Article
-
Mark
Macrophages : past, present and future
(
- Contribution to journal › Debate/Note/Editorial
-
Mark
Of DAMPs and macrophages
(
- Contribution to journal › Debate/Note/Editorial
- 2012
-
Mark
Streptococcal M1 protein triggers farnesyltransferase-dependent formation of CXC chemokines in alveolar macrophages and neutrophil infiltration in the lung.
(
- Contribution to journal › Article
-
Mark
Streptococcal M1 Protein-Provoked CXC Chemokine Formation, Neutrophil Recruitment and Lung Damage Are Regulated by Rho-Kinase Signaling.
(
- Contribution to journal › Article
-
Mark
Stimulation of blood mononuclear cells with bacterial virulence factors leads to the release of pro-coagulant and pro-inflammatory microparticles.
(
- Contribution to journal › Article
-
Mark
p38 Mitogen-activated protein kinase signaling regulates streptococcal M1 protein-induced neutrophil activation and lung injury.
(
- Contribution to journal › Article
-
Mark
Modulation of the Coagulation System During Severe Streptococcal Disease.
2012) In Current Topics in Microbiology and Immunology(
- Contribution to journal › Article
-
Mark
Increased plasma levels of heparin-binding protein in patients with shock: a prospective, cohort study.
(
- Contribution to journal › Article
-
Mark
Bacterial Abuse of Mammalian Extracellular Proteases during Tissue Invasion and Infection
2012) p.157-180(
- Chapter in Book/Report/Conference proceeding › Book chapter
- 2011
-
Mark
Protein C Inhibitor
(
- Contribution to journal › Article
-
Mark
Simvastatin regulates CXC chemokine formation in streptococcal M1 protein-induced neutrophil infiltration in the lung
(
- Contribution to journal › Article
-
Mark
Coagulation, an ancestral serine protease cascade, exerts a novel function in early immune defense.
(
- Contribution to journal › Article
-
Mark
Binding characteristics of thrombin-activatable fibrinolysis inhibitor to streptococcal surface collagen-like proteins A and B
(
- Contribution to journal › Article
-
Mark
beta(2)-Glycoprotein I: a novel component of innate immunity
(
- Contribution to journal › Article
-
Mark
Induction of Auto-Antibodies Against β(2) -Glycoprotein I in Mice by Protein H of Streptococcus Pyogenes.
(
- Contribution to journal › Article
-
Mark
STREPTOCOCCAL M1 PROTEIN-INDUCED LUNG INJURY IS INDEPENDENT OF PLATELETS IN MICE.
(
- Contribution to journal › Article
-
Mark
Coagulation Systems of Invertebrates and Vertebrates and Their Roles in Innate Immunity: The Same Side of Two Coins?
(
- Contribution to journal › Article
-
Mark
Hemostasis in Invertebrates and Vertebrates: An Evolutionary Excursion.
(
- Contribution to journal › Article
-
Mark
Adsorption of Components of the Plasma Kinin-forming System on the Surface of Porphyromonas gingivalis Involves Gingipains as the Major Docking Platforms.
(
- Contribution to journal › Article
-
Mark
The discovery of pathogen recognition receptors (PRR)
(
- Contribution to journal › Debate/Note/Editorial
-
Mark
A summing-up of 2010 in the journal of innate immunity
(
- Contribution to journal › Debate/Note/Editorial
-
Mark
Foreword
(
- Chapter in Book/Report/Conference proceeding › Foreword/Postscript
-
Mark
Sepsis - pro-inflammatory and anti-inflammatory responses : Good, bad or ugly?
2011) 17.(
- Book/Report › Anthology (editor)
-
Mark
Evaluation of potential biomarkers for the discrimination of bacterial and viral infections
(
- Contribution to journal › Article
- 2010
-
Mark
Heparin-binding protein as a biomarker of circulatory failure during severe infections: A report of three cases
(
- Contribution to journal › Debate/Note/Editorial
-
Mark
M1 protein from Streptococcus pyogenes induces nitric oxidemediated vascular hyporesponsiveness to phenylephrine: Involvement of toll-like receptor activation.
(
- Contribution to journal › Article
-
Mark
Elevated Plasma Levels of Antimicrobial Polypeptides in Patients with Severe Sepsis.
(
- Contribution to journal › Article
-
Mark
Heparin-Binding Protein: A Potential Biomarker in Sepsis? Reply
(
- Contribution to journal › Letter
-
Mark
Contact system activation in severe infectious diseases.
(
- Contribution to journal › Scientific review
-
Mark
Binding characteristics of thrombin-activatable fibrinolysis inhibitor to streptococcal surface collagen-like proteins A and B
(
- Contribution to journal › Published meeting abstract
-
Mark
Streptococcal M1 protein-induced lung injury is independent of platelets
2010) 45th Congress of the European-Society-for-Surgical-Research In British Journal of Surgery 97. p.44-44(
- Contribution to journal › Published meeting abstract
-
Mark
Pathogen Entrapment by Transglutaminase-A Conserved Early Innate Immune Mechanism
(
- Contribution to journal › Article
-
Mark
Streptococcal inhibitor of complement-mediated lysis (SIC): an anti-inflammatory virulence determinant
(
- Contribution to journal › Article
-
Mark
Aberrant Inflammatory Response to Streptococcus pyogenes in Mice Lacking Myeloid Differentiation Factor 88.
(
- Contribution to journal › Article
-
Mark
Streptococcal M proteins and their role as virulence determinants.
(
- Contribution to journal › Article
-
Mark
Journal of Innate Immunity : Editors' choice
(
- Contribution to journal › Debate/Note/Editorial
- 2009
-
Mark
Protein C inhibitor--a novel antimicrobial agent.
(
- Contribution to journal › Article
-
Mark
Neutrophil-derived heparin binding protein-A mediator of increased vascular permeability after burns?
(
- Contribution to journal › Article
-
Mark
Activation of TAFI on the Surface of Streptococcus pyogenes Evokes Inflammatory Reactions by Modulating the Kallikrein/Kinin System
(
- Contribution to journal › Article
-
Mark
Treatment of invasive streptococcal infection with a peptide derived from human high molecular weight kininogen.
(
- Contribution to journal › Article
-
Mark
Neutrophil-Derived Hyperresistinemia in Severe Acute Streptococcal Infections
(
- Contribution to journal › Article
-
Mark
Activation of the Human Contact System on Neutrophil Extracellular Traps
(
- Contribution to journal › Article
-
Mark
Heparin-binding protein: an early marker of circulatory failure in sepsis.
(
- Contribution to journal › Article
-
Mark
Bacterial proteases disarming host defense
(
- Contribution to journal › Debate/Note/Editorial
- 2008
-
Mark
Neutrophil degranulation mediates severe lung damage triggered by streptococcal M1 protein
(
- Contribution to journal › Article
-
Mark
Soluble M1 protein of Streptococcus pyogenes triggers potent T cell activation.
(
- Contribution to journal › Article
-
Mark
Neutrophil secretion products pave the way for inflammatory monocytes
(
- Contribution to journal › Article
-
Mark
The antibacterial activity of peptides derived from human beta-2 glycoprotein I is inhibited by protein H and M1 protein from Streptococcus pyogenes.
(
- Contribution to journal › Article
-
Mark
Immunological Mechanisms Underlying the Genetic Predisposition to Severe Staphylococcus aureus Infection in the Mouse Model
(
- Contribution to journal › Article
-
Mark
Kinin B2 Receptor-Mediated Bradykinin Internalization and Metalloendopeptidase EP24.15-Dependent Intracellular Bradykinin Degradation.
(
- Contribution to journal › Article
-
Mark
Infectious Agents, the Contact System, and Innate Immunity
2008) 41st Congress of the German-Society-for-Transfusion-Medicine-and-Immunohematology/Interdiscip linary European-Society-for-Hemapheresis-and-Hemotherapy p.60-70(
- Chapter in Book/Report/Conference proceeding › Paper in conference proceeding
-
Mark
Regulation of kinin B(2) receptors by bradykinin in human lung cells.
(
- Contribution to journal › Article
-
Mark
Antibodies against a surface protein of Streptococcus pyogenes promote a pathological inflammatory response.
(
- Contribution to journal › Article
-
Mark
Neutrophil primary granule proteins HBP and HNP1-3 boost bacterial phagocytosis by human and murine macrophages.
(
- Contribution to journal › Article
- 2007
-
Mark
M protein from Streptococcus pyogenes induces tissue factor expression and pro-coagulant activity in human monocytes
(
- Contribution to journal › Article
-
Mark
Thrombin activatable fibrinolysis inhibitor binds to Streptococcus pyogenes by interacting with collagen-like surface proteins A and B.
(
- Contribution to journal › Article
-
Mark
The dual role of the contact system in bacterial infectious disease
(
- Contribution to journal › Article
-
Mark
Haemostasis, vascular biology, and infectious agents.
(
- Contribution to journal › Letter
- 2006
-
Mark
Kinin receptor expression during Staphylococcus aureus infection.
(
- Contribution to journal › Article
-
Mark
Activation of human polymorphonuclear neutrophils by streptolysin O from Streptococcus pyogenes leads to the release of proinflammatory mediators.
(
- Contribution to journal › Article
-
Mark
Streptococcal M protein: a multipotent and powerful inducer of inflammation.
(
- Contribution to journal › Article
-
Mark
The contact system-a novel branch of innate immunity generating antibacterial peptides.
(
- Contribution to journal › Article
- 2004
-
Mark
Heparin binding protein is increased in chronic leg ulcer fluid and released from granulocytes by secreted products of Pseudomonas aeruginosa.
(
- Contribution to journal › Article
-
Mark
M Protein, a Classical Bacterial Virulence Determinant, Forms Complexes with Fibrinogen that Induce Vascular Leakage
(
- Contribution to journal › Article
- 2003
-
Mark
The conversion of fibrinogen to fibrin at the surface of curliated E. coli bacteria leads to the generation of proinflammatory fibrinopeptides.
(
- Contribution to journal › Article
-
Mark
Superantigens from Staphylococcus aureus induce procoagulant activity and monocyte tissue factor expression in whole blood and mononuclear cells via IL-1beta.
(
- Contribution to journal › Article
-
Mark
Interactions between surface proteins of Streptococcus pyogenes and coagulation factors modulate clotting of human plasma.
(
- Contribution to journal › Article
-
Mark
Contact activation by pathogenic bacteria: a virulence mechanism contributing to the pathophysiology of sepsis.
(
- Contribution to journal › Article
-
Mark
Modulation of membrane traffic by microorganisms.
(
- Contribution to journal › Article
-
Mark
Superantigens from Staphylococcus aureus induce procoagulant activity and monocyte tissue factor expression in whole blood and mononuclear cells via IL-1 beta
(
- Contribution to journal › Article
- 2002
-
Mark
Identification of two protein-binding and functional regions of curli, a surface organelle and virulence determinant of Escherichia coli
(
- Contribution to journal › Article
-
Mark
Contact-system activation in children with vasculitis.
(
- Contribution to journal › Article
-
Mark
Secretion of heparin-binding protein from human neutrophils is determined by its localization in azurophilic granules and secretory vesicles.
(
- Contribution to journal › Article
-
Mark
Peptidoglycan from Staphylococcus aureus induces tissue factor expression and procoagulant activity in human monocytes.
(
- Contribution to journal › Article
- 2001
-
Mark
Heparin-binding protein (HBP/CAP37): A missing link in neutrophil-evoked alteration of vascular permeability
(
- Contribution to journal › Article
-
Mark
Zinc-dependent conformational changes in domain D5 of high molecular mass kininogen modulate contact activation
(
- Contribution to journal › Article
-
Mark
Staphylococcus aureus induces release of bradykinin in human plasma
(
- Contribution to journal › Article
- 2000
-
Mark
Severe lung lesions caused by Salmonella are prevented by inhibition of the contact system
(
- Contribution to journal › Article
-
Mark
Modulation of hemostatic mechanisms in bacterial infectious diseases
(
- Contribution to journal › Scientific review
-
Mark
Signaling via β2 integrins triggers neutrophil-dependent alteration in endothelial barrier function
(
- Contribution to journal › Article
- 1999
-
Mark
Heparin-binding protein targeted to mitochondrial compartments protects endothelial cells from apoptosis
(
- Contribution to journal › Article
- 1998
-
Mark
Activation of the contact-phase system on bacterial surfaces - A clue to serious comlications in infections deseases
(
- Contribution to journal › Article
- 1997
-
Mark
Absorption of kininogen from human plasma by Streptococcus pyogenes is followed by the release of bradykinin
(
- Contribution to journal › Article
- 1996
-
Mark
Streptococcal cysteine proteinase releases kinins: a novel virulence mechanism
(
- Contribution to journal › Article
-
Mark
Binding of Kininogen to the surface of Streptococcus pyogenes in plasma environment is followed by release of bradykinin.
1996) In Historielärarnas Förenings Årsskrift(
- Contribution to journal › Article
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Mark
Isolation and characterization of the kininogen-binding protein p33 from endothelial cells : Identity with the gClq receptor
(
- Contribution to journal › Article
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Mark
Mapping of the discontinuous kininogen binding site of prekallikrein : A distal binding segment is located in the heavy chain domain A4
(
- Contribution to journal › Article
- 1995
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Mark
Human kininogens interact with M protein, a bacterial surface protein and virulence determinant.
(
- Contribution to journal › Article
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Mark
Identification of an endothelial cell binding site on kininogen domain D3
(
- Contribution to journal › Article
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Mark
Mapping the cell binding site on high molecular weight kininogen domain 5
(
- Contribution to journal › Article
- 1993
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Mark
Mapping of the high molecular weight kininogen binding site of prekallikrein. Evidence for a discontinuous epitope formed by distinct segments of the prekallikrein heavy chain
(
- Contribution to journal › Article
- 1992
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Mark
Mapping of the H-kininogen binding site exposed by the prekallikrein heavy chain
(
- Contribution to journal › Article